Wnt/β-catenin signaling is differentially regulated by Gα proteins and contributes to fibrous dysplasia. - Wikidata - wikimedia - TriplyDB

Wnt/β-catenin signaling is differentially regulated by Gα proteins and contributes to fibrous dysplasia.

Wnt/β-catenin signaling is differentially regulated by Gα proteins and contributes to fibrous dysplasia.

http://www.wikidata.org/entity/Q35647471

about

G Protein-Coupled Receptors in Cancer Wnts' fashion statement: from body stature to dysplasia.Osteoclasts: more than 'bone eaters'.Fibro-osseous lesions of the craniofacial bones: β-catenin immunohistochemical analysis and CTNNB1 and APC mutation analysis.Molecular, phenotypic aspects and therapeutic horizons of rare genetic bone disorders.Progressive osseous heteroplasia: diagnosis, treatment, and prognosis The G protein α subunit Gαs is a tumor suppressor in Sonic hedgehog-driven medulloblastoma Protein kinase A activation enhances β-catenin transcriptional activity through nuclear localization to PML bodies.Vegfa regulates perichondrial vascularity and osteoblast differentiation in bone development G-Protein α-Subunit Gsα Is Required for Craniofacial Morphogenesis.New therapeutic targets in rare genetic skeletal diseases.Wnt signaling in bone development and disease: making stronger bone with Wnts Role of Regulators of G Protein Signaling Proteins in Bone Physiology and Pathophysiology.Novel designer receptors to probe GPCR signaling and physiology Activation of Hedgehog signaling by loss of GNAS causes heterotopic ossification.Role of regulator of G protein signaling proteins in bone.Gsα Controls Cortical Bone Quality by Regulating Osteoclast Differentiation via cAMP/PKA and β-Catenin Pathways.Osteoblast dysfunctions in bone diseases: from cellular and molecular mechanisms to therapeutic strategies.Alternative Wnt Signaling Activates YAP/TAZ.Loss of Gi G-Protein-Coupled Receptor Signaling in Osteoblasts Accelerates Bone Fracture Healing.Genetic evidence that β-arrestins are dispensable for the initiation of β2-adrenergic receptor signaling to ERK.Ossifications in Albright Hereditary Osteodystrophy: Role of Genotype, Inheritance, Sex, Age, Hormonal Status, and BMI.Expression of an active Gαs mutant in skeletal stem cells is sufficient and necessary for fibrous dysplasia initiation and maintenance.Cancer driver G-protein coupled receptor (GPCR) induced β-catenin nuclear localization: the transcriptional junction.Induced Gnas(R201H) expression from the endogenous Gnas locus causes fibrous dysplasia by up-regulating Wnt/β-catenin signaling.Overexpression of Gα11 in Osteoblast Lineage Cells Suppresses the Osteoanabolic Response to Intermittent PTH and Exercise.The curious case of Gαs gain-of-function in neoplasia.

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Wnt/β-catenin signaling is differentially regulated by Gα proteins and contributes to fibrous dysplasia.

http://www.wikidata.org/entity/Q35647471

description

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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PNAS.1114656108

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Proceedings of the National Academy of Sciences of the United States of America

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Wnt/β-catenin signaling is dif ...... tributes to fibrous dysplasia.

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P2093

Daniel Palmer

http://www.w3.org/2001/XMLSchema#string

Francesco S Celi

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Jean B Regard

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Jean-Marc Guettier

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Jurgen Wess

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Lee S Weinstein

http://www.w3.org/2001/XMLSchema#string

Michael T Collins

http://www.w3.org/2001/XMLSchema#string

Min Chen

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Natasha Cherman

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Nisan Bhattacharyya

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P2860

Galpha 13 requires palmitoylation for plasma membrane localization, Rho-dependent signaling, and promotion of p115-RhoGEF membrane binding

Mutant Frizzled 4 associated with vitreoretinopathy traps wild-type Frizzled in the endoplasmic reticulum by oligomerization

Intestinal polyposis in mice with a dominant stable mutation of the beta-catenin gene

A mutation in the LDL receptor-related protein 5 gene results in the autosomal dominant high-bone-mass trait

High bone mass in mice expressing a mutant LRP5 gene

Thyroid carcinoma in the McCune-Albright syndrome: contributory role of activating Gs alpha mutations

LDL receptor-related protein 5 (LRP5) affects bone accrual and eye development

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Crypt stem cells as the cells-of-origin of intestinal cancer

Canonical Wnt signaling in differentiated osteoblasts controls osteoclast differentiation

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10.1073/PNAS.1114656108

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