Scorpion toxin BmK I directly activates Nav1.8 in primary sensory neurons to induce neuronal hyperexcitability in rats.
about
Brain natriuretic peptide suppresses pain induced by BmK I, a sodium channel-specific modulator, in rats.Local knockdown of Nav1.6 relieves pain behaviors induced by BmK I.Functional up-regulation of Nav1.8 sodium channel on dorsal root ganglia neurons contributes to the induction of scorpion sting pain.
P2860
Scorpion toxin BmK I directly activates Nav1.8 in primary sensory neurons to induce neuronal hyperexcitability in rats.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
@zh-cn
name
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@ast
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@en
type
label
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@ast
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@en
prefLabel
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@ast
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@en
P2093
P2860
P1433
P1476
Scorpion toxin BmK I directly ...... nal hyperexcitability in rats.
@en
P2093
Feng Jiang
Liming Hua
Yonghua Ji
Yunlu Jiao
Zhenwei Li
P2860
P2888
P304
P356
10.1007/S13238-015-0154-4
P577
2015-04-24T00:00:00Z