Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β. - Wikidata - wikimedia - TriplyDB

Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.

Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.

http://www.wikidata.org/entity/Q35659179

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Helminth infections and host immune regulation Intravital immunofluorescence for visualizing the microcirculatory and immune microenvironments in the mouse ear dermis Chronic filarial infection provides protection against bacterial sepsis by functionally reprogramming macrophages Unraveling the Hygiene Hypothesis of helminthes and autoimmunity: origins, pathophysiology, and clinical applications Helminthic therapy: improving mucosal barrier function Therapeutic effect of Schistosoma japonicum cystatin on bacterial sepsis in mice.Nematode-induced interference with vaccination efficacy targets follicular T helper cell induction and is preserved after termination of infection Comparative analysis of the secretome from a model filarial nematode (Litomosoides sigmodontis) reveals maximal diversity in gravid female parasites.Helminth infections decrease host susceptibility to immune-mediated diseases.Helminths and their implication in sepsis - a new branch of their immunomodulatory behaviour?Systemic impact of intestinal helminth infections.Beyond genetics. Influence of dietary factors and gut microbiota on type 1 diabetes.Parasitic helminths and their beneficial impact on type 1 and type 2 diabetes.Nematode asparaginyl-tRNA synthetase resolves intestinal inflammation in mice with T-cell transfer colitis.Parasitic nematode-induced modulation of body weight and associated metabolic dysfunction in mouse models of obesity.Induction and regulation of pathogenic Th17 cell responses in schistosomiasis.Th2 and eosinophil responses suppress inflammatory arthritis.The hygiene hypothesis: current perspectives and future therapies Helminth infection and type 1 diabetes.Regulatory T cell subsets in filarial infection and their function.Type 2 immunity and wound healing: evolutionary refinement of adaptive immunity by helminths.Embryonic trophoblasts induce decidual regulatory T cell differentiation and maternal-fetal tolerance through thymic stromal lymphopoietin instructing dendritic cells.Viruses and Type 1 diabetes: a dynamic labile equilibrium.The pathologic and clinical intersection of atopic and autoimmune disease.Autoimmunity in 2012.Induction of regulatory cells by helminth parasites: exploitation for the treatment of inflammatory diseases.CD4 T cell differentiation in type 1 diabetes.Strongyloides infection in rodents: immune response and immune regulation.Brugia malayi soluble and excretory-secretory proteins attenuate development of streptozotocin-induced type 1 diabetes in mice.Are Regulatory T Cells Defective in Type 1 Diabetes and Can We Fix Them?Immunomodulation by helminths: Similar impact on type 1 and type 2 diabetes?Litomosoides sigmodontis induces TGF-β receptor responsive, IL-10-producing T cells that suppress bystander T-cell proliferation in mice.Differential immune responses in mice infected with the tissue-dwelling nematode Trichinella zimbabwensis.Therapeutic potential of helminths in autoimmune diseases: helminth-derived immune-regulators and immune balance.Foxp3(+) regulatory T cells in mouse models of type 1 diabetes Is there a role for gut microbiota in type 1 diabetes pathogenesis?Escherichia coli-induced immune paralysis is not exacerbated during chronic filarial infection.Expression profiles of long non-coding RNAs located in autoimmune disease-associated regions reveal immune cell-type specificity.Immune modulation of Th1, Th2, and T-reg transcriptional factors differing from cytokine levels in Schistosoma japonicum infection.The hygiene hypothesis in autoimmunity: the role of pathogens and commensals.

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Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.

http://www.wikidata.org/entity/Q35659179

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Helminth protection against au ...... mune shift and requires TGF-β.

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JIMMUNOL.1100335

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Journal of Immunology

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Helminth protection against au ...... mune shift and requires TGF-β.

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David Larson

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Edward Mitre

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Ellen Mueller

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Fabian Gondorf

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J Thomas Stocker

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Kateryna Soloviova

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Kristin E Killoran

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Kristin V Tarbell

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Marc P Hübner

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Marina N Torrero

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P2860

Intestinal nematode infection ameliorates experimental colitis in mice

Inhibition of autoimmune type 1 diabetes by gastrointestinal helminth infection

Trichuris suis therapy in Crohn's disease

Trichuris suis therapy for active ulcerative colitis: a randomized controlled trial

Association between parasite infection and immune responses in multiple sclerosis

Helminths as governors of immune-mediated inflammation

TGF-beta: a master of all T cell trades

Identification of tgh-2, a filarial nematode homolog of Caenorhabditis elegans daf-7 and human transforming growth factor beta, expressed in microfilarial and adult stages of Brugia malayi.

IL-2 reverses established type 1 diabetes in NOD mice by a local effect on pancreatic regulatory T cells

Helminth secretions induce de novo T cell Foxp3 expression and regulatory function through the TGF-β pathway.

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51884405

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