Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAE - Wikidata - wikimedia - TriplyDB

Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAE

Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAE

http://www.wikidata.org/entity/Q35669064

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Th17 Cells Pathways in Multiple Sclerosis and Neuromyelitis Optica Spectrum Disorders: Pathophysiological and Therapeutic Implications Monoclonal antibody therapies for the treatment of relapsing-remitting multiple sclerosis: differentiating mechanisms and clinical outcomes Leukocyte integrins: role in leukocyte recruitment and as therapeutic targets in inflammatory disease Focal transient CNS vessel leak provides a tissue niche for sequential immune cell accumulation during the asymptomatic phase of EAE induction.NLRP3 inflammasome induces chemotactic immune cell migration to the CNS in experimental autoimmune encephalomyelitis.α4-integrins control viral meningoencephalitis through differential recruitment of T helper cell subsets.Interleukin 17A Promotes Lymphocytes Adhesion and Induces CCL2 and CXCL1 Release from Brain Endothelial Cells.Aquaporin-4 antibodies in patients treated with natalizumab for suspected MS.An interferon-β-resistant and NLRP3 inflammasome-independent subtype of EAE with neuronal damage Galectin-8 as an immunosuppressor in experimental autoimmune encephalomyelitis and a target of human early prognostic antibodies in multiple sclerosis.Activation of the STING adaptor attenuates experimental autoimmune encephalitis.Cytokine-regulated neutrophil recruitment is required for brain but not spinal cord inflammation during experimental autoimmune encephalomyelitis.The immune response after hypoxia-ischemia in a mouse model of preterm brain injury.Deletion of IL-33R (ST2) abrogates resistance to EAE in BALB/C mice by enhancing polarization of APC to inflammatory phenotype Distinct pattern of lesion distribution in multiple sclerosis is associated with different circulating T-helper and helper-like innate lymphoid cell subsets.Th17-biased RORγt transgenic mice become susceptible to a viral model for multiple sclerosis.Immunological Markers for PML Prediction in MS Patients Treated with Natalizumab Integrin alpha L controls the homing of regulatory T cells during CNS autoimmunity in the absence of integrin alpha 4.Unmasking and provoking severe disease activity in a patient with NMO spectrum disorder Developmental endothelial locus-1 is a homeostatic factor in the central nervous system limiting neuroinflammation and demyelination.Early treatment with anti-VLA-4 mAb can prevent the infiltration and/or development of pathogenic CD11b+CD4+ T cells in the CNS during progressive EAE.B-cell very late antigen-4 deficiency reduces leukocyte recruitment and susceptibility to central nervous system autoimmunity.T cell-associated α4β7 but not α4β1 integrin is required for the induction and perpetuation of chronic colitis Interaction of astrocytes and T cells in physiological and pathological conditions Eomesodermin-expressing T-helper cells are essential for chronic neuroinflammation.Phenotype of Antigen Unexperienced TH Cells in the Inflamed Central Nervous System in Experimental Autoimmune Encephalomyelitis.Multiple functional therapeutic effects of TnP: A small stable synthetic peptide derived from fish venom in a mouse model of multiple sclerosis.Group A Streptococcus intranasal infection promotes CNS infiltration by streptococcal-specific Th17 cells Cancer regression and neurological toxicity following anti-MAGE-A3 TCR gene therapy.Origins and significance of astrogliosis in the multiple sclerosis model, MOG peptide EAE Targeting endothelial CD146 attenuates neuroinflammation by limiting lymphocyte extravasation to the CNS.Mechanisms regulating regional localization of inflammation during CNS autoimmunity.Tumour suppressor death-associated protein kinase targets cytoplasmic HIF-1α for Th17 suppression.Opioid growth factor and low-dose naltrexone impair central nervous system infiltration by CD4 + T lymphocytes in established experimental autoimmune encephalomyelitis, a model of multiple sclerosis Intercellular adhesion molecule 1 mediates migration of Th1 and Th17 cells across human retinal vascular endothelium B7-H1 shapes T-cell-mediated brain endothelial cell dysfunction and regional encephalitogenicity in spontaneous CNS autoimmunity.Cisplatin induces tolerogenic dendritic cells in response to TLR agonists via the abundant production of IL-10, thereby promoting Th2- and Tr1-biased T-cell immunity.Expression and function of the homeostatic molecule Del-1 in endothelial cells and the periodontal tissue.Tolerogenic Dendritic Cells Generated with Tofacitinib Ameliorate Experimental Autoimmune Encephalomyelitis through Modulation of Th17/Treg Balance.Review of Animal Models of Neuromyelitis Optica.

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P2860

Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAE

http://www.wikidata.org/entity/Q35669064

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Th17 lymphocytes traffic to th ...... integrin expression during EAE

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Th17 lymphocytes traffic to th ...... integrin expression during EAE

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Th17 lymphocytes traffic to th ...... integrin expression during EAE

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P1433

Journal of Experimental Medicine

P1476

Th17 lymphocytes traffic to th ...... integrin expression during EAE

@en

P2093

Bernhard Hemmer

http://www.w3.org/2001/XMLSchema#string

Franziska Petermann

http://www.w3.org/2001/XMLSchema#string

Malte C Claussen

http://www.w3.org/2001/XMLSchema#string

Rajneesh Srivastava

http://www.w3.org/2001/XMLSchema#string

Veit Rothhammer

http://www.w3.org/2001/XMLSchema#string

P2860

The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

Fate mapping of IL-17-producing T cells in inflammatory responses

CXCR7 influences leukocyte entry into the CNS parenchyma by controlling abluminal CXCL12 abundance during autoimmunity

Stabilizing the VE-cadherin-catenin complex blocks leukocyte extravasation and vascular permeability

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

IL-17 and Th17 Cells

Transforming growth factor-beta induces development of the T(H)17 lineage

VLA-4 integrin concentrates at the peripheral supramolecular activation complex of the immune synapse and drives T helper 1 responses

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2465-2476

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scholarly article

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10.1084/JEM.20110434

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12

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208

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2011-10-24T00:00:00Z

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51743396

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22025301

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3256959

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