Protein kinase C enhances human sodium channel hNav1.7 resurgent currents via a serine residue in the domain III-IV linker.
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Navβ4 regulates fast resurgent sodium currents and excitability in sensory neuronsPost-translational modifications of voltage-gated sodium channels in chronic pain syndromesAntihyperalgesic effect of tetrodotoxin in rat models of persistent muscle pain.Biphasic voltage-dependent inactivation of human NaV 1.3, 1.6 and 1.7 Na+ channels expressed in rodent insulin-secreting cells.Posttranslational Modification of Sodium Channels.[EXPRESS] Nav1.7 is phosphorylated by Fyn tyrosine kinase which modulates channel expression and gating in a cell type-dependent manner.BDNF-TrkB Signaling Coupled to nPKCε and cPKCβI Modulate the Phosphorylation of the Exocytotic Protein Munc18-1 During Synaptic Activity at the Neuromuscular Junction.
P2860
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P2860
Protein kinase C enhances human sodium channel hNav1.7 resurgent currents via a serine residue in the domain III-IV linker.
description
2014 nî lūn-bûn
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2014年の論文
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name
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@ast
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@en
type
label
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@ast
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@en
prefLabel
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@ast
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@en
P2093
P2860
P1433
P1476
Protein kinase C enhances huma ...... e in the domain III-IV linker.
@en
P2093
Birgit T Priest
Eric S Nisenbaum
Jeffrey L Krajewski
Kelly L Knopp
Theodore R Cummins
P2860
P304
P356
10.1016/J.FEBSLET.2014.09.011
P407
P50
P577
2014-09-19T00:00:00Z