Activation of growth hormone releasing hormone (GHRH) receptor stimulates cardiac reverse remodeling after myocardial infarction (MI) - Wikidata - wikimedia - TriplyDB

Activation of growth hormone releasing hormone (GHRH) receptor stimulates cardiac reverse remodeling after myocardial infarction (MI)

Activation of growth hormone releasing hormone (GHRH) receptor stimulates cardiac reverse remodeling after myocardial infarction (MI)

http://www.wikidata.org/entity/Q35673689

about

Growth Hormone-Releasing Hormone and Its Analogues: Significance for MSCs-Mediated Angiogenesis Potential new role of the GHSR-1a-mediated signaling pathway in cardiac remodeling after myocardial infarction (Review).Local activation of cardiac stem cells for post-myocardial infarction cardiac repair Agonists of growth hormone-releasing hormone stimulate self-renewal of cardiac stem cells and promote their survival.Coronary Microvascular Function and Beyond: The Crosstalk between Hormones, Cytokines, and Neurotransmitters New therapeutic approach to heart failure due to myocardial infarction based on targeting growth hormone-releasing hormone receptor.Growth hormone-releasing hormone agonists reduce myocardial infarct scar in swine with subacute ischemic cardiomyopathy.S-Nitrosoglutathione Reductase Deficiency Enhances the Proliferative Expansion of Adult Heart Progenitors and Myocytes Post Myocardial Infarction Novel GHRH antagonists suppress the growth of human malignant melanoma by restoring nuclear p27 function.cKit+ cardiac progenitors of neural crest origin.GHRH antagonist when combined with cytotoxic agents induces S-phase arrest and additive growth inhibition of human colon cancer.Synthesis of new potent agonistic analogs of growth hormone-releasing hormone (GHRH) and evaluation of their endocrine and cardiac activities.Transplantation of pancreatic islets to adrenal gland is promoted by agonists of growth-hormone-releasing hormone.S-nitrosoglutathione reductase (GSNOR) enhances vasculogenesis by mesenchymal stem cells Profound Actions of an Agonist of Growth Hormone-Releasing Hormone on Angiogenic Therapy by Mesenchymal Stem Cells.Pretreatment of Cardiac Stem Cells With Exosomes Derived From Mesenchymal Stem Cells Enhances Myocardial Repair Endothelial cells overexpressing IL-8 receptor reduce cardiac remodeling and dysfunction following myocardial infarction.Peripheral activities of growth hormone-releasing hormone.Agonistic analogs of growth hormone releasing hormone (GHRH) promote wound healing by stimulating the proliferation and survival of human dermal fibroblasts through ERK and AKT pathways High-Intensity Exercise Reduces Cardiac Fibrosis and Hypertrophy but Does Not Restore the Nitroso-Redox Imbalance in Diabetic Cardiomyopathy Expression of hypothalamic neurohormones and their receptors in the human eye Growth hormone-releasing hormone attenuates cardiac hypertrophy and improves heart function in pressure overload-induced heart failure.Regulation of Vascular Calcification by Growth Hormone-Releasing Hormone and its Agonists.Growth hormone-releasing hormone (GHRH) and its agonists inhibit hepatic and tumoral secretion of IGF-1.

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Activation of growth hormone releasing hormone (GHRH) receptor stimulates cardiac reverse remodeling after myocardial infarction (MI)

http://www.wikidata.org/entity/Q35673689

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Activation of growth hormone r ...... ter myocardial infarction (MI)

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Activation of growth hormone r ...... ter myocardial infarction (MI)

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Proceedings of the National Academy of Sciences of the United States of America

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Activation of growth hormone r ...... ter myocardial infarction (MI)

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Adriana V Treuer

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Claudia O Rodrigues

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Daniel R Gonzalez

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Ellena C Paulino

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Joshua M Hare

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Lauro M Takeuchi

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Norman L Block

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Raul Dulce

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Rosemeire M Kanashiro-Takeuchi

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Sarah M Selem

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P2860

Expression and function of c-kit in hemopoietic progenitor cells

Role of cardiac stem cells in cardiac pathophysiology: a paradigm shift in human myocardial biology

c-Kit--a hematopoietic cell essential receptor tyrosine kinase

Cardiomyocyte GATA4 functions as a stress-responsive regulator of angiogenesis in the murine heart

Identification and functionality of proteomes secreted by rat cardiac stem cells and neonatal cardiomyocytes

Growth hormone-releasing hormone promotes survival of cardiac myocytes in vitro and protects against ischaemia-reperfusion injury in rat heart

Cardioprotective effects of growth hormone-releasing hormone agonist after myocardial infarction

Paracrine mechanisms in adult stem cell signaling and therapy

Bone marrow mesenchymal stem cells stimulate cardiac stem cell proliferation and differentiation

Sex-specific impact of aldosterone receptor antagonism on ventricular remodeling and gene expression after myocardial infarction.

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2

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Konstantinos E Hatzistergos

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2011-12-27T00:00:00Z

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51954354

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