Positive feedback between oncogenic KRAS and HIF-1α confers drug resistance in colorectal cancer

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Mechanisms underlying 18F-fluorodeoxyglucose accumulation in colorectal cancer Inhibition of Hif1α prevents both trauma-induced and genetic heterotopic ossification

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Positive feedback between oncogenic KRAS and HIF-1α confers drug resistance in colorectal cancer

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Positive feedback between onco ...... esistance in colorectal cancer

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Fuming Lei

http://www.w3.org/2001/XMLSchema#string

Qingmin Zeng

http://www.w3.org/2001/XMLSchema#string

Wanshui Rong

http://www.w3.org/2001/XMLSchema#string

Wenbing Sun

http://www.w3.org/2001/XMLSchema#string

Yanzhao Wang

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Markers of resistance to anti-EGFR therapy in colorectal cancer

Mechanism of regulation of the hypoxia-inducible factor-1 alpha by the von Hippel-Lindau tumor suppressor protein

EGFR and downstream genetic alterations in KRAS/BRAF and PI3K/AKT pathways in colorectal cancer: implications for targeted therapy

K-Ras(G12C) inhibitors allosterically control GTP affinity and effector interactions

KRAS mutation status is predictive of response to cetuximab therapy in colorectal cancer.

Effect of KRAS oncogene substitutions on protein behavior: implications for signaling and clinical outcome

Colorectal cancer incidence and mortality in Texas 1990-1992: a comparison of rural classifications.

Mammalian target of rapamycin regulates murine and human cell differentiation through STAT3/p63/Jagged/Notch cascade

Hypoxia activates the K-ras proto-oncogene to stimulate angiogenesis and inhibit apoptosis in colon cancer cells.

KRAS G13D Mutation and Sensitivity to Cetuximab or Panitumumab in a Colorectal Cancer Cell Line Model.

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1229-1237

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scholarly article

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10.2147/OTT.S80017

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English

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2015-05-26T00:00:00Z

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277961948

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26060408

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colorectal cancer

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4454194

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Positive feedback between oncogenic KRAS and HIF-1α confers drug resistance in colorectal cancer

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P2860

Positive feedback between oncogenic KRAS and HIF-1α confers drug resistance in colorectal cancer

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