Mitochondrial dysfunction induced by a post-translationally modified amyloid linked to a familial mutation in an alternative model of neurodegeneration.
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Potentiation of neurotoxicity in double-mutant mice with Pink1 ablation and A53T-SNCA overexpression.Cytosolic Extract of Human Adipose Stem Cells Reverses the Amyloid Beta-Induced Mitochondrial Apoptosis via P53/Foxo3a Pathway.Oxidative stress and mitochondria-mediated cell death mechanisms triggered by the familial Danish dementia ADan amyloidAβ truncated species: Implications for brain clearance mechanisms and amyloid plaque deposition.
P2860
Mitochondrial dysfunction induced by a post-translationally modified amyloid linked to a familial mutation in an alternative model of neurodegeneration.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@ast
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@en
type
label
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@ast
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@en
prefLabel
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@ast
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@en
P2860
P1476
Mitochondrial dysfunction indu ...... ve model of neurodegeneration.
@en
P2093
Jorge Ghiso
Krysti Todd
P2860
P304
P356
10.1016/J.BBADIS.2014.09.010
P407
P433
P577
2014-09-28T00:00:00Z