Reduced CD36-dependent tissue sequestration of Plasmodium-infected erythrocytes is detrimental to malaria parasite growth in vivo.
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Liver accumulation of Plasmodium chabaudi-infected red blood cells and modulation of regulatory T cell and dendritic cell responsesIn vivo and in vitro characterization of a Plasmodium liver stage-specific promoterCD36 and Fyn kinase mediate malaria-induced lung endothelial barrier dysfunction in mice infected with Plasmodium bergheiThe machinery underlying malaria parasite virulence is conserved between rodent and human malaria parasitesProteome mapping of Plasmodium: identification of the P. yoelii remodellomeThe Plasmodium translocon of exported proteins (PTEX) component thioredoxin-2 is important for maintaining normal blood-stage growthThe Plasmodium PHIST and RESA-Like Protein Families of Human and Rodent Malaria ParasitesGlucagon-like peptide-1 analogue, liraglutide, in experimental cerebral malaria: implications for the role of oxidative stress in cerebral malariaProteomic and genetic analyses demonstrate that Plasmodium berghei blood stages export a large and diverse repertoire of proteinsHost erythrocyte environment influences the localization of exported protein 2, an essential component of the Plasmodium translocon.Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous VacuoleImaging Plasmodium immunobiology in the liver, brain, and lung.Effect of mature blood-stage Plasmodium parasite sequestration on pathogen biomass in mathematical and in vivo models of malaria.ICAM-1 is a key receptor mediating cytoadherence and pathology in the Plasmodium chabaudi malaria modelIron overload in Plasmodium berghei-infected placenta as a pathogenesis mechanism of fetal death.Liver-inherent immune system: its role in blood-stage malaria.Sequestration and histopathology in Plasmodium chabaudi malaria are influenced by the immune response in an organ-specific mannerVariant surface antigens of malaria parasites: functional and evolutionary insights from comparative gene family classification and analysis.CD36 recruits α₅β₁ integrin to promote cytoadherence of P. falciparum-infected erythrocytes.In Vivo Function of PTEX88 in Malaria Parasite Sequestration and Virulence.The exported Plasmodium berghei protein IBIS1 delineates membranous structures in infected red blood cells.Plasmodium berghei bio-burden correlates with parasite lactate dehydrogenase: application to murine Plasmodium diagnostics.Where have all the parasites gone? Modelling early malaria parasite sequestration dynamicsCharacterization of the Plasmodium Interspersed Repeats (PIR) proteins of Plasmodium chabaudi indicates functional diversityThe utility of Plasmodium berghei as a rodent model for anti-merozoite malaria vaccine assessment.An ultrasensitive NanoLuc-based luminescence system for monitoring Plasmodium berghei throughout its life cycleMurine Model for Preclinical Studies of Var2CSA-Mediated Pathology Associated with Malaria in PregnancyMultidrug ATP-binding cassette transporters are essential for hepatic development of Plasmodium sporozoites.Cytoadherence of Plasmodium berghei-infected red blood cells to murine brain and lung microvascular endothelial cells in vitro.Stuck in a rut? Reconsidering the role of parasite sequestration in severe malaria syndromes.Phylogenetic profiles of all membrane transport proteins of the malaria parasite highlight new drug targetsReduced erythrocyte susceptibility and increased host clearance of young parasites slows Plasmodium growth in a murine model of severe malaria.Immunobiology of Plasmodium in liver and brain.The immunological balance between host and parasite in malaria.Signaling Strategies of Malaria Parasite for Its Survival, Proliferation, and Infection during Erythrocytic Stage.Antecedent Nippostrongylus infection alters the lung immune response to Plasmodium berghei.CD36 receptor regulates malaria-induced immune responses primarily at early blood stage infection contributing to parasitemia control and resistance to mortality.Ultrastructure of the lung in a murine model of malaria-associated acute lung injury/acute respiratory distress syndrome.Marked variation in prevalence of malaria-protective human genetic polymorphisms across Uganda.Improved methods for haemozoin quantification in tissues yield organ-and parasite-specific information in malaria-infected mice.
P2860
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P2860
Reduced CD36-dependent tissue sequestration of Plasmodium-infected erythrocytes is detrimental to malaria parasite growth in vivo.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@ast
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@en
type
label
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@ast
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@en
prefLabel
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@ast
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@en
P2093
P2860
P50
P921
P356
P1476
Reduced CD36-dependent tissue ...... laria parasite growth in vivo.
@en
P2093
Alan W Thomas
Clemens H M Kocken
Erica M Pasini
Irene O C M Vroegrijk
Jai Ramesar
Joanna A M Braks
Matthias Mann
Sjoerd G van Duinen
P2860
P304
P356
10.1084/JEM.20110762
P407
P577
2011-12-19T00:00:00Z