Methamphetamine-induced changes in the object recognition memory circuit. - Wikidata - wikimedia - TriplyDB

Methamphetamine-induced changes in the object recognition memory circuit.

Methamphetamine-induced changes in the object recognition memory circuit.

http://www.wikidata.org/entity/Q35685517

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SN79, a sigma receptor antagonist, attenuates methamphetamine-induced astrogliosis through a blockade of OSMR/gp130 signaling and STAT3 phosphorylation Serotonin, neural markers, and memory Nucleus accumbens shell excitability is decreased by methamphetamine self-administration and increased by 5-HT2C receptor inverse agonism and agonism.Cognitive deficits associated with combined HIV gp120 expression and chronic methamphetamine exposure in mice.The effects of methamphetamine self-administration on cortical monoaminergic deficits induced by subsequent high-dose methamphetamine administrations Extinction-dependent alterations in corticostriatal mGluR2/3 and mGluR7 receptors following chronic methamphetamine self-administration in rats Sex differences in escalation of methamphetamine self-administration: cognitive and motivational consequences in rats.Perirhinal Cortex mGlu5 Receptor Activation Reduces Relapse to Methamphetamine Seeking by Restoring Novelty Salience.Chronic methamphetamine abuse and corticostriatal deficits revealed by neuroimaging.Selective, retrieval-independent disruption of methamphetamine-associated memory by actin depolymerization.Estrogen and Progesterone Replacement Therapy Prevent Methamphetamine-Induced Synaptic Plasticity Impairment in Ovariectomized Rats.Long-term effects of exposure to methamphetamine in adolescent rats Methamphetamine self-administration attenuates hippocampal serotonergic deficits: role of brain-derived neurotrophic factor.Methamphetamine influences on brain and behavior: unsafe at any speed?Drugs of abuse and increased risk of psychosis development.Transcriptional and epigenetic substrates of methamphetamine addiction and withdrawal: evidence from a long-access self-administration model in the rat.Object-in-place associative recognition memory depends on glutamate receptor neurotransmission within two defined hippocampal-cortical circuits: a critical role for AMPA and NMDA receptors in the hippocampus, perirhinal, and prefrontal cortices.Failure to Recognize Novelty after Extended Methamphetamine Self-Administration Results from Loss of Long-Term Depression in the Perirhinal Cortex Nicotine Administration Attenuates Methamphetamine-Induced Novel Object Recognition Deficits.Prior methamphetamine self-administration attenuates serotonergic deficits induced by subsequent high-dose methamphetamine administrations.Immediate and lasting effects of chronic daily methamphetamine exposure on activation of cells in hypothalamic-pituitary-adrenal axis-associated brain regions.Cocaine and methamphetamine induce opposing changes in BOLD signal response in rats.Modafinil improves methamphetamine-induced object recognition deficits and restores prefrontal cortex ERK signaling in mice Chronic administration of the methylxanthine propentofylline impairs reinstatement to cocaine by a GLT-1-dependent mechanism.Modafinil restores methamphetamine induced object-in-place memory deficits in rats independent of glutamate N-methyl-D-aspartate receptor expression.Methamphetamine addiction: involvement of CREB and neuroinflammatory signaling pathways.Chronic methamphetamine self-administration disrupts cortical control of cognition.Current understanding of methamphetamine-associated dopaminergic neurodegeneration and psychotoxic behaviors.Methamphetamine decreases CD4 T cell frequency and alters pro-inflammatory cytokine production in a model of drug abuse.Modeling human methamphetamine use patterns in mice: chronic and binge methamphetamine exposure, reward function and neurochemistry.The evaluation of AZ66, an optimized sigma receptor antagonist, against methamphetamine-induced dopaminergic neurotoxicity and memory impairment in mice.Sensitivity to Chronic Methamphetamine Administration and Withdrawal in Mice with Relaxin-3/RXFP3 Deficiency.Effects of chronic administration of fenproporex on cognitive and non-cognitive behaviors.Intranasal insulin treatment restores cognitive deficits and insulin signaling impairment induced by repeated methamphetamine exposure.Melatonin attenuates methamphetamine-induced inhibition of proliferation of adult rat hippocampal progenitor cells in vitro.Methamphetamine-induced changes in the mice hippocampal neuropeptide Y system: implications for memory impairment.A Single High Dose of Methamphetamine Reduces Monoamines and Impairs Egocentric and Allocentric Learning and Memory in Adult Male Rats.Neurocognitive dysfunction following repeated binge-like self-administration of the synthetic cathinone 3,4-methylenedioxypyrovalerone (MDPV).

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P2860

Methamphetamine-induced changes in the object recognition memory circuit.

http://www.wikidata.org/entity/Q35685517

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Methamphetamine-induced changes in the object recognition memory circuit.

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Methamphetamine-induced changes in the object recognition memory circuit.

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Methamphetamine-induced changes in the object recognition memory circuit.

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Methamphetamine-induced changes in the object recognition memory circuit.

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Methamphetamine-induced changes in the object recognition memory circuit.

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J.NEUROPHARM.2011.11.003

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Neuropharmacology

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Methamphetamine-induced changes in the object recognition memory circuit

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Jacqueline F McGinty

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Lauren A Ramsey

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Marek Schwendt

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Ronald E See

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P2860

The episodic memory system: neurocircuitry and disorders

Adrenergic pharmacology and cognition: focus on the prefrontal cortex

Time-course of methamphetamine-induced neurotoxicity in rat caudate-putamen after single-dose treatment

Methamphetamine self-administration is associated with persistent biochemical alterations in striatal and cortical dopaminergic terminals in the rat

Extended access to methamphetamine self-administration affects sensorimotor gating in rats.

Varied access to intravenous methamphetamine self-administration differentially alters adult hippocampal neurogenesis.

Loss of object recognition memory produced by extended access to methamphetamine self-administration is reversed by positive allosteric modulation of metabotropic glutamate receptor 5

Methamphetamine self-administration produces attentional set-shifting deficits and alters prefrontal cortical neurophysiology in rats.

Structural and metabolic brain changes in the striatum associated with methamphetamine abuse.

Association of dopamine transporter reduction with psychomotor impairment in methamphetamine abusers.

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1119-1126

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scholarly article

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10.1016/J.NEUROPHARM.2011.11.003

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2

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62

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Carmela M Reichel

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2011-11-18T00:00:00Z

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51829246

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3262904

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