Kgp and RgpB, but not RgpA, are important for Porphyromonas gingivalis virulence in the murine periodontitis model.
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Reversible redox regulation of specificity of Arg-gingipain B in Porphyromonas gingivalisLow-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complementProtease-activated receptor 2 has pivotal roles in cellular mechanisms involved in experimental periodontitisStructural Insights into the PorK and PorN Components of the Porphyromonas gingivalis Type IX Secretion SystemPorphyromonas gingivalis Gingipains Trigger a Proinflammatory Response in Human Monocyte-derived Macrophages Through the p38α Mitogen-activated Protein Kinase Signal Transduction PathwayStructural insights unravel the zymogenic mechanism of the virulence factor gingipain K from Porphyromonas gingivalis, a causative agent of gum disease from the human oral microbiome.The core genome of the anaerobic oral pathogenic bacterium Porphyromonas gingivalis.Inactive Gingipains from P. gingivalis Selectively Skews T Cells toward a Th17 Phenotype in an IL-6 Dependent MannerVirulence factors of the oral spirochete Treponema denticola.Tetratricopeptide repeat protein-associated proteins contribute to the virulence of Porphyromonas gingivalis.Porphyromonas gingivalis infection increases osteoclastic bone resorption and osteoblastic bone formation in a periodontitis mouse model.Porphyromonas gingivalis-derived lysine gingipain enhances osteoclast differentiation induced by tumor necrosis factor-α and interleukin-1β but suppresses that by interleukin-17A: importance of proteolytic degradation of osteoprotegerin by lysine giDichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins.Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in ratsLysine-specific gingipain promotes lipopolysaccharide- and active-vitamin D3-induced osteoclast differentiation by degrading osteoprotegerinStructure and mechanism of cysteine peptidase gingipain K (Kgp), a major virulence factor of Porphyromonas gingivalis in periodontitis.Propeptide-mediated inhibition of cognate gingipain proteinases.Porphyromonas gingivalis-derived RgpA-Kgp Complex Activates the Macrophage Urokinase Plasminogen Activator System: IMPLICATIONS FOR PERIODONTITISFOXO responses to Porphyromonas gingivalis in epithelial cells.Involvement of an Skp-Like Protein, PGN_0300, in the Type IX Secretion System of Porphyromonas gingivalis.Cleavage of IgG1 in gingival crevicular fluid is associated with the presence of Porphyromonas gingivalisThe use of rodent models to investigate host-bacteria interactions related to periodontal diseases.Porphyromonas gingivalis RgpA-Kgp proteinase-adhesin complexes penetrate gingival tissue and induce proinflammatory cytokines or apoptosis in a concentration-dependent manner.Mucosal Langerhans Cells Promote Differentiation of Th17 Cells in a Murine Model of Periodontitis but Are Not Required for Porphyromonas gingivalis-Driven Alveolar Bone DestructionPorphyromonas gulae Has Virulence and Immunological Characteristics Similar to Those of the Human Periodontal Pathogen Porphyromonas gingivalis.Oxantel disrupts polymicrobial biofilm development of periodontal pathogens.Strategies for the inhibition of gingipains for the potential treatment of periodontitis and associated systemic diseases.Determination of Active Phagocytosis of Unopsonized Porphyromonas gingivalis by Macrophages and Neutrophils Using the pH-Sensitive Fluorescent Dye pHrodo.Streptococcus cristatus ArcA interferes with Porphyromonas gingivalis pathogenicity in mice.Cleavage of protease-activated receptors on an immortalized oral epithelial cell line by Porphyromonas gingivalis gingipains.GM-CSF and uPA are required for Porphyromonas gingivalis-induced alveolar bone loss in a mouse periodontitis model.Structure of the lysine specific protease Kgp from Porphyromonas gingivalis, a target for improved oral healthMetabolic crosstalk regulates Porphyromonas gingivalis colonization and virulence during oral polymicrobial infection.The role of RgpA in the pathogenicity of Porphyromonas gingivalis in the murine periodontitis model.Genes Contributing to Porphyromonas gingivalis Fitness in Abscess and Epithelial Cell Colonization Environments.A therapeutic Porphyromonas gingivalis gingipain vaccine induces neutralising IgG1 antibodies that protect against experimental periodontitis.Molecular mechanisms of Porphyromonas gingivalis-host cell interaction on periodontal diseases.Predominant bacteria recovered from a periodontitis site in a hamster model raised by silk-ligature with Porphyromonas gingivalis infection.kgp, rgpA, and rgpB DNA vaccines induce antibody responses in experimental peri-implantitis.
P2860
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P2860
Kgp and RgpB, but not RgpA, are important for Porphyromonas gingivalis virulence in the murine periodontitis model.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@ast
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@en
type
label
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@ast
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@en
prefLabel
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@ast
Kgp and RgpB, but not RgpA, ar ...... he murine periodontitis model.
@en
P2093
P2860
P356
P1476
Kgp and RgpB, but not RgpA, ar ...... the murine periodontitis model
@en
P2093
Gail C Brammar
Nada Slakeski
Rishi D Pathirana
P2860
P304
P356
10.1128/IAI.01627-06
P407
P577
2007-01-12T00:00:00Z