Residual Disease in a Novel Xenograft Model of RUNX1-Mutated, Cytogenetically Normal Acute Myeloid Leukemia

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Residual Disease in a Novel Xenograft Model of RUNX1-Mutated, Cytogenetically Normal Acute Myeloid Leukemia

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http://www.wikidata.org/entity/Q35692708

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2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

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2015年论文

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2015年论文

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name

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

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Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

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Item

label

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

@ast

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

@en

prefLabel

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

@ast

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

@en

P1476

Residual Disease in a Novel Xe ...... Normal Acute Myeloid Leukemia

@en

P2093

Allison Eberhardt

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Jason R Myers

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Marlene Balys

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Michael W Becker

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Nancy Wang

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Umayal Sivagnanalingam

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P2860

RUNX1 mutations in acute myeloid leukemia: results from a comprehensive genetic and clinical analysis from the AML study group.

ASXL1 mutations identify a high-risk subgroup of older patients with primary cytogenetically normal AML within the ELN Favorable genetic category.

Prognostic relevance of integrated genetic profiling in acute myeloid leukemia.

RUNX1 mutations are associated with poor outcome in younger and older patients with cytogenetically normal acute myeloid leukemia and with distinct gene and MicroRNA expression signatures.

Modeling the initiation and progression of human acute leukemia in mice

Genomic and epigenomic landscapes of adult de novo acute myeloid leukemia.

RUNX1 mutations in cytogenetically normal acute myeloid leukemia are associated with a poor prognosis and up-regulation of lymphoid genes.

AML cells are differentially sensitive to chemotherapy treatment in a human xenograft model.

Environment-mediated drug resistance: a major contributor to minimal residual disease.

Reciprocal leukemia-stroma VCAM-1/VLA-4-dependent activation of NF-κB mediates chemoresistance.

P304

e0132375

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P31

scholarly article

P356

10.1371/JOURNAL.PONE.0132375

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Laura Maria Calvi

Jason Mendler

John M Ashton

P577

2015-07-15T00:00:00Z

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P5875

281822790

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P698

26177509

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P921

xenograft

P932

4503761

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