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Therapeutic potential of mGluR5 targeting in Alzheimer's disease

Therapeutic potential of mGluR5 targeting in Alzheimer's disease

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A review on mitochondrial restorative mechanism of antioxidants in Alzheimer's disease and other neurological conditions Amyloid-β(25-35) Modulates the Expression of GirK and KCNQ Channel Genes in the Hippocampus.Aβ and Inflammatory Stimulus Activate Diverse Signaling Pathways in Monocytic Cells: Implications in Retaining Phagocytosis in Aβ-Laden Environment Hippocampal metabotropic glutamate receptor long-term depression in health and disease: focus on mitogen-activated protein kinase pathways.Glutamate levels control HT22 murine hippocampal cell death by regulating biphasic patterns of Erk1/2 activation: role of metabolic glutamate receptor 5.Autoantibodies to Synaptic Receptors and Neuronal Cell Surface Proteins in Autoimmune Diseases of the Central Nervous System.Astrocyte dysfunction in Alzheimer disease.The frontal cortex as a network hub controlling mood and cognition: Probing its neurochemical substrates for improved therapy of psychiatric and neurological disorders.Mdm2 mediates FMRP- and Gp1 mGluR-dependent protein translation and neural network activity.Roles for Arc in metabotropic glutamate receptor-dependent LTD and synapse elimination: Implications in health and disease.Molecular Mechanisms of Lithium Action: Switching the Light on Multiple Targets for Dementia Using Animal Models

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Therapeutic potential of mGluR5 targeting in Alzheimer's disease

http://www.wikidata.org/entity/Q35698522

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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Therapeutic potential of mGluR5 targeting in Alzheimer's disease

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FNINS.2015.00215

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Frontiers in Neuroscience

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Therapeutic potential of mGluR5 targeting in Alzheimer's disease

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Anil Kumar

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Dinesh K Dhull

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P2860

Cellular prion protein mediates impairment of synaptic plasticity by amyloid-beta oligomers

Alzheimer's disease and Down's syndrome: Treating two paths to dementia

Metabotropic glutamate receptor 5 is a coreceptor for Alzheimer aβ oligomer bound to cellular prion protein

Autistic-like social behaviour in Shank2-mutant mice improved by restoring NMDA receptor function

Synthetic amyloid-beta oligomers impair long-term memory independently of cellular prion protein

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Selective mGluR5 antagonists MPEP and SIB-1893 decrease NMDA or glutamate-mediated neuronal toxicity through actions that reflect NMDA receptor antagonism

From development to dysfunction: microglia and the complement cascade in CNS homeostasis

M1 and M2 immune activation in Parkinson's Disease: Foe and ally?

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

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10.3389/FNINS.2015.00215

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Alzheimer's disease

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