Effects of α-2A adrenergic receptor agonist on time and risk preference in primates - Wikidata - wikimedia - TriplyDB

Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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The Potential for Misuse and Abuse of Medications in ADHD: A Review Neuromodulation of thought: flexibilities and vulnerabilities in prefrontal cortical network synapses Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation Reducing substance use during adolescence: a translational framework for prevention Targeting Prefrontal Cortical Systems for Drug Development: Potential Therapies for Cognitive Disorders A computational psychiatry approach identifies how alpha-2A noradrenergic agonist Guanfacine affects feature-based reinforcement learning in the macaque.Impaired flexibility in decision making in rats after administration of the pharmacological stressor yohimbine.The neural bases of cognitive processes in gambling disorder.A translational investigation targeting stress-reactivity and prefrontal cognitive control with guanfacine for smoking cessation Economic choices reveal probability distortion in macaque monkeys Alpha 2A adrenergic receptor agonist, guanfacine, attenuates cocaine-related impairments of inhibitory response control and working memory in animal models.Guanfacine for the treatment of cognitive disorders: a century of discoveries at Yale Pregabalin- and topiramate-mediated regulation of cognitive and motor impulsivity in DBA/2 mice.Guanfacine Extended Release: A New Pharmacological Treatment Option in Europe.Decision making: from neuroscience to psychiatry Guanfacine extended release for the treatment of attention-deficit/hyperactivity disorder in children and adolescents.Noradrenergic modulation of risk/reward decision making.Dopamine reward prediction error responses reflect marginal utility.From habits to self-regulation: how do we change?The effect of cognitive challenge on delay discounting.Noradrenergic α2A-receptor stimulation in the ventral hippocampus reduces impulsive decision-making.α2A -Adrenergic receptor polymorphisms and mRNA expression levels are associated with delay discounting in cocaine users.Inactivation of Medial Frontal Cortex Changes Risk Preference

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

http://www.wikidata.org/entity/Q35721180

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Psychopharmacology

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Effects of α-2A adrenergic receptor agonist on time and risk preference in primates

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Amy F T Arnsten

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Irina Bobeica

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Nao J Gamo

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P2860

Dopamine in motivational control: rewarding, aversive, and alerting

Stress signalling pathways that impair prefrontal cortex structure and function

The neuropsychopharmacology of fronto-executive function: monoaminergic modulation

The use of α-2A adrenergic agonists for the treatment of attention-deficit/hyperactivity disorder

Two types of dopamine neuron distinctly convey positive and negative motivational signals

Alpha2A-adrenoceptor stimulation improves prefrontal cortical regulation of behavior through inhibition of cAMP signaling in aging animals

State-dependent decisions cause apparent violations of rationality in animal choice

Prospect Theory: An Analysis of Decision under Risk

An integrative theory of locus coeruleus-norepinephrine function: adaptive gain and optimal performance

Predictive reward signal of dopamine neurons

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