Genetic progression and divergence in pancreatic carcinoma - Wikidata - wikimedia - TriplyDB

Genetic progression and divergence in pancreatic carcinoma

Genetic progression and divergence in pancreatic carcinoma

http://www.wikidata.org/entity/Q35745480

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Palladin mutation causes familial pancreatic cancer and suggests a new cancer mechanism HIN-1, a putative cytokine highly expressed in normal but not cancerous mammary epithelial cells What is the origin of pancreatic adenocarcinoma?Targeted destruction of the orchestration of the pancreatic stroma and tumor cells in pancreatic cancer cases: molecular basis for therapeutic implications Whole-exome sequencing of human pancreatic cancers and characterization of genomic instability caused by MLH1 haploinsufficiency and complete deficiency Exdpf is a key regulator of exocrine pancreas development controlled by retinoic acid and ptf1a in zebrafish.KLF4 is a novel candidate tumor suppressor gene in pancreatic ductal carcinoma.Precursor lesions of pancreatic cancer.Genetic progression in the pancreatic ducts.Utilization of ancillary studies in the cytologic diagnosis of biliary and pancreatic lesions: The Papanicolaou Society of Cytopathology Guidelines.Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma.Update on pancreatic intraepithelial neoplasia.Loss of p16INK4a results in increased glucocorticoid receptor activity during fibrosarcoma development.Pathological appraisal of lines of resection for bile duct carcinoma.Utilization of ancillary studies in the cytologic diagnosis of biliary and pancreatic lesions: the Papanicolaou Society of Cytopathology guidelines for pancreatobiliary cytology Pancreatic cancer stromal biology and therapy.The pathology of ductal-type pancreatic carcinomas and pancreatic intraepithelial neoplasia: insights for clinicians.Allelic loss is often the first hit in the biallelic inactivation of the p53 and DPC4 genes during pancreatic carcinogenesis Loss of heterozygosity or intragenic mutation, which comes first?Advances in biomarker research for pancreatic cancer.Molecular genetics of pancreatic ductal adenocarcinomas and recent implications for translational efforts.Stromal biology of pancreatic cancer.Molecular genetics of pancreatic intraepithelial neoplasia.Genetic alterations in pancreatic cancer.Epigenetics and epigenetic alterations in pancreatic cancer.Emerging molecular biology of pancreatic cancer.The molecular biology of pancreatic cancer.Transcriptomic and CRISPR/Cas9 technologies reveal FOXA2 as a tumor suppressor gene in pancreatic cancer.Loss of heterozygosity and microsatellite instability as predictive markers among Iranian esophageal cancer patients.Microsatellite alterations in phenotypically normal esophageal squamous epithelium and metaplasia-dysplasia-adenocarcinoma sequence.In vitro models of pancreatic cancer for translational oncology research.Pancreatic intraepithelial neoplasia-can we detect early pancreatic cancer?Enhancer Reprogramming Promotes Pancreatic Cancer Metastasis.Two cases of pancreatic ductal adenocarcinoma with intrapancreatic metastasis.The fatal attraction between pro-prion and filamin A: prion as a marker in human cancers.Transcriptional regulation of urokinase-type plasminogen activator receptor by hypoxia-inducible factor 1 is crucial for invasion of pancreatic and liver cancer.Loss of heterozygosity predicts poor survival after resection of pancreatic adenocarcinoma.Frequency of K-ras mutations in pancreatic intraductal neoplasias associated with pancreatic ductal adenocarcinoma and chronic pancreatitis: a meta-analysis Risk of pancreatic ductal adenocarcinoma in chronic pancreatitis.Oncogenic KRAS activates hedgehog signaling pathway in pancreatic cancer cells.

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P2860

Genetic progression and divergence in pancreatic carcinoma

http://www.wikidata.org/entity/Q35745480

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name

Genetic progression and divergence in pancreatic carcinoma

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Genetic progression and divergence in pancreatic carcinoma

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Genetic progression and divergence in pancreatic carcinoma

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Genetic progression and divergence in pancreatic carcinoma

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Genetic progression and divergence in pancreatic carcinoma

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Genetic progression and divergence in pancreatic carcinoma

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The American Journal of Pathology

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Genetic progression and divergence in pancreatic carcinoma

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P2093

H Fujii

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H Watanabe

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M Yamano

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N Kadowaki

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T Shirai

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T Takagaki

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P2860

Human mitogen-activated protein kinase kinase 4 as a candidate tumor suppressor

Alterations in pancreatic, biliary, and breast carcinomas support MKK4 as a genetically targeted tumor suppressor gene

Frequent mutations of CDKN2 in primary pancreatic adenocarcinomas

Genetic alterations during colorectal-tumor development

Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinoma

Most human carcinomas of the exocrine pancreas contain mutant c-K-ras genes

DPC4, a candidate tumor suppressor gene at human chromosome 18q21.1

Pathology of incipient pancreatic cancer.

p53 mutations in pancreatic carcinoma and evidence of common involvement of homocopolymer tracts in DNA microdeletions.

Tumor-suppressive pathways in pancreatic carcinoma.

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