Targeting PBK/TOPK decreases growth and survival of glioma initiating cells in vitro and attenuates tumor growth in vivo - Wikidata - wikimedia - TriplyDB

Targeting PBK/TOPK decreases growth and survival of glioma initiating cells in vitro and attenuates tumor growth in vivo

Targeting PBK/TOPK decreases growth and survival of glioma initiating cells in vitro and attenuates tumor growth in vivo

http://www.wikidata.org/entity/Q35750188

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Brain tissue banking for stem cells for our future Knockdown of NAT12/NAA30 reduces tumorigenic features of glioblastoma-initiating cells Combined expressional analysis, bioinformatics and targeted proteomics identify new potential therapeutic targets in glioblastoma stem cells PDZ binding kinase (PBK) is a theranostic target for nasopharyngeal carcinoma: driving tumor growth via ROS signaling and correlating with patient survival Radiation-inducible miR-770-5p sensitizes tumors to radiation through direct targeting of PDZ-binding kinase.Identification of potential key genes associated with glioblastoma based on the gene expression profile Overexpression of PBK/TOPK relates to tumour malignant potential and poor outcome of gastric carcinoma.PDZ binding kinase, regulated by FoxM1, enhances malignant phenotype via activation of β-Catenin signaling in hepatocellular carcinoma.Ginsenoside Rh2 inhibiting HCT116 colon cancer cell proliferation through blocking PDZ-binding kinase/T-LAK cell-originated protein kinase.MYCN induces neuroblastoma in primary neural crest cells.Preoperative imaging markers and PDZ-binding kinase tissue expression predict low-risk disease in endometrial hyperplasias and low grade cancers Impact of a novel biomarker, T-LAK cell-originating protein kinase (TOPK) expression on outcome in malignant glioma.Targeting the T-Lak cell originated protein kinase by OTS964 shrinks the size of power-law coded heterogeneous glioma stem cell populations.Current Challenges and Opportunities in Treating Glioblastoma.T-lymphokine-activated killer cell-originated protein kinase (TOPK) as a prognostic factor and a potential therapeutic target in glioma.Enhanced Expression of the Key Mitosis Regulator Cyclin B1 Is Mediated by PDZ-Binding Kinase in Islets of Pregnant Mice.MicroRNA-216b-3p inhibits lung adenocarcinoma cell growth via regulating PDZ binding kinase/T-LAK-cell-originated protein kinase.Patterns of Invasive Growth in Malignant Gliomas-The Hippocampus Emerges as an Invasion-Spared Brain Region.

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Targeting PBK/TOPK decreases growth and survival of glioma initiating cells in vitro and attenuates tumor growth in vivo

http://www.wikidata.org/entity/Q35750188

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Molecular Cancer

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Targeting PBK/TOPK decreases g ...... ttenuates tumor growth in vivo

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Awais A Mughal

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Biljana Stangeland

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Birthe Mikkelsen

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Cecilie J Sandberg

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Hege B Fjerdingstad

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Jinan Behnan

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Joel C Glover

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Mrinal Joel

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Sheryl Palmero

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Wayne Murrell

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WikiPathways: pathway editing for the people

Gene ontology: tool for the unification of biology

PBK/TOPK interacts with the DBD domain of tumor suppressor p53 and modulates expression of transcriptional targets including p21

WebGestalt: an integrated system for exploring gene sets in various biological contexts.

Gene Expression Omnibus: NCBI gene expression and hybridization array data repository

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

The KEGG resource for deciphering the genome

Stem cells, cancer, and cancer stem cells

Identification of human brain tumour initiating cells

Identification of a cancer stem cell in human brain tumors

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Iver Arne Langmoen

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