Adaptation of the Brucellae to their intracellular niche. - Wikidata - wikimedia - TriplyDB

Adaptation of the Brucellae to their intracellular niche.

Adaptation of the Brucellae to their intracellular niche.

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Whole-Genome Analyses of Speciation Events in Pathogenic Brucellae The complex microbiota of raw milk An atypical riboflavin pathway is essential for Brucella abortus virulence PHIDIAS: a pathogen-host interaction data integration and analysis system.Systematic targeted mutagenesis of Brucella melitensis 16M reveals a major role for GntR regulators in the control of virulence.The pea nodule environment restores the ability of a Rhizobium leguminosarum lipopolysaccharide acpXL mutant to add 27-hydroxyoctacosanoic acid to its lipid A.BBP: Brucella genome annotation with literature mining and curation.Mariner mutagenesis of Brucella melitensis reveals genes with previously uncharacterized roles in virulence and survival.Brucella control of dendritic cell maturation is dependent on the TIR-containing protein Btp1.Enzymatic, immunological and phylogenetic characterization of Brucella suis urease.Complete genome of Phenylobacterium zucineum--a novel facultative intracellular bacterium isolated from human erythroleukemia cell line K562.Brucella abortus induces the secretion of proinflammatory mediators from glial cells leading to astrocyte apoptosis The Brucella abortus Cu,Zn superoxide dismutase is required for optimal resistance to oxidative killing by murine macrophages and wild-type virulence in experimentally infected mice.Requirement of MgtC for Brucella suis intramacrophage growth: a potential mechanism shared by Salmonella enterica and Mycobacterium tuberculosis for adaptation to a low-Mg2+ environment.Opsonized virulent Brucella abortus replicates within nonacidic, endoplasmic reticulum-negative, LAMP-1-positive phagosomes in human monocytes Global analysis of quorum sensing targets in the intracellular pathogen Brucella melitensis 16 M Human brucellosis is characterized by an intense Th1 profile associated with a defective monocyte function.The Brucella suis type IV secretion system assembles in the cell envelope of the heterologous host Agrobacterium tumefaciens and increases IncQ plasmid pLS1 recipient competence The Brucella abortus xthA-1 gene product participates in base excision repair and resistance to oxidative killing but is not required for wild-type virulence in the mouse model Comparative analysis of the early transcriptome of Brucella abortus--infected monocyte-derived macrophages from cattle naturally resistant or susceptible to brucellosis Characterization and protective property of Brucella abortus cydC and looP mutants.Transcriptional profile of the intracellular pathogen Brucella melitensis following HeLa cells infection.Brucellosis Ontology (IDOBRU) as an extension of the Infectious Disease Ontology.Nanomedicine as an emerging approach against intracellular pathogens The TolC homologue of Brucella suis is involved in resistance to antimicrobial compounds and virulence.Brucella abortus requires the heme transporter BhuA for maintenance of chronic infection in BALB/c mice.Pathogen-endoplasmic-reticulum interactions: in through the out door.Intracellularly induced cyclophilins play an important role in stress adaptation and virulence of Brucella abortus.Metabolic control of cell division in α-proteobacteria by a NAD-dependent glutamate dehydrogenase Subversion of innate immune responses by Brucella through the targeted degradation of the TLR signaling adapter, MAL The Brucella abortus general stress response system regulates chronic mammalian infection and is controlled by phosphorylation and proteolysis.Bartonella and Brucella--weapons and strategies for stealth attack.Full-Length cDNA Cloning, Molecular Characterization and Differential Expression Analysis of Lysophospholipase I from Ovis aries.Survival of the fittest: how Brucella strains adapt to their intracellular niche in the host.The manganese transporter MntH is a critical virulence determinant for Brucella abortus 2308 in experimentally infected mice.Analyses of Brucella pathogenesis, host immunity, and vaccine targets using systems biology and bioinformatics.Brucella evasion of adaptive immunity.Brucella melitensis triggers time-dependent modulation of apoptosis and down-regulation of mitochondrion-associated gene expression in mouse macrophages.Brucella abortus Invasion of Osteocytes Modulates Connexin 43 and Integrin Expression and Induces Osteoclastogenesis via Receptor Activator of NF-κB Ligand and Tumor Necrosis Factor Alpha Secretion Silencing of VAMP3 expression does not affect Brucella melitensis infection in mouse macrophages.

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Adaptation of the Brucellae to their intracellular niche.

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2004 nî lūn-bûn

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Adaptation of the Brucellae to their intracellular niche.

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Adaptation of the Brucellae to their intracellular niche.

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Adaptation of the Brucellae to their intracellular niche.

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Adaptation of the Brucellae to their intracellular niche.

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Adaptation of the Brucellae to their intracellular niche.

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Adaptation of the Brucellae to their intracellular niche.

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J.1365-2958.2004.04017.X

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Molecular Microbiology

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Adaptation of the Brucellae to their intracellular niche.

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Bryan H Bellaire

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James A Cardelli

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Michelle Wright Valderas

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R Martin Roop

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P2860

A chromosomally encoded two-component sensory transduction system is required for virulence of Agrobacterium tumefaciens.

The temperature-sensitive growth and survival phenotypes of Escherichia coli cydDC and cydAB strains are due to deficiencies in cytochrome bd and are corrected by exogenous catalase and reducing agents.

The RNA-binding protein HF-I plays a global regulatory role which is largely, but not exclusively, due to its role in expression of the sigmaS subunit of RNA polymerase in Escherichia coli.

Brucella abortus strain 2308 produces brucebactin, a highly efficient catecholic siderophore.

What is the nature of the replicative niche of a stealthy bug named Brucella?

Brucella abortus Lipopolysaccharide in Murine Peritoneal Macrophages Acts as a Down-Regulator of T Cell Activation

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621-630

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10.1111/J.1365-2958.2004.04017.X

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8602805

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