about
Sphingosine-1-phosphate lyase potentiates apoptosis via p53- and p38-dependent pathways and is down-regulated in colon cancerHypoxia-Inducible Factors (HIFs) and Phosphorylation: Impact on Stability, Localization, and TransactivityInduction of oxidative metabolism by mitochondrial frataxin inhibits cancer growth: Otto Warburg revisitedInhibition of p38 MAPK suppresses inflammatory cytokine induction by etoposide, 5-fluorouracil, and doxorubicin without affecting tumoricidal activityBalance between MKK6 and MKK3 mediates p38 MAPK associated resistance to cisplatin in NSCLCEGFR Tyrosine 845 Phosphorylation-Dependent Proliferation and Transformation of Breast Cancer Cells Require Activation of p38 MAPK.A spatiotemporally coordinated cascade of protein kinase C activation controls isoform-selective translocation.Identifying core gene modules in glioblastoma based on multilayer factor-mediated dysfunctional regulatory networks through integrating multi-dimensional genomic data.Conserved regulation of MAP kinase expression by PUF RNA-binding proteins.Onconase responsive genes in human mesothelioma cells: implications for an RNA damaging therapeutic agent.Signaling pathway networks mined from human pituitary adenoma proteomics data.PE‑induced apoptosis in SMMC‑7721 cells: involvement of Erk and Stat signalling pathways.Cisplatin induces cytotoxicity through the mitogen-activated protein kinase pathways and activating transcription factor 3.Production of IL-1β by bone marrow-derived macrophages in response to chemotherapeutic drugs: synergistic effects of doxorubicin and vincristine.Antagonist effect of triptolide on AKT activation by truncated retinoid X receptor-alphaTelekin suppresses human hepatocellular carcinoma cells in vitro by inducing G2/M phase arrest via the p38 MAPK signaling pathway.Serine 59 phosphorylation of {alpha}B-crystallin down-regulates its anti-apoptotic function by binding and sequestering Bcl-2 in breast cancer cells.Galangin induces B16F10 melanoma cell apoptosis via mitochondrial pathway and sustained activation of p38 MAPK.Branched motifs enable long-range interactions in signaling networks through retrograde propagationFrequent genetic abnormalities of the PI3K/AKT pathway in primary ovarian cancer predict patient outcomeLigand-dependent regulation of the activity of the orphan nuclear receptor, small heterodimer partner (SHP), in the repression of bile acid biosynthetic CYP7A1 and CYP8B1 genes.Role of stress-activated MAP kinase P38 in cisplatin- and DTT-induced apoptosis of the esophageal carcinoma cell line Eca109.Integrative genomic signatures of hepatocellular carcinoma derived from nonalcoholic Fatty liver disease.Activation of p38 Mitogen-Activated Protein Kinase in Gaucher's Diseasep38 mitogen-activated protein kinase inhibits USP22 transcription in HeLa cells.P38 MAPK expression and activation predicts failure of response to CHOP in patients with Diffuse Large B-Cell Lymphoma.Interleukin-6 and its receptor, key players in hepatobiliary inflammation and cancerThe use of variations in proteomes to predict, prevent, and personalize treatment for clinically nonfunctional pituitary adenomasMEK inhibition suppresses cell invasion and migration in ovarian cancers with activation of ERK1/2.Idiopathic pulmonary fibrosis : new concepts in pathogenesis and implications for drug therapyOpposing roles of mitogenic and stress signaling pathways in the induction of cancer dormancyProliferation enhanced by NGF-NTRK1 signaling makes pancreatic cancer cells more sensitive to 2DG-induced apoptosis.Estradiol promotes the development of a fibrotic phenotype and is increased in the serum of patients with systemic sclerosisTargeting signaling pathways in epithelial ovarian cancer.A long-term surviving patient with recurrent low-grade serous ovarian carcinoma treated with the MEK1/2 inhibitor, selumetinib.KRAS or BRAF mutation status is a useful predictor of sensitivity to MEK inhibition in ovarian cancerCytoprotective Mechanism of Cyanidin and Delphinidin against Oxidative Stress-Induced Tenofibroblast Death.Vitamin E succinate induces NAG-1 expression in a p38 kinase-dependent mechanismMutational analysis of KRAS, BRAF, and TP53 genes of ovarian serous carcinomas in Korean women.Phosphorylation of Plk1 at Ser326 regulates its functions during mitotic progression
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
p38 MAP kinase: a convergence point in cancer therapy.
@ast
p38 MAP kinase: a convergence point in cancer therapy.
@en
type
label
p38 MAP kinase: a convergence point in cancer therapy.
@ast
p38 MAP kinase: a convergence point in cancer therapy.
@en
prefLabel
p38 MAP kinase: a convergence point in cancer therapy.
@ast
p38 MAP kinase: a convergence point in cancer therapy.
@en
P1476
p38 MAP kinase: a convergence point in cancer therapy.
@en
P2093
Andrew R Hallahan
James M Olson
P304
P356
10.1016/J.MOLMED.2004.01.007
P577
2004-03-01T00:00:00Z