Oxidative stress as a cause of nigral cell death in Parkinson's disease and incidental Lewy body disease. The Royal Kings and Queens Parkinson's Disease Research Group.
about
Vesicular monoamine transporter 2: role as a novel target for drug developmentParkinson's Disease: The Mitochondria-Iron LinkMechanisms of altered redox regulation in neurodegenerative diseases--focus on S--glutathionylationSerotonin 1A Receptors on Astrocytes as a Potential Target for the Treatment of Parkinson's DiseaseAnti-Oxidants in Parkinson's Disease Therapy: A Critical Point of ViewNeuronal NOS inhibitor that reduces oxidative DNA lesions and neuronal sensitivity increases the expression of intact c-fos transcripts after brain injuryA review of the mechanism of injury and treatment approaches for illness resulting from exposure to water-damaged buildings, mold, and mycotoxinsLipid pathway alterations in Parkinson's disease primary visual cortexFurther structure-activity relationships study of substituted dithiolethiones as glutathione-inducing neuroprotective agentsGlutathione depletion and overproduction both initiate degeneration of nigral dopaminergic neuronsRole of iron in neurodegenerative diseases.Loss of UCHL1 promotes age-related degenerative changes in the enteric nervous systemSelective neuronal vulnerability to oxidative stress in the brain.Pathological correlations between traumatic brain injury and chronic neurodegenerative diseases.Critical role of truncated α-synuclein and aggregates in Parkinson's disease and incidental Lewy body disease.The antioxidant drink effective microorganism-X (EM-X) pre-treatment attenuates the loss of nigrostriatal dopaminergic neurons in 6-hydroxydopamine-lesion rat model of Parkinson's disease.Dopamine mercapturate can augment dopaminergic neurodegeneration.Synergy between glutathione peroxidase-1 and astrocytic growth factors suppresses free radical generation and protects dopaminergic neurons against 6-hydroxydopamine.Cognitive-enhancing effect of quercetin in a rat model of Parkinson's disease induced by 6-hydroxydopamine.An imbalance in antioxidant defense affects cellular function: the pathophysiological consequences of a reduction in antioxidant defense in the glutathione peroxidase-1 (Gpx1) knockout mouse.The biochemical basis of Parkinson's disease: the role of catecholamine o-quinones: a review-discussion.Levodopa deactivates enzymes that regulate thiol-disulfide homeostasis and promotes neuronal cell death: implications for therapy of Parkinson's disease.Characterization of intracellular elevation of glutathione (GSH) with glutathione monoethyl ester and GSH in brain and neuronal cultures: relevance to Parkinson's disease.Nonselenium glutathione peroxidase in human brain : elevated levels in Parkinson's disease and dementia with lewy bodiesProtein-bound acrolein: potential markers for oxidative stressDifferential vulnerability of substantia nigra and corpus striatum to oxidative insult induced by reduced dietary levels of essential fatty acids.Antioxidative and steroid systems in neurological and psychiatric disorders.Modulation of microglial pro-inflammatory and neurotoxic activity for the treatment of Parkinson's disease.Potent induction of total cellular GSH and NQO1 as well as mitochondrial GSH by 3H-1,2-dithiole-3-thione in SH-SY5Y neuroblastoma cells and primary human neurons: protection against neurocytotoxicity elicited by dopamine, 6-hydroxydopamine, 4-hydroxProtection of MPTP-induced neuroinflammation and neurodegeneration by Pycnogenol.Oxidative and nitrative protein modifications in Parkinson's diseaseGlutathione deficiency in Gclm null mice results in complex I inhibition and dopamine depletion following paraquat administrationEffects of Oxidative Stress and Testosterone on Pro-Inflammatory Signaling in a Female Rat Dopaminergic Neuronal Cell LineGlutathione metabolism and Parkinson's disease.Nitric oxide in health and disease of the nervous system.Oxidative damage to the c-fos gene and reduction of its transcription after focal cerebral ischemia.Restless Legs Syndrome in shift workers: A cross sectional study on male assembly workers.Redox regulation of mitochondrial function with emphasis on cysteine oxidation reactions.Antioxidant gene therapy against neuronal cell death.Immunohistochemical detection of 4-hydroxynonenal protein adducts in Parkinson disease.
P2860
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P2860
Oxidative stress as a cause of nigral cell death in Parkinson's disease and incidental Lewy body disease. The Royal Kings and Queens Parkinson's Disease Research Group.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Oxidative stress as a cause of ...... nson's Disease Research Group.
@ast
Oxidative stress as a cause of ...... nson's Disease Research Group.
@en
type
label
Oxidative stress as a cause of ...... nson's Disease Research Group.
@ast
Oxidative stress as a cause of ...... nson's Disease Research Group.
@en
prefLabel
Oxidative stress as a cause of ...... nson's Disease Research Group.
@ast
Oxidative stress as a cause of ...... nson's Disease Research Group.
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P2093
P356
P1433
P1476
Oxidative stress as a cause of ...... nson's Disease Research Group.
@en
P2093
P356
10.1002/ANA.410320714
P478
P577
1992-01-01T00:00:00Z