Treatment with incomplete Freund's adjuvant and Listeria monocytogenes delays diabetes via an interleukin-17-secretion-independent pathway

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Treatment with incomplete Freund's adjuvant and Listeria monocytogenes delays diabetes via an interleukin-17-secretion-independent pathway

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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Treatment with incomplete Freu ...... -secretion-independent pathway

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Treatment with incomplete Freu ...... -secretion-independent pathway

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Treatment with incomplete Freu ...... -secretion-independent pathway

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Treatment with incomplete Freu ...... -secretion-independent pathway

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Treatment with incomplete Freu ...... -secretion-independent pathway

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Treatment with incomplete Freu ...... -secretion-independent pathway

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P1476

Treatment with incomplete Freu ...... -secretion-independent pathway

@en

P2093

Hai-Ping Wang

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Zhi-Gao He

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P2860

NKT cells stimulated by long fatty acyl chain sulfatides significantly reduce the incidence of type 1 diabetes in nonobese diabetic mice [corrected]

Reversal of established autoimmune diabetes by restoration of endogenous beta cell function

Breeding of a non-obese, diabetic strain of mice

Alpha-galactosylceramide promotes killing of Listeria monocytogenes within the macrophage phagosome through invariant NKT-cell activation

Inflammatory tendencies and overproduction of IL-17 in the colon of young NOD mice are counteracted with diet change.

Prevention of type I diabetes in NOD mice by adjuvant immunotherapy.

Complete Freund's adjuvant-induced T cells prevent the development and adoptive transfer of diabetes in nonobese diabetic mice.

Going both ways: immune regulation via CD1d-dependent NKT cells.

NOD mice and autoimmunity.

Regulation of type 1 diabetes by NKT cells.

P304

1934-1938

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scholarly article

P356

10.3892/ETM.2015.2328

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P433

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P478

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P577

2015-03-03T00:00:00Z

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P5875

279728877

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P698

26136917

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P921

Listeria monocytogenes

P932

4471723

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