Elevated concentrations of inflammation-related proteins in postnatal blood predict severe developmental delay at 2 years of age in extremely preterm infants. - Wikidata - wikimedia - TriplyDB

Elevated concentrations of inflammation-related proteins in postnatal blood predict severe developmental delay at 2 years of age in extremely preterm infants.

Elevated concentrations of inflammation-related proteins in postnatal blood predict severe developmental delay at 2 years of age in extremely preterm infants.

http://www.wikidata.org/entity/Q35757348

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Intermittent or sustained systemic inflammation and the preterm brain Interleukin-1 beta guides the migration of cortical neurons Febrile illness and pro-inflammatory cytokines are associated with lower neurodevelopmental scores in Bangladeshi infants living in poverty.Retinopathy of prematurity and brain damage in the very preterm newborn The Development of Extremely Preterm Infants Born to Women Who Had Genitourinary Infections During Pregnancy Neuroprotective potential of erythropoietin in neonates; design of a randomized trial.Genomic biomarkers of prenatal intrauterine inflammation in umbilical cord tissue predict later life neurological outcomes Early postnatal illness severity scores predict neurodevelopmental impairments at 10 years of age in children born extremely preterm.Urinary Levels of IL-1β and GDNF in Preterm Neonates as Potential Biomarkers of Motor Development: A Prospective Study.Patent ductus arteriosus ligation and adverse outcomes: causality or bias?Effect of sustained postnatal systemic inflammation on hippocampal volume and function in mice.Systemic inflammation and cerebral palsy risk in extremely preterm infants.Systemic inflammation associated with severe intestinal injury in extremely low gestational age newborns Chorioamnionitis and early childhood outcomes among extremely low-gestational-age neonates.Neonatal magnesium levels correlate with motor outcomes in premature infants: a long-term retrospective cohort study.The breadth and type of systemic inflammation and the risk of adverse neurological outcomes in extremely low gestation newborns.Systemic inflammation in the extremely low gestational age newborn following maternal genitourinary infections Elevated endogenous erythropoietin concentrations are associated with increased risk of brain damage in extremely preterm neonates Systems approach to the study of brain damage in the very preterm newborn.Elevated blood levels of inflammation-related proteins are associated with an attention problem at age 24 mo in extremely preterm infants.Relationships among the concentrations of 25 inflammation-associated proteins during the first postnatal weeks in the blood of infants born before the 28th week of gestation Late Enteral Feedings Are Associated with Intestinal Inflammation and Adverse Neonatal Outcomes The Human Microbiome during Bacterial Vaginosis.Girls and Boys Born before 28 Weeks Gestation: Risks of Cognitive, Behavioral, and Neurologic Outcomes at Age 10 Years Circulating Inflammatory-Associated Proteins in the First Month of Life and Cognitive Impairment at Age 10 Years in Children Born Extremely Preterm.Systemic inflammation associated with mechanical ventilation among extremely preterm infants.Erythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia.Systemic inflammation on postnatal days 21 and 28 and indicators of brain dysfunction 2years later among children born before the 28th week of gestation.Inflammation-initiating illnesses, inflammation-related proteins, and cognitive impairment in extremely preterm infants.Two-hit model of brain damage in the very preterm newborn: small for gestational age and postnatal systemic inflammation.The role of systemic inflammation linking maternal BMI to neurodevelopment in children Histological chorioamnionitis shapes the neonatal transcriptomic immune response.Sexual epigenetic dimorphism in the human placenta: implications for susceptibility during the prenatal period.The Relationship of Maternal Prepregnancy Body Mass Index and Pregnancy Weight Gain to Neurocognitive Function at Age 10 Years among Children Born Extremely Preterm.Clinical chorioamnionitis at term VI: acute chorioamnionitis and funisitis according to the presence or absence of microorganisms and inflammation in the amniotic cavity.A "multi-hit" model of neonatal white matter injury: cumulative contributions of chronic placental inflammation, acute fetal inflammation and postnatal inflammatory events.Both antenatal and postnatal inflammation contribute information about the risk of brain damage in extremely preterm newborns.Cognitive functioning at the age of 10 years among children born extremely preterm: a latent profile approach.Antenatal and Neonatal Antecedents of Executive Dysfunctions in Extremely Preterm Children.ATS Core Curriculum 2017: Part II. Pediatric Pulmonary Medicine.

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Elevated concentrations of inflammation-related proteins in postnatal blood predict severe developmental delay at 2 years of age in extremely preterm infants.

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Deborah Hirtz

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Elizabeth N Allred

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P2860

Chronic lung disease and developmental delay at 2 years of age in children born before 28 weeks' gestation.

The ELGAN study of the brain and related disorders in extremely low gestational age newborns.

Brain damage in preterm newborns: might enhancement of developmentally regulated endogenous protection open a door for prevention?

Neurodevelopment of extremely preterm infants who had necrotizing enterocolitis with or without late bacteremia

Interleukin-6 concentrations in umbilical cord plasma are elevated in neonates with white matter lesions associated with periventricular leukomalacia.

Development and reliability of a system to classify gross motor function in children with cerebral palsy.

Blood protein concentrations in the first two postnatal weeks that predict bronchopulmonary dysplasia among infants born before the 28th week of gestation

Cytokines and perinatal brain damage.

Inflammatory brain damage in preterm newborns--dry numbers, wet lab, and causal inferences.

The transcriptome of the fetal inflammatory response syndrome

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