Gene expression in vivo shows that Helicobacter pylori colonizes an acidic niche on the gastric surface. - Wikidata - wikimedia - TriplyDB

Gene expression in vivo shows that Helicobacter pylori colonizes an acidic niche on the gastric surface.

Gene expression in vivo shows that Helicobacter pylori colonizes an acidic niche on the gastric surface.

http://www.wikidata.org/entity/Q35758698

about

Critical role of RecN in recombinational DNA repair and survival of Helicobacter pylori A second-generation protein-protein interaction network of Helicobacter pylori Bacterial genome instability Iron deficiency accelerates Helicobacter pylori-induced carcinogenesis in rodents and humans Phylogeographic origin of Helicobacter pylori determines host-adaptive responses upon coculture with gastric epithelial cells.Recombination-based in vivo expression technology identifies Helicobacter pylori genes important for host colonization Cytoplasmic histidine kinase (HP0244)-regulated assembly of urease with UreI, a channel for urea and its metabolites, CO2, NH3, and NH4(+), is necessary for acid survival of Helicobacter pylori.Built shallow to maintain homeostasis and persistent infection: insight into the transcriptional regulatory network of the gastric human pathogen Helicobacter pylori.In situ expression of cagA and risk of gastroduodenal disease in Helicobacter pylori-infected children.Ammonium metabolism enzymes aid Helicobacter pylori acid resistance.Revealing the dynamics of polymicrobial infections: implications for antibiotic therapy.A novel DNA-binding protein plays an important role in Helicobacter pylori stress tolerance and survival in the host.Helicobacter pylori: Genomic Insight into the Host-Pathogen Interaction.In depth analysis of the Helicobacter pylori cag pathogenicity island transcriptional responses.Life at the margins: modulation of attachment proteins in Helicobacter pylori Molecular aspects of bacterial pH sensing and homeostasis.In vivo expression of Helicobacter pylori virulence genes in patients with gastritis, ulcer, and gastric cancer.Use of the chinchilla model for nasopharyngeal colonization to study gene expression by Moraxella catarrhalis Genetic Diversity as Consequence of a Microaerobic and Neutrophilic Lifestyle Alterations in Helicobacter pylori triggered by contact with gastric epithelial cells Multiple Acid Sensors Control Helicobacter pylori Colonization of the Stomach Polymorphisms of the acid sensing histidine kinase gene arsS in Helicobacter pylori populations from anatomically distinct gastric sites.Identification of Campylobacter jejuni genes involved in the response to acidic pH and stomach transit.Measuring Escherichia coli Gene Expression during Human Urinary Tract Infections High dietary salt intake exacerbates Helicobacter pylori-induced gastric carcinogenesis High Dose Ilaprazole/Amoxicillin as First-Line Regimen for Helicobacter pylori Infection in Korea.The role of ExbD in periplasmic pH homeostasis in Helicobacter pylori Structural analysis of the DNA-binding domain of the Helicobacter pylori response regulator ArsR.The Helicobacter pylori CZB Cytoplasmic Chemoreceptor TlpD Forms an Autonomous Polar Chemotaxis Signaling Complex That Mediates a Tactic Response to Oxidative Stress.Helicobacter Pylori associated global gastric cancer burden.Helicobacter pylori impedes acid-induced tightening of gastric epithelial junctions.Environmental determinants of transformation efficiency in Helicobacter pylori.Gastric infection by Helicobacter pylori.Gastric infection by Helicobacter pylori.Bacterial histone-like proteins: roles in stress resistance.A histone-like protein of Helicobacter pylori protects DNA from stress damage and aids host colonization.Strategies used by helicobacter pylori to establish persistent infection Eradication of Helicobacter pylori Infection.Survival of Helicobacter pylori in gastric acidic territory.Investigating the Role of Helicobacter pylori PriA Protein.

P2860

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P2860

Gene expression in vivo shows that Helicobacter pylori colonizes an acidic niche on the gastric surface.

http://www.wikidata.org/entity/Q35758698

description

2007 nî lūn-bûn

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Gene expression in vivo shows ...... niche on the gastric surface.

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Proceedings of the National Academy of Sciences of the United States of America

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Gene expression in vivo shows ...... niche on the gastric surface.

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David R Scott

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Elizabeth A Marcus

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George Sachs

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Jane Oh

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Yi Wen

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P2860

Primer3 on the WWW for general users and for biologist programmers

Characterization of the ArsRS regulon of Helicobacter pylori, involved in acid adaptation

Impaired mucus-bicarbonate barrier in Helicobacter pylori-infected mice.

The gastric transitional zones: neglected links between gastroduodenal pathology and helicobacter ecology.

Helicobacter pylori rocF is required for arginase activity and acid protection in vitro but is not essential for colonization of mice or for urease activity.

Chemotaxis plays multiple roles during Helicobacter pylori animal infection.

Comparison of Helicobacter pylori virulence gene expression in vitro and in the Rhesus macaque.

Helicobacter pylori possesses two CheY response regulators and a histidine kinase sensor, CheA, which are essential for chemotaxis and colonization of the gastric mucosa

Acid-induced gene expression in Helicobacter pylori: study in genomic scale by microarray.

Helicobacter pylori motility.

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7235-7240

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104

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Helicobacter pylori

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1855417

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