Androgen deprivation induces rapid involution and recovery of human prostate vasculature - Wikidata - wikimedia - TriplyDB

Androgen deprivation induces rapid involution and recovery of human prostate vasculature

Androgen deprivation induces rapid involution and recovery of human prostate vasculature

http://www.wikidata.org/entity/Q35760385

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Mechanistic Insights into Molecular Targeting and Combined Modality Therapy for Aggressive, Localized Prostate Cancer Primary xenografts of human prostate tissue as a model to study angiogenesis induced by reactive stroma Multimodal imaging guided preclinical trials of vascular targeting in prostate cancer.Androgen receptor in human endothelial cells.Vascular responses to radiotherapy and androgen-deprivation therapy in experimental prostate cancer.Development of a reactive stroma associated with prostatic intraepithelial neoplasia in EAF2 deficient mice.Selective targeting of bioengineered platelets to prostate cancer vasculature: new paradigm for therapeutic modalities Targeting stromal androgen receptor suppresses prolactin-driven benign prostatic hyperplasia (BPH).The role of intracrine androgen metabolism, androgen receptor and apoptosis in the survival and recurrence of prostate cancer during androgen deprivation therapy.Carbogen gas and radiotherapy outcomes in prostate cancer.Role of androgen and vitamin D receptors in endothelial cells from benign and malignant human prostate.Fibroblasts prolong serum prostate-specific antigen decline after androgen deprivation therapy in prostate cancer.Endothelial cells enhance prostate cancer metastasis via IL-6→androgen receptor→TGF-β→MMP-9 signals.Apoptosis in Living Animals Is Assisted by Scavenger Cells and Thus May Not Mainly Go through the Cytochrome C-Caspase Pathway.Endostatin inhibits androgen-independent prostate cancer growth by suppressing nuclear receptor-mediated oxidative stress.Serum-free complete medium, an alternative medium to mimic androgen deprivation in human prostate cancer cell line models.Androgens modulate male-derived endothelial cell homeostasis using androgen receptor-dependent and receptor-independent mechanisms.Anti-angiogenic activity of PSA-derived peptides.Endothelial cells promote metastasis of prostate cancer by enhancing autophagy

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P2860

Androgen deprivation induces rapid involution and recovery of human prostate vasculature

http://www.wikidata.org/entity/Q35760385

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2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

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AJPENDO.00210.2010

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American Journal of Physiology - Endocrinology and Metabolism

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Androgen deprivation induces rapid involution and recovery of human prostate vasculature

@en

P2093

Alejandro Godoy

http://www.w3.org/2001/XMLSchema#string

Danny R Gray

http://www.w3.org/2001/XMLSchema#string

Gary J Smith

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James L Mohler

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Jeffrey M Yau

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Paula Sotomayor

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R Robert Vethanayagam

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Swaroop Singh

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Viviana P Montecinos

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P2860

Immunolocalization of von Willebrand protein in Weibel-Palade bodies of human endothelial cells

Targeting HIF-1 for cancer therapy

Incidental prostatic carcinoma. A predictive role of neoangiogenesis and comparison with other prognostic factors.

Managing benign prostatic hyperplasia.

Androgen receptor localization in different cell types of the adult rat prostate.

Endothelial cell death, angiogenesis, and microvascular function after castration in an androgen-dependent tumor: role of vascular endothelial growth factor

Angiogenesis in wound healing.

The effects of androgen deprivation on the prostate gland: cell death mediated by vascular regression.

Angiogenesis: the role of the microenvironment in flipping the switch.

Vascular endothelial growth factor and vascular adjustments to perturbations in oxygen homeostasis.

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