Global H3K4me3 genome mapping reveals alterations of innate immunity signaling and overexpression of JMJD3 in human myelodysplastic syndrome CD34+ cells.
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Deconstructing innate immune signaling in myelodysplastic syndromesDeregulation of innate immune and inflammatory signaling in myelodysplastic syndromesJMJD3 as an epigenetic regulator in development and diseaseToll-like receptor alterations in myelodysplastic syndromeJMJD3 suppresses stem cell-like characteristics in breast cancer cells by downregulation of Oct4 independently of its demethylase activity.Genome-wide analysis of histone modifications in human endometrial stromal cells.Expression of PD-L1, PD-L2, PD-1 and CTLA4 in myelodysplastic syndromes is enhanced by treatment with hypomethylating agents.Overexpression of the toll-like receptor (TLR) signaling adaptor MYD88, but lack of genetic mutation, in myelodysplastic syndromes.Overexpression of miR-125a in myelodysplastic syndrome CD34+ cells modulates NF-κB activation and enhances erythroid differentiation arrestA phase I study of oral ARRY-614, a p38 MAPK/Tie2 dual inhibitor, in patients with low or intermediate-1 risk myelodysplastic syndromes.A cell-permeable ester derivative of the JmjC histone demethylase inhibitor IOX1Rps14 haploinsufficiency causes a block in erythroid differentiation mediated by S100A8 and S100A9Epigenetics of myelodysplastic syndromes.Regulatory variation: an emerging vantage point for cancer biology.The roles of Jumonji-type oxygenases in human disease.Integrating genetics and epigenetics in myelodysplastic syndromes: advances in pathogenesis and disease evolution.The inflammatory microenvironment in MDS.Constitutive IRF8 expression inhibits AML by activation of repressed immune response signaling.KDM6B modulates MAPK pathway mediating multiple myeloma cell growth and survival.Fanconi Anemia Mesenchymal Stromal Cells-Derived Glycerophospholipids Skew Hematopoietic Stem Cell Differentiation Through Toll-Like Receptor Signaling.Increased PD-1/STAT1 ratio may account for the survival benefit in decitabine therapy for lower risk myelodysplastic syndrome.The histone demethylase Jmjd3 regulates zebrafish myeloid development by promoting spi1 expression.Genetic rescue of lineage-balanced blood cell production reveals a crucial role for STAT3 antiinflammatory activity in hematopoiesis.Therapeutic potential of GSK-J4, a histone demethylase KDM6B/JMJD3 inhibitor, for acute myeloid leukemia.The role of p53 in myelodysplastic syndromes and acute myeloid leukemia: molecular aspects and clinical implications.IRAK1 expression in bone marrow cells does not impact patient outcomes in myelodysplastic syndromes.JMJD3 facilitates C/EBPβ-centered transcriptional program to exert oncorepressor activity in AML
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Global H3K4me3 genome mapping reveals alterations of innate immunity signaling and overexpression of JMJD3 in human myelodysplastic syndrome CD34+ cells.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@ast
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@en
type
label
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@ast
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@en
prefLabel
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@ast
Global H3K4me3 genome mapping ...... splastic syndrome CD34+ cells.
@en
P2093
P2860
P356
P1433
P1476
Global H3K4me3 genome mapping ...... ysplastic syndrome CD34+ cells
@en
P2093
C Bueso-Ramos
G Garcia-Manero
H Kantarjian
P2860
P2888
P304
P356
10.1038/LEU.2013.91
P577
2013-03-29T00:00:00Z