Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity. - Wikidata - wikimedia - TriplyDB

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

http://www.wikidata.org/entity/Q35769609

about

"Inflammaging" as a Druggable Target: A Senescence-Associated Secretory Phenotype-Centered View of Type 2 Diabetes Redox control of senescence and age-related disease Extracellular microRNAs and endothelial hyperglycaemic memory: a therapeutic opportunity?Aging and adipose tissue: potential interventions for diabetes and regenerative medicine Obesity altered T cell metabolism and the response to infection.Regulation of Transcription Factors by Reactive Oxygen Species and Nitric Oxide in Vascular Physiology and Pathology.Identification of a novel senolytic agent, navitoclax, targeting the Bcl-2 family of anti-apoptotic factors Targeting senescent cells enhances adipogenesis and metabolic function in old age 17α-Estradiol Alleviates Age-related Metabolic and Inflammatory Dysfunction in Male Mice Without Inducing Feminization Senotherapy: growing old and staying young?Screening of a kinase library reveals novel pro-senescence kinases and their common NF-κB-dependent transcriptional program.Growth Hormone Receptor Antagonist Transgenic Mice Have Increased Subcutaneous Adipose Tissue Mass, Altered Glucose Homeostasis and No Change in White Adipose Tissue Cellular Senescence Exercise Prevents Diet-Induced Cellular Senescence in Adipose Tissue.Leukocyte telomere length and mortality risk in patients with type 2 diabetes.Inflammatory Response in the CNS: Friend or Foe?Evaluation of the Chinese herbal medicine Jinlida in type 2 diabetes patients based on stratification: Results of subgroup analysis from a 12-week trial.Perspective: Targeting the JAK/STAT pathway to fight age-related dysfunction.Vascular endothelium in diabetes.Physiological Aging: Links Among Adipose Tissue Dysfunction, Diabetes, and Frailty.Pathophysiology of heart failure and frailty: a common inflammatory origin?Pathophysiological Fundamentals of Diabetic Cardiomyopathy.Energetic interventions for healthspan and resiliency with aging.Metabolic Regulation of Angiogenesis in Diabetes and Aging.Growth Hormone's Effect on Adipose Tissue: Quality versus Quantity.The association between Alu hypomethylation and severity of type 2 diabetes mellitus.Identification of HSP90 inhibitors as a novel class of senolytics.Age-related modulation of plasmatic beta-Galactosidase activity in healthy subjects and in patients affected by T2DM.Short-term sustained hyperglycaemia fosters an archetypal senescence-associated secretory phenotype in endothelial cells and macrophages.Nitric oxide-heat shock protein axis in menopausal hot flushes: neglected metabolic issues of chronic inflammatory diseases associated with deranged heat shock response.Extracellular vesicles and aging.The ominous triad of adipose tissue dysfunction: inflammation, fibrosis, and impaired angiogenesis.Mitochondrial (Dys) Function in Inflammaging: Do MitomiRs Influence the Energetic, Oxidative, and Inflammatory Status of Senescent Cells?Pancreatic β Cell Regeneration as a Possible Therapy for Diabetes.Urinary DcR2 is a novel biomarker for tubulointerstitial injury in patients with diabetic nephropathy.Physical Exercise Enhanced Heat Shock Protein 60 Expression and Attenuated Inflammation in the Adipose Tissue of Human Diabetic Obese.Diabetes prevalence in rural Indigenous Guatemala: A geographic-randomized cross-sectional analysis of risk Linking a role of lncRNAs (long non-coding RNAs) with insulin resistance, accelerated senescence, and inflammation in patients with type 2 diabetes

P2860

Q26747251-4046C34B-0C97-4547-9E89-325869959768 Q28074230-49F0D7DA-D25B-4399-B149-ED5C5772826E Q28079163-AE2F713E-34F7-4EA1-BB37-B7C1052D0A17 Q28079178-D44AFA92-4572-4FB2-9129-823B45EE386C Q30235016-E7E265EC-C50D-4BB6-8E85-E7DED887982B Q30275422-8C3A897B-2948-4FCD-B384-1AD2FD9DA170 Q34045737-44F483E9-0E77-4065-B1B3-C144B4ADEB75 Q34505890-F870BC93-690A-4081-AE70-5454539B3FA2 Q34510747-B7A2F3D1-0A43-4A10-BF50-BB15869464A1 Q34681446-A8569B11-8887-4150-81D2-7AB8E4058F06 Q35846096-FC7FE592-F102-4223-BF1E-8B6D2E1C2CA4 Q36599265-5D36FA93-C57A-420E-8DB3-9CD1D4148551 Q36930817-6F06F695-6B07-4958-9F25-ED4E3104DE5F Q37588202-9DC673A9-0DE8-49FA-A791-EB6A266C22A3 Q38790379-50DEB9A7-42D1-4408-A023-3085EEB63D23 Q38832869-CD4BEC72-3B4A-4798-A76A-55416D32D4A2 Q38848396-1E9BF80E-D7BD-4980-8825-4FA4B5ED0453 Q39010377-8AD22B2F-C8A5-4C1E-98B3-6F45B0FE4EC4 Q39032248-51FA4B21-3219-4ABD-8DCF-A9C5842B649B Q39165266-6E2EE639-026A-4B9A-B155-2D214DC16C29 Q39195672-C4090751-36E4-4B14-8D31-F2FA019B2DB7 Q39291992-FEE1A1A2-37D8-4231-A393-81C11E16C26A Q39373990-6DB86B29-B32F-4C97-8B7F-6C65925AACA7 Q41573026-BD926204-65EA-4A59-812B-D9A9D3A9DF32 Q41588205-59397EBD-AF10-4C02-B935-9326A7571683 Q41606906-8C58F555-54C7-4741-B321-AB252BEDC228 Q47102273-26834DD9-56EB-4BBD-87B4-469FE5259941 Q47148937-3F89F82E-74BF-4D63-A064-B417DB9AA755 Q47807793-C9D79633-C776-4D2F-B7CE-0E41F5F6546A Q49352096-66A11DED-7C3C-4204-9351-0A0C1D99F1E6 Q49430910-8F6F8B29-C5AC-4F98-AB88-F0C95D6536D7 Q49748248-70A054A0-0FB2-4915-89DB-BE368F406350 Q50178948-BB536531-D043-45D7-9D45-DAEEF89B6B5A Q51081666-F6436C2F-0F48-418D-9764-7261D8AC72D7 Q55265913-0C3F574E-64D8-4BF9-A166-32A69CCC919B Q57170163-E212FFED-327F-4915-930F-8F145CD5CE2A Q58713896-BC5B25E6-4189-4EDB-8BDA-977C811FCD1E

P2860

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

http://www.wikidata.org/entity/Q35769609

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@ast

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@en

type

label

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@ast

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@en

prefLabel

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@ast

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity.

@en

P1433

Q35769609-4B43CB51-95B1-45E4-B299-E358BB8C9262

P1476

Q35769609-0A4B4E67-564E-4C58-A59E-7DE38F3C2B2D

P2093

Q35769609-AA6C72D7-2993-4C25-B735-DE475F912A1C

Q35769609-DDF61420-7770-4E97-8018-E4ACA8C22AC9

Q35769609-F3B62E05-9375-4DE1-94CB-4F4443AC6A63

P2860

Q35769609-02C3BFE0-3BA8-414E-8FA4-CA19E5320E38

Q35769609-055713E3-AF57-4100-A54C-C7B1FD28986C

Q35769609-06B2532B-D333-41D9-BE6A-4984C56FCD36

Q35769609-0AF8FE2C-8844-410C-AF98-01D977CD1923

Q35769609-0CA5352A-67EF-44A5-969F-ABAE83B537BC

Q35769609-0CCD2D64-2A75-45D2-BC86-B2397FEB6B17

Q35769609-0E0E792F-C812-472F-9C59-F5BEE4E14EAC

Q35769609-12808F0A-587A-47AF-A3FC-C682EE28DEDB

Q35769609-1554180A-6520-4AB0-9741-510990A60C36

Q35769609-16513EC4-5442-47C5-91F6-E71F5C51AAC7

P304

Q35769609-2358E7A8-5DE1-4869-B911-401F7E4D9F1F

P31

Q35769609-7A614244-844C-478B-9804-B2212FAE881D

P356

Q35769609-4F097436-CB99-44DC-A628-312FBD2A0AB8

P407

Q35769609-4B1D39BA-545B-4975-8ED1-5EFFA6A0E5DB

P433

Q35769609-751B70FA-819E-4DAF-B4C7-7E92DAEA2B25

P478

Q35769609-675DD2AB-2F13-4900-91A0-AC4104BD86CD

P50

Q35769609-03E25683-313E-469A-855B-B9C18ACA48E2

Q35769609-1A1BCC7A-1EEF-4136-83B3-8BC2DF0D0534

Q35769609-BA88F660-578E-4435-9221-B7F4E5C7C911

P577

Q35769609-7D6CCCCB-391F-428D-99B1-648975FE0457

P698

Q35769609-FCF1ED21-829A-4AB2-9A6B-4DCF59BDB933

P921

Q35769609-6D21CE26-60BD-4733-8016-56007C8BD3F5

Q35769609-71B674B6-1BC1-40DC-AF6C-9E656C4AF4AF

P932

Q35769609-28504D7E-B313-4C44-96E8-3B167E782682

P356

P1433

P1476

Cellular Senescence in Type 2 Diabetes: A Therapeutic Opportunity

@en

P2093

Eduardo N Chini

http://www.w3.org/2001/XMLSchema#string

James L Kirkland

http://www.w3.org/2001/XMLSchema#string

Tamara Tchkonia

http://www.w3.org/2001/XMLSchema#string

P2860

Feedback between p21 and reactive oxygen production is necessary for cell senescence

Autophagy mediates the mitotic senescence transition

Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

Central role of ceramide biosynthesis in body weight regulation, energy metabolism, and the metabolic syndrome

Immunosenescence: emerging challenges for an ageing population

The hallmarks of aging

The prevalence of comorbid depression in adults with diabetes: a meta-analysis

Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders

Prevalence of childhood and adult obesity in the United States, 2011-2012

Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors

P304

2289-2298

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.2337/DB14-1820

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

64

http://www.w3.org/2001/XMLSchema#string

P50

Nathan K. LeBrasseur

P577

2015-07-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

26106186

http://www.w3.org/2001/XMLSchema#string

P921

type-2 diabetes

maturity-onset diabetes of the young

P932

4477358

http://www.w3.org/2001/XMLSchema#string