Apoptosis is abundant in human atherosclerotic lesions, especially in inflammatory cells (macrophages and T cells), and may contribute to the accumulation of gruel and plaque instability.
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Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instabilityMucosal tolerance to a combination of ApoB and HSP60 peptides controls plaque progression and stabilizes vulnerable plaque in Apob(tm2Sgy)Ldlr(tm1Her)/J miceCathepsin D--many functions of one aspartic proteaseMolecular imaging of atherosclerotic plaques targeted to oxidized LDL receptor LOX-1 by SPECT/CT and magnetic resonance.Radiolabelled probes for imaging of atherosclerotic plaques.Inflammatory mediators are perpetuated in macrophages resistant to apoptosis induced by hypoxia.Vascular smooth muscle cell apoptosis in atherosclerosis.Chlamydia pneumoniae augments the oxidized low-density lipoprotein-induced death of mouse macrophages by a caspase-independent pathwayApoptosis in the vasculature: mechanisms and functional importance.The role of nucleotides and purinergic signaling in apoptotic cell clearance - implications for chronic inflammatory diseases.Cystic medial degeneration of the aorta is associated with p53 accumulation, Bax upregulation, apoptotic cell death, and cell proliferation.Helper T cells and atherosclerosis: the cytokine webComplement C1q reduces early atherosclerosis in low-density lipoprotein receptor-deficient mice.Control of apoptosis during angiogenesis by survivin expression in endothelial cells.Localization of apoptotic macrophages at the site of plaque rupture in sudden coronary deathInsulin resistance, metabolic stress, and atherosclerosis.Poor maternal nutrition programmes a pro-atherosclerotic phenotype in ApoE-/- mice.The RIP1-RIP3 Complex Mediates Osteocyte Necroptosis after Ovariectomy in Rats.Lymphocytes are important in early atherosclerosis.TRAIL-expressing T cells induce apoptosis of vascular smooth muscle cells in the atherosclerotic plaque.Assessment of Coronary Plaque Vulnerability with Optical Coherence TomographyNoninvasive imaging of apoptosis in cardiovascular disease.Monoclonal antibodies against oxidized low-density lipoprotein bind to apoptotic cells and inhibit their phagocytosis by elicited macrophages: evidence that oxidation-specific epitopes mediate macrophage recognition.Histological topographical comparisons of atherosclerosis progression in juveniles and young adults.A peripheral blood gene expression score is associated with plaque volume and phenotype by intravascular ultrasound with radiofrequency backscatter analysis: results from the ATLANTA study.Dual molecular imaging for targeting metalloproteinase activity and apoptosis in atherosclerosis: molecular imaging facilitates understanding of pathogenesis.Autoantibodies to posttranslational modifications in rheumatoid arthritis.A2 adenosine receptors and vascular pathologies.Eating the Dead to Keep Atherosclerosis at Bay.Suppression of Akt signaling induces Fas ligand expression: involvement of caspase and Jun kinase activation in Akt-mediated Fas ligand regulation.P2 receptors in atherosclerosis and postangioplasty restenosis.Paracrine control of endothelial cell survival.Nuclear factor-kappaB and caspases co-operatively regulate the activation and apoptosis of human macrophages.Crosstalk between red blood cells and the immune system and its impact on atherosclerosis.Apoptosis in cardiovascular diseaseThe prevention of endothelial dysfunction through endothelial cell apoptosis inhibition in a hypercholesterolemic rabbit model: the effect of L-arginine supplementationPhenotypic high-throughput screening in atherosclerosis research: focus on macrophages.PP2A contributes to endothelial death in high glucose: inhibition by benfotiamine.The presence of apoptotic cells in human atherosclerotic lesions.RNA synthesis and splicing interferes with DNA in situ end labeling techniques used to detect apoptosis.
P2860
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P2860
Apoptosis is abundant in human atherosclerotic lesions, especially in inflammatory cells (macrophages and T cells), and may contribute to the accumulation of gruel and plaque instability.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Apoptosis is abundant in human ...... gruel and plaque instability.
@ast
Apoptosis is abundant in human ...... gruel and plaque instability.
@en
type
label
Apoptosis is abundant in human ...... gruel and plaque instability.
@ast
Apoptosis is abundant in human ...... gruel and plaque instability.
@en
prefLabel
Apoptosis is abundant in human ...... gruel and plaque instability.
@ast
Apoptosis is abundant in human ...... gruel and plaque instability.
@en
P2860
P1476
Apoptosis is abundant in human ...... f gruel and plaque instability
@en
P2093
Björkerud B
Björkerud S
P2860
P304
P407
P577
1996-08-01T00:00:00Z