Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
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The cytomegalovirus-encoded chemokine receptor US28 promotes intestinal neoplasia in transgenic miceModulation of Chemokine Responses: Synergy and CooperativityChemokine interaction with synergy-inducing molecules: fine tuning modulation of cell trafficking.Increase of chemokine interferon-inducible protein-10 (IP-10) in the serum of patients with autoimmune liver diseases and increase of its mRNA expression in hepatocytesNeutralization of interleukin-18 reduces severity in murine colitis and intestinal IFN-gamma and TNF-alpha production.Chemokine expression in IBD. Mucosal chemokine expression is unselectively increased in both ulcerative colitis and Crohn's disease.The proinflammatory CXC-chemokines GRO-alpha/CXCL1 and MIG/CXCL9 are concomitantly expressed in ulcerative colitis and decrease during treatment with topical corticosteroids.Transcript levels of different cytokines and chemokines correlate with clinical and endoscopic activity in ulcerative colitisAtorvastatin reduces plasma levels of chemokine (CXCL10) in patients with Crohn's disease.Delayed resolution of acute inflammation in ulcerative colitis is associated with elevated cytokine release downstream of TLR4.Dietary phenethylisothiocyanate attenuates bowel inflammation in mice.CXCR4 antagonist AMD3100 attenuates colonic damage in mice with experimental colitis.Temporal and anatomic relationship between virus replication and cytokine gene expression after vaginal simian immunodeficiency virus infection.Alternative medicines as emerging therapies for inflammatory bowel diseases.The role of chemokines and chemokine receptors in mucosal inflammation.Desensitization of T lymphocyte function by CXCR3 ligands in human hepatocellular carcinoma.Eldelumab [Anti-IP-10] Induction Therapy for Ulcerative Colitis: A Randomised, Placebo-Controlled, Phase 2b Study.Role of chemokines and chemokine receptors in the gastrointestinal tract.Noncontaminated dietary oats may hamper normalization of the intestinal immune status in childhood celiac diseaseCrucial role for CD69 in the pathogenesis of dextran sulphate sodium-induced colitis.Analysis of gene expression in ceca of Helicobacter hepaticus-infected A/JCr mice before and after development of typhlitis.Cobalt ions induce chemokine secretion in a variety of systemic cell linesAugmented expression of secondary lymphoid tissue chemokine and EBI1 ligand chemokine in Crohn's diseaseCCR2 expressing CD4+ T lymphocytes are preferentially recruited to the ileum in Crohn's disease.Chemokines in eosinophil-associated gastrointestinal disorders.Chemokines in inflammatory bowel disease.P-selectin glycoprotein ligand-1 is needed for sequential recruitment of T-helper 1 (Th1) and local generation of Th17 T cells in dextran sodium sulfate (DSS) colitis.Eosinophil recruitment in type-2 hypersensitivity pulmonary granulomas: source and contribution of monocyte chemotactic protein-3 (CCL7)Chemokine expression dynamics in mycobacterial (type-1) and schistosomal (type-2) antigen-elicited pulmonary granuloma formation.Immunopathogenesis of IBD: current state of the art.Chemokine and cytokine levels in inflammatory bowel disease patients.CXCL10/IP-10: a missing link between inflammation and anti-angiogenesis in preeclampsia?Generation of gut-homing T cells and their localization to the small intestinal mucosa.Formylpeptide receptor-2 contributes to colonic epithelial homeostasis, inflammation, and tumorigenesis.CXCL10 blockade protects mice from cyclophosphamide-induced cystitisMurine norovirus: an intercurrent variable in a mouse model of bacteria-induced inflammatory bowel disease.The functional significance behind expressing two IL-8 receptor types on PMN.Anti-IP-10 antibody (BMS-936557) for ulcerative colitis: a phase II randomised study.Inflammatory bowel disease and intestinal cancer: a paradigm of the Yin-Yang interplay between inflammation and cancer.Mucosal biomarkers in inflammatory bowel disease: key pathogenic players or disease predictors?
P2860
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P2860
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@ast
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@en
type
label
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@ast
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@en
prefLabel
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@ast
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@en
P2093
P2860
P1476
Increased expression of IP-10, IL-8, MCP-1, and MCP-3 in ulcerative colitis.
@en
P2093
D F Robbiani
F Rizzello
M Baggiolini
M Campieri
M Uguccioni
P Gionchetti
P2860
P304
P356
10.1016/S0002-9440(10)65128-0
P407
P577
1999-08-01T00:00:00Z