Modulation of the Host Lipid Landscape to Promote RNA Virus Replication: The Picornavirus Encephalomyocarditis Virus Converges on the Pathway Used by Hepatitis C Virus - Wikidata - wikimedia - TriplyDB

Modulation of the Host Lipid Landscape to Promote RNA Virus Replication: The Picornavirus Encephalomyocarditis Virus Converges on the Pathway Used by Hepatitis C Virus

Modulation of the Host Lipid Landscape to Promote RNA Virus Replication: The Picornavirus Encephalomyocarditis Virus Converges on the Pathway Used by Hepatitis C Virus

http://www.wikidata.org/entity/Q35787779

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Neuroinvasion and Inflammation in Viral Central Nervous System Infections Assembly and release of infectious hepatitis C virus involving unusual organization of the secretory pathway Building Viral Replication Organelles: Close Encounters of the Membrane Types Mutations in Encephalomyocarditis Virus 3A Protein Uncouple the Dependency of Genome Replication on Host Factors Phosphatidylinositol 4-Kinase IIIα and Oxysterol-Binding Protein An siRNA screen for ATG protein depletion reveals the extent of the unconventional functions of the autophagy proteome in virus replication.Transcriptional profiling of host cell responses to encephalomyocarditis virus (EMCV).Foot-and-mouth disease virus genome replication is unaffected by inhibition of type III phosphatidylinositol-4-kinases.Foot-and-mouth disease virus replicates independently of phosphatidylinositol 4-phosphate and type III phosphatidylinositol 4-kinases.(+) RNA virus replication compartments: a safe home for (most) viral replication.Lipid Tales of Viral Replication and Transmission.Interferon Control of the Sterol Metabolic Network: Bidirectional Molecular Circuitry-Mediating Host Protection.Tyrphostin AG1478 Inhibits Encephalomyocarditis Virus and Hepatitis C Virus by Targeting Phosphatidylinositol 4-Kinase IIIα.Single-cell analysis reveals the relevance of foot-and-mouth disease virus persistence to emopamil-binding protein gene expression in host cells.Negative charge and membrane-tethered viral 3B cooperate to recruit viral RNA dependent RNA polymerase 3D pol.Inhibition of cytosolic phospholipase A2α impairs an early step of coronavirus replication in cell culture.A model of OSBP-mediated cholesterol supply to Aichi virus RNA replication sites involving protein-protein interactions among viral proteins, ACBD3, OSBP, VAP-A/B, and SAC1.Escaping Host Factor PI4KB Inhibition: Enterovirus Genomic RNA Replication in the Absence of Replication Organelles.microRNA-219 Reduces Viral Load and Pathologic Changes in Theiler's Virus-Induced Demyelinating Disease.The Origin, Dynamic Morphology, and PI4P-Independent Formation of Encephalomyocarditis Virus Replication Organelles.Phospholipid synthesis fueled by lipid droplets drives the structural development of poliovirus replication organelles

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Modulation of the Host Lipid Landscape to Promote RNA Virus Replication: The Picornavirus Encephalomyocarditis Virus Converges on the Pathway Used by Hepatitis C Virus

http://www.wikidata.org/entity/Q35787779

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Modulation of the Host Lipid L ...... hway Used by Hepatitis C Virus

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JOURNAL.PPAT.1005185

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Modulation of the Host Lipid L ...... hway Used by Hepatitis C Virus

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Christian Harak

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Cristina M Dorobantu

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Frank J M van Kuppeveld

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Hilde M van der Schaar

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Qian Feng

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P2860

The lipid kinase phosphatidylinositol-4 kinase III alpha regulates the phosphorylation status of hepatitis C virus NS5A

Saffold virus, a human Theiler's-like cardiovirus, is ubiquitous and causes infection early in life

Analyses of the radiation of birnaviruses from diverse host phyla and of their evolutionary affinities with other double-stranded RNA and positive strand RNA viruses using robust structure-based multiple sequence alignments and advanced phylogenetic

ACBD3-mediated recruitment of PI4KB to picornavirus RNA replication sites

Recruitment and activation of a lipid kinase by hepatitis C virus NS5A is essential for integrity of the membranous replication compartment

A four-step cycle driven by PI(4)P hydrolysis directs sterol/PI(4)P exchange by the ER-Golgi tether OSBP

Translocation of oxysterol binding protein to Golgi apparatus triggered by ligand binding

Hijacking components of the cellular secretory pathway for replication of poliovirus RNA

Architecture and biogenesis of plus-strand RNA virus replication factories

Membranous replication factories induced by plus-strand RNA viruses

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10.1371/JOURNAL.PPAT.1005185

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9

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Alexander E Gorbalenya

Jeroen R.P.M. Strating

Lucian Albulescu

Mirjam R van Kampen

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2015-09-25T00:00:00Z

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26406250

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