TMEM16A overexpression contributes to tumor invasion and poor prognosis of human gastric cancer through TGF-β signaling. - Wikidata - wikimedia - TriplyDB

TMEM16A overexpression contributes to tumor invasion and poor prognosis of human gastric cancer through TGF-β signaling.

TMEM16A overexpression contributes to tumor invasion and poor prognosis of human gastric cancer through TGF-β signaling.

http://www.wikidata.org/entity/Q35793867

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Modulating Ca²⁺ signals: a common theme for TMEM16, Ist2, and TMC.Knockdown of TMEM14A expression by RNAi inhibits the proliferation and invasion of human ovarian cancer cells.MicroRNA-381 inhibits the metastasis of gastric cancer by targeting TMEM16A expression.Ion channels or aquaporins as novel molecular targets in gastric cancer.TMEM16A/ANO1 suppression improves response to antibody-mediated targeted therapy of EGFR and HER2/ERBB2.The Mechanistic Role of the Calcium-Activated Chloride Channel ANO1 in Tumor Growth and Signaling.Genomic landscape of gastric cancer: molecular classification and potential targets.Molecular, biophysical, and pharmacological properties of calcium-activated chloride channels.Cell-specific mechanisms of TMEM16A Ca2+-activated chloride channel in cancer.Suppression of CLC-3 chloride channel reduces the aggressiveness of glioma through inhibiting nuclear factor-κB pathway.Cell-specific regulation of proliferation by Ano1/TMEM16A in breast cancer with different ER, PR, and HER2 status.Inhibition of transmembrane member 16A calcium-activated chloride channels by natural flavonoids contributes to flavonoid anticancer effects.TMEM207 hinders the tumour suppressor function of WWOX in oral squamous cell carcinoma.Testes-specific protease 50 (TSP50) promotes invasion and metastasis by inducing EMT in gastric cancer.Expression of ANO1/DOG1 is associated with shorter survival and progression of breast carcinomas.Overexpression of chloride channel-3 predicts unfavorable prognosis and promotes cellular invasion in gastric cancer.

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P2860

TMEM16A overexpression contributes to tumor invasion and poor prognosis of human gastric cancer through TGF-β signaling.

http://www.wikidata.org/entity/Q35793867

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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name

TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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type

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TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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prefLabel

TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

@ast

TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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TMEM16A overexpression contrib ...... ancer through TGF-β signaling.

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Bin Luo

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De-Jian Lu

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Fang Liu

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Qing-Hua Cao

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Rong-Cheng Luo

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Xiao-Fang Lu

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Xiao-Guang Wang

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P2860

CFTR and Anoctamin 1 (ANO1) contribute to cAMP amplified exocytosis and insulin secretion in human and murine pancreatic beta-cells

Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling

Structure and function of TMEM16 proteins (anoctamins)

The Ca(2+) -activated Cl(-) channel, ANO1 (TMEM16A), is a double-edged sword in cell proliferation and tumorigenesis

Tissue microarrays for high-throughput molecular profiling of tumor specimens

Enhanced expression of ANO1 in head and neck squamous cell carcinoma causes cell migration and correlates with poor prognosis

Transforming growth factor-β1 and -β2 in gastric precancer and cancer and roles in tumor-cell interactions with peripheral blood mononuclear cells in vitro

TMEM16A, a membrane protein associated with calcium-dependent chloride channel activity.

The novel marker, DOG1, is expressed ubiquitously in gastrointestinal stromal tumors irrespective of KIT or PDGFRA mutation status.

Substrate selectivity of epidermal growth factor-receptor ligand sheddases and their regulation by phorbol esters and calcium influx.

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