Targeting aPKC disables oncogenic signaling by both the EGFR and the proinflammatory cytokine TNFα in glioblastoma
about
Additive effects of cherlerythrine chloride combination with erlotinib in human non-small cell lung cancer cells.Loss of the polarity protein PAR3 activates STAT3 signaling via an atypical protein kinase C (aPKC)/NF-κB/interleukin-6 (IL-6) axis in mouse mammary cells.Urotensin-II receptor stimulation of cardiac L-type Ca2+ channels requires the βγ subunits of Gi/o-protein and phosphatidylinositol 3-kinase-dependent protein kinase C β1 isoform.Expression of polarity genes in human cancer.Therapeutic control and resistance of the EGFR-driven signaling network in glioblastoma.Zeta Inhibitory Peptide Disrupts Electrostatic Interactions That Maintain Atypical Protein Kinase C in Its Active Conformation on the Scaffold p62.Phosphorylation of NR2B NMDA subunits by protein kinase C in arcuate nucleus contributes to inflammatory pain in ratsHistone Deacetylase-3/CAGE Axis Targets EGFR Signaling and Regulates the Response to Anti-Cancer DrugsProtein Scaffolds Control Localized Protein Kinase Cζ Activity.Hypertonic saline protects brain endothelial cells against hypoxia correlated to the levels of epidermal growth factor receptor and interleukin-1β.Reversing the Paradigm: Protein Kinase C as a Tumor Suppressor.Protein kinase C as a tumor suppressor.Melatonin-mediated inhibition of Purkinje neuron P-type Ca²⁺ channels in vitro induces neuronal hyperexcitability through the phosphatidylinositol 3-kinase-dependent protein kinase C delta pathway.Tumor-derived fibulin-3 activates pro-invasive NF-κB signaling in glioblastoma cells and their microenvironment.Targeting atypical protein kinase C iota reduces viability in glioblastoma stem-like cells via a notch signaling mechanism.Failure of the PTEN/aPKC/Lgl Axis Primes Formation of Adult Brain Tumours in Drosophila.Regulatory effects of miR-155 and miR-146a on repolarization and inflammatory cytokine secretion in human alveolar macrophages in vitro.aPKCζ-dependent Repression of Yap is Necessary for Functional Restoration of Irradiated Salivary Glands with IGF-1.ω-3 and ω-6 Fatty Acids Modulate Conventional and Atypical Protein Kinase C Activities in a Brain Fatty Acid Binding Protein Dependent Manner in Glioblastoma Multiforme.Protein kinase C: perfectly balanced.Macrophages and Galectin 3 Control Bacterial Burden in Acute and Subacute Murine Leptospirosis That Determines Chronic Kidney Fibrosis
P2860
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P2860
Targeting aPKC disables oncogenic signaling by both the EGFR and the proinflammatory cytokine TNFα in glioblastoma
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@ast
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@en
type
label
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@ast
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@en
prefLabel
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@ast
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@en
P2093
P2860
P1433
P1476
Targeting aPKC disables oncoge ...... cytokine TNFα in glioblastoma
@en
P2093
Andrea E Errasti
Anthony S Perry
Christopher Hulme
Daniel Gibbs
Edward K Mandell
Elisabeth J Rushing
Eugenio A Carrera-Silva
Joseph C Loftus
Justin D Dietrich
Kenneth Aldape
P2860
P356
10.1126/SCISIGNAL.2005196
P577
2014-08-12T00:00:00Z