MAP kinases couple hindbrain-derived catecholamine signals to hypothalamic adrenocortical control mechanisms during glycemia-related challenges.
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Rescuing Perishable Neuroanatomical Information from a Threatened Biodiversity Hotspot: Remote Field Methods for Brain Tissue Preservation Validated by Cytoarchitectonic Analysis, Immunohistochemistry, and X-Ray Microcomputed TomographyEstrogen-related receptor β deficiency alters body composition and response to restraint stress.The rate of fall of blood glucose determines the necessity of forebrain-projecting catecholaminergic neurons for male rat sympathoadrenal responsesRole of paraventricular nucleus-projecting norepinephrine/epinephrine neurons in acute and chronic stress.PACAP-deficient mice show attenuated corticosterone secretion and fail to develop depressive behavior during chronic social defeat stress.Limiting glucocorticoid secretion increases the anorexigenic property of Exendin-4Rapid-onset hypoglycemia suppresses Fos expression in discrete parts of the ventromedial nucleus of the hypothalamusHindbrain catecholamine neurons control rapid switching of metabolic substrate use during glucoprivation in male rats.Controlling feeding behavior by chemical or gene-directed targeting in the brain: what's so spatial about our methods?Absence of glucocorticoids augments stress-induced Mkp1 mRNA expression within the hypothalamic-pituitary-adrenal axis.Neural input is critical for arcuate hypothalamic neurons to mount intracellular signaling responses to systemic insulin and deoxyglucose challenges in male rats: implications for communication within feeding and metabolic control networksStimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons.Identifying links in the chain: the dynamic coupling of catecholamines, peptide synthesis, and peptide release in hypothalamic neuroendocrine neurons.Ascending mechanisms of stress integration: Implications for brainstem regulation of neuroendocrine and behavioral stress responses.Intact catecholamine inputs to the forebrain are required for appropriate regulation of corticotrophin-releasing hormone and vasopressin gene expression by corticosterone in the rat paraventricular nucleusIs there cross talk between portal and hypothalamic glucose-sensing circuits?Social transmission and buffering of synaptic changes after stress.Catecholaminergic Projections into an Interconnected Forebrain Network Control the Sensitivity of Male Rats to Diet-Induced Obesity.Neurotransmitter Diversity In Pre-Synaptic Terminals Located in the Parvicellular Neuroendocrine Paraventricular Nucleus of the Rat and Mouse Hypothalamus.
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P2860
MAP kinases couple hindbrain-derived catecholamine signals to hypothalamic adrenocortical control mechanisms during glycemia-related challenges.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
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2011年论文
@zh-cn
name
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@ast
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@en
type
label
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@ast
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@en
prefLabel
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@ast
MAP kinases couple hindbrain-d ...... g glycemia-related challenges.
@en
P2093
P2860
P50
P1476
MAP kinases couple hindbrain-d ...... ng glycemia-related challenges
@en
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Alan G Watts
Graciela Sanchez-Watts
Kimberly L Kaminski
Todd A Ponzio
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P304
18479-18491
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10.1523/JNEUROSCI.4785-11.2011
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P577
2011-12-01T00:00:00Z