Functional repression of cAMP response element in 6-hydroxydopamine-treated neuronal cells. - Wikidata - wikimedia - TriplyDB

Functional repression of cAMP response element in 6-hydroxydopamine-treated neuronal cells.

Functional repression of cAMP response element in 6-hydroxydopamine-treated neuronal cells.

http://www.wikidata.org/entity/Q35809297

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Cardiolipin externalization to the outer mitochondrial membrane acts as an elimination signal for mitophagy in neuronal cells Nuclear transport, oxidative stress, and neurodegeneration Regulation of the autophagy protein LC3 by phosphorylation Role of protein kinase A in regulating mitochondrial function and neuronal development: implications to neurodegenerative diseases How Parkinsonian toxins dysregulate the autophagy machinery Glucose-dependent insulinotropic polypeptide-mediated up-regulation of beta-cell antiapoptotic Bcl-2 gene expression is coordinated by cyclic AMP (cAMP) response element binding protein (CREB) and cAMP-responsive CREB coactivator 2 Altered transcription factor trafficking in oxidatively-stressed neuronal cells.Chronic NMDA administration to rats increases brain pro-apoptotic factors while decreasing anti-Apoptotic factors and causes cell death.Mitochondrial quality control: insights on how Parkinson's disease related genes PINK1, parkin, and Omi/HtrA2 interact to maintain mitochondrial homeostasis.Decreased SIRT2 activity leads to altered microtubule dynamics in oxidatively-stressed neuronal cells: implications for Parkinson's disease.Reactive oxygen/nitrogen species and their functional correlations in neurodegenerative diseases.Enhanced glutamate, IP3 and cAMP activity in the cerebral cortex of unilateral 6-hydroxydopamine induced Parkinson's rats: effect of 5-HT, GABA and bone marrow cell supplementation.Cytoplasmic location of α1A voltage-gated calcium channel C-terminal fragment (Cav2.1-CTF) aggregate is sufficient to cause cell death.Efficacy of lovastatin on learning and memory deficits caused by chronic intermittent hypoxia-hypercapnia: through regulation of NR2B-containing NMDA receptor-ERK pathway.Mitochondrially localized PKA reverses mitochondrial pathology and dysfunction in a cellular model of Parkinson's disease.Formation of dopamine adducts derived from brain polyunsaturated fatty acids: mechanism for Parkinson disease Autophagic stress in neuronal injury and disease.6-Hydroxydopamine induces mitochondrial ERK activation.Identification of embryonic stem cell-derived midbrain dopaminergic neurons for engraftment Location, location, location: altered transcription factor trafficking in neurodegeneration.Expression of Nrf2 in neurodegenerative diseases Autophagy in neuroprotection and neurodegeneration: A question of balance.Mitochondrially localized ERK2 regulates mitophagy and autophagic cell stress: implications for Parkinson's disease.Chronic administration of mood stabilizers upregulates BDNF and bcl-2 expression levels in rat frontal cortex.TLR4 signaling in VTA dopaminergic neurons regulates impulsivity through tyrosine hydroxylase modulation.Mitochondrial kinases in Parkinson's disease: converging insights from neurotoxin and genetic models Tickled PINK1: mitochondrial homeostasis and autophagy in recessive Parkinsonism.Back to the tubule: microtubule dynamics in Parkinson's disease.CREB: a multifaceted regulator of neuronal plasticity and protection.Therapeutic use of omega-3 fatty acids in bipolar disorder.Gene regulation as a potential avenue for the treatment of neurodegenerative disorders.Mutant Huntingtin Disrupts the Nuclear Pore Complex.Can Cyclic Nucleotide Phosphodiesterase Inhibitors Be Drugs for Parkinson's Disease?Neuroprotective effects of nicotinamide N-methyltransferase and its metabolite 1-methylnicotinamide.The role of Ca2+ signaling in Parkinson's disease.Toxicity of 6-hydroxydopamine: live cell imaging of cytoplasmic redox flux.Synucleins and Gene Expression: Ramblers in a Crowd or Cops Regulating Traffic?Long-term exposure of 3T3 fibroblast cells to endocrine disruptors alters sensitivity to oxidative injury.PINK1 regulates mitochondrial trafficking in dendrites of cortical neurons through mitochondrial PKA.Lamotrigine Reduces β-Site AβPP-Cleaving Enzyme 1 Protein Levels Through Induction of Autophagy.

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P2860

Functional repression of cAMP response element in 6-hydroxydopamine-treated neuronal cells.

http://www.wikidata.org/entity/Q35809297

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

Functional repression of cAMP ...... pamine-treated neuronal cells.

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Functional repression of cAMP ...... pamine-treated neuronal cells.

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type

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Functional repression of cAMP ...... pamine-treated neuronal cells.

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Functional repression of cAMP ...... pamine-treated neuronal cells.

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prefLabel

Functional repression of cAMP ...... pamine-treated neuronal cells.

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Functional repression of cAMP ...... pamine-treated neuronal cells.

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Journal of Biological Chemistry

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Functional repression of cAMP ...... opamine-treated neuronal cells

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P2093

Elisabeth M Chalovich

http://www.w3.org/2001/XMLSchema#string

Jian-hui Zhu

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John Caltagarone

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Robert Bowser

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P2860

Fetal Alz-50 clone 1, a novel zinc finger protein, binds a specific DNA sequence and acts as a transcriptional regulator

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Sustained extracellular signal-regulated kinase activation by 6-hydroxydopamine: implications for Parkinson's disease

JNK2 translocates to the mitochondria and mediates cytochrome c release in PC12 cells in response to 6-hydroxydopamine

Mitochondrial cyclic AMP response element-binding protein (CREB) mediates mitochondrial gene expression and neuronal survival

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17870-17881

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scholarly article

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10.1074/JBC.M602632200

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26

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281

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Charleen T. Chu

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2006-04-18T00:00:00Z

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16621793

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1876691

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