KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors. - Wikidata - wikimedia - TriplyDB

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

http://www.wikidata.org/entity/Q35809846

about

Targeted therapy of gastrointestinal stromal tumours Hyperplasia of interstitial cells of cajal in sprouty homolog 4 deficient mice KIT mutations are common in incidental gastrointestinal stromal tumors one centimeter or less in size.A great majority of GISTs with PDGFRA mutations represent gastric tumors of low or no malignant potential.Germline mutations of KIT in gastrointestinal stromal tumor (GIST) and mastocytosis Differentially expressed proteins in gastrointestinal stromal tumors with KIT and PDGFRA mutations ALK rearrangement in sickle cell trait-associated renal medullary carcinoma The complexity of KIT gene mutations and chromosome rearrangements and their clinical correlation in gastrointestinal stromal (pacemaker cell) tumors The histopathological differential diagnosis of gastrointestinal stromal tumours CD117+ small cell lung cancer lacks the asp 816-->val point mutation in exon 17.Surgical treatment and prognostic analysis for gastrointestinal stromal tumors (GISTs) of the small intestine: before the era of imatinib mesylate Severe paraneoplastic hypoglycemia in a patient with a gastrointestinal stromal tumor with an exon 9 mutation: a case report.Chromosome copy number changes carry prognostic information independent of KIT/PDGFRA point mutations in gastrointestinal stromal tumors Gastrointestinal stromal tumor and its targeted therapeutics.Malignant gastrointestinal stromal tumor of the gallbladder.Small gastrointestinal stromal tumor concomitant with early gastric cancer: a case report.Synthesis and positron emission tomography studies of carbon-11-labeled imatinib (Gleevec).Repression of c-kit and its downstream substrates by GATA-1 inhibits cell proliferation during erythroid maturation C-kit and PDGFRA gene mutations in triple negative breast cancer.Adjuvant imatinib mesylate after resection of localised, primary gastrointestinal stromal tumour: a randomised, double-blind, placebo-controlled trial.Analysis of gene expression profiles of soft tissue sarcoma using a combination of knowledge-based filtering with integration of multiple statistics.Gastrointestinal stromal tumors (GIST): C-kit mutations, CD117 expression, differential diagnosis and targeted cancer therapy with Imatinib.Gastrointestinal stromal tumors (GIST): a model for molecule-based diagnosis and treatment of solid tumors.Updates on the management of gastrointestinal stromal tumors.Gastrointestinal stromal tumors.Prevalence of c-KIT mutations in gonadoblastoma and dysgerminomas of patients with disorders of sex development (DSD) and ovarian dysgerminomas.Preclinical evaluation of KIT/PDGFRA and mTOR inhibitors in gastrointestinal stromal tumors using small animal FDG PET.Gastrointestinal stromal tumors (GISTs) with KIT and PDGFRA mutations have distinct gene expression profiles.Correlation of KIT and platelet-derived growth factor receptor alpha mutations with gene activation and expression profiles in gastrointestinal stromal tumors.Array comparative genomic hybridization analysis of chromosomal imbalances and their target genes in gastrointestinal stromal tumors.Ménétrier disease and gastrointestinal stromal tumors: hyperproliferative disorders of the stomach.Management of resectable gastrointestinal stromal tumor.The role of mutational analysis of KIT and PDGFRA in gastrointestinal stromal tumors in a clinical setting.Diagnosis of gastrointestinal stromal tumors: a consensus approach.Gastrointestinal stromal tumors: chemotherapy and imatinib.Gastrointestinal stromal tumors: rationale for surgical adjuvant trials with imatinib.Gastrointestinal stromal tumor - an evolving concept.The GIST of targeted cancer therapy: a tumor (gastrointestinal stromal tumor), a mutated gene (c-kit), and a molecular inhibitor (STI571).Kaposi's sarcoma-associated herpesvirus-induced upregulation of the c-kit proto-oncogene, as identified by gene expression profiling, is essential for the transformation of endothelial cells Membrane-to-nucleus signaling links insulin-like growth factor-1- and stem cell factor-activated pathways.

P2860

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P2860

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

http://www.wikidata.org/entity/Q35809846

description

2000 nî lūn-bûn

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年论文

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2000年论文

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2000年论文

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name

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@ast

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@en

type

label

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@ast

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@en

prefLabel

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@ast

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@en

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The American Journal of Pathology

P1476

KIT extracellular and kinase domain mutations in gastrointestinal stromal tumors.

@en

P2093

B P Rubin

http://www.w3.org/2001/XMLSchema#string

C D Fletcher

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C J Chen

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G Demetri

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J A Fletcher

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M L Lux

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S Singer

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S Xiao

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T L Biase

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T Maclure

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P2860

Effect of c-kit mutation on prognosis of gastrointestinal stromal tumors.

Gain-of-function mutations of c-kit in human gastrointestinal stromal tumors

Substitution of an aspartic acid results in constitutive activation of c-kit receptor tyrosine kinase in a rat tumor mast cell line RBL-2H3

Constitutive activation of fibroblast growth factor receptor 3 by mutations responsible for the lethal skeletal dysplasia thanatophoric dysplasia type I

Activating mutations in the extracellular domain of the fibroblast growth factor receptor 2 function by disruption of the disulfide bond in the third immunoglobulin-like domain

FGFR activation in skeletal disorders: too much of a good thing

Mutations of c-kit JM domain are found in a minority of human gastrointestinal stromal tumors.

Activating c-kit gene mutations in human germ cell tumors

Disturbed intestinal movement, bile reflux to the stomach, and deficiency of c-kit-expressing cells in Ws/Ws mutant rats.

W/kit gene required for interstitial cells of Cajal and for intestinal pacemaker activity.

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791-795

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10.1016/S0002-9440(10)64946-2

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1876850

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