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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

http://www.wikidata.org/entity/Q35813671

about

Platelets in Pulmonary Immune Responses and Inflammatory Lung Diseases Activation states of blood eosinophils in asthma Platelets in patients with aspirin-exacerbated respiratory disease Eosinophil Activation Status in Separate Compartments and Association with Asthma In situ re-endothelialization via multifunctional nanoscaffolds.Platelet-driven leukotriene C4-mediated airway inflammation in mice is aspirin-sensitive and depends on T prostanoid receptors.Platelets from Asthmatic Individuals Show Less Reliance on Glycolysis Current and future biomarkers in allergic asthma.Biomarkers of eosinophil involvement in allergic and eosinophilic diseases: review of phenotypic and serum markers including a novel assay to quantify levels of soluble Siglec-8 Stage-dependent association of BDNF and TGF-β1 with lung function in stable COPD.Anti-IL-5 attenuates activation and surface density of β(2) -integrins on circulating eosinophils after segmental antigen challenge.Integrin activation States and eosinophil recruitment in asthma.α(M)β(2) integrin-mediated adhesion and motility of IL-5-stimulated eosinophils on periostin.Platelets in lung biology.Markers of vascular perturbation correlate with airway structural change in asthma.The Peripheral Blood Eosinophil Proteome.In utero exposure to second-hand smoke aggravates the response to ovalbumin in adult mice.Platelets and allergic inflammation.Platelet-Eosinophil Interactions As a Potential Therapeutic Target in Allergic Inflammation and Asthma.Cutting edge: Leukotriene C4 activates mouse platelets in plasma exclusively through the type 2 cysteinyl leukotriene receptor.Update in asthma 2012.Platelets coming of age: implications for our understanding of allergic inflammation.Severe bronchial asthma in children: a review of novel biomarkers used as predictors of the disease.TIPE2 is negatively correlated with tissue factor and thrombospondin-1 expression in patients with bronchial asthma.Platelet Munc13-4 regulates hemostasis, thrombosis and airway inflammation.

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P2860

Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

http://www.wikidata.org/entity/Q35813671

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

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RCCM.201109-1712OC

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American Journal of Respiratory and Critical Care Medicine

P1476

Platelet activation, P-selectin, and eosinophil β1-integrin activation in asthma

@en

P2093

Deane F Mosher

http://www.w3.org/2001/XMLSchema#string

Kristin A Gunderson

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Mats W Johansson

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Nizar N Jarjour

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Shih-Tsung Han

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P2860

Platelet P-selectin levels in relation to plasma soluble P-selectin and beta-thromboglobulin levels in atrial fibrillation

Severe asthma: lessons learned from the National Heart, Lung, and Blood Institute Severe Asthma Research Program.

Identification of asthma phenotypes using cluster analysis in the Severe Asthma Research Program.

Absence of alpha 4 but not beta 2 integrins restrains development of chronic allergic asthma using mouse genetic models

Role of the platelet chemokine platelet factor 4 (PF4) in hemostasis and thrombosis.

Characterization of the severe asthma phenotype by the National Heart, Lung, and Blood Institute's Severe Asthma Research Program.

Analyses of asthma severity phenotypes and inflammatory proteins in subjects stratified by sputum granulocytes

Asthma. From bronchoconstriction to airways inflammation and remodeling.

Eosinophils: offenders or general bystanders in allergic airway disease and pulmonary immunity?

Endotyping asthma: new insights into key pathogenic mechanisms in a complex, heterogeneous disease.

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498-507

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10.1164/RCCM.201109-1712OC

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185

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William W Busse

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22227382

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3297102

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