The Mechanism of Interferon Refractoriness During Hepatitis C Virus Infection and Its Reversal with a Peroxisome Proliferator-Activated Receptor α Agonist
about
Hepatitis C Virus Driven AXL Expression Suppresses the Hepatic Type I Interferon ResponseTAM receptor tyrosine kinase function and the immunopathology of liver disease.HELZ2 Is an IFN Effector Mediating Suppression of Dengue Virus.Defective monocyte oxidative burst predicts infection in alcoholic hepatitis and is associated with reduced expression of NADPH oxidase.Analysis of TLR7, SOCS1 and ISG15 immune genes expression in the peripheral blood of responder and non-responder patients with chronic Hepatitis C.
P2860
The Mechanism of Interferon Refractoriness During Hepatitis C Virus Infection and Its Reversal with a Peroxisome Proliferator-Activated Receptor α Agonist
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
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name
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@ast
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@en
type
label
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@ast
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@en
prefLabel
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@ast
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@en
P2093
P2860
P356
P1476
The Mechanism of Interferon Re ...... r-Activated Receptor α Agonist
@en
P2093
Enoch S Tay
Jacob George
John McLauchlan
Mahsa Shahidi
Mark W Douglas
Scott A Read
P2860
P304
P356
10.1089/JIR.2014.0209
P577
2015-03-03T00:00:00Z