Inflammation induces dermal Vγ4+ γδT17 memory-like cells that travel to distant skin and accelerate secondary IL-17-driven responses.
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Recent advances in understanding psoriasisIL-17-producing γδ T cells switch migratory patterns between resting and activated states.IL-17A-producing resident memory γδ T cells orchestrate the innate immune response to secondary oral Listeria monocytogenes infectionMigratory and adhesive cues controlling innate-like lymphocyte surveillance of the pathogen-exposed surface of the lymph node.IL-1β and IL-23 Promote Extrathymic Commitment of CD27(+)CD122(-) γδ T Cells to γδT17 CellsIL-2 Shapes the Survival and Plasticity of IL-17-Producing γδ T Cells.The role of IL 23 in the treatment of psoriasis.IL-17+ γδ T cells as kick-starters of inflammation.Cardiovascular and Metabolic Diseases Comorbid with Psoriasis: Beyond the Skin.The majority of murine γδ T cells at the maternal-fetal interface in pregnancy produce IL-17.An Ocular Commensal Protects against Corneal Infection by Driving an Interleukin-17 Response from Mucosal γδ T Cells.Hapten-Specific T Cell-Mediated Skin Inflammation: Flow Cytometry Analysis of Mouse Skin Inflammatory Infiltrate.Immune-Stimulatory Effects of Rapamycin Are Mediated by Stimulation of Antitumor γδ T Cells.Three distinct developmental pathways for adaptive and two IFN-γ-producing γδ T subsets in adult thymus.Essential Role of CARD14 in Murine Experimental Psoriasis.γδ T Cells Contribute to Injury in the Developing Brain.Human papillomavirus oncoproteins induce a reorganization of epithelial-associated γδ T cells promoting tumor formation.IL-17-Secreting γδ T Cells Are Completely Dependent upon CCR6 for Homing to Inflamed Skin.Macrophages Induce Long-Term Trapping of γδ T Cells with Innate-like Properties within Secondary Lymphoid Organs in the Steady State.Proinflammatory Cytokine Environments Can Drive Interleukin-17 Overexpression by γ/δ T Cells in Systemic Juvenile Idiopathic Arthritis.Contextual control of skin immunity and inflammation by Corynebacterium.Psoriasis: A STAT3-Centric View.Dermal Vγ4+T cells enhance the IMQ-induced psoriasis-like skin inflammatidon in re-challenged mice.Clonally expanded γδ T cells protect against Staphylococcus aureus skin reinfection.γδ T cells come to stay: Innate skin memory in the Aldara model.Dermal IL-17-producing γδ T cells establish long-lived memory in the skin.Interleukin-17 in skin: psoriasis and yonder.Vγ4 T Cells Inhibit the Pro-healing Functions of Dendritic Epidermal T Cells to Delay Skin Wound Closure Through IL-17A.The Emerging Complexity of γδT17 Cells.Functions of Vγ4 T Cells and Dendritic Epidermal T Cells on Skin Wound Healing.Lymph node γδ and αβ CD8+ T cells share migratory properties.
P2860
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P2860
Inflammation induces dermal Vγ4+ γδT17 memory-like cells that travel to distant skin and accelerate secondary IL-17-driven responses.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@ast
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@en
type
label
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@ast
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@en
prefLabel
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@ast
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@en
P2093
P2860
P356
P1476
Inflammation induces dermal Vγ ...... ondary IL-17-driven responses.
@en
P2093
Elizabeth E Gray
Francisco Ramírez-Valle
Jason G Cyster
P2860
P304
P356
10.1073/PNAS.1508990112
P407
P577
2015-06-15T00:00:00Z