High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX. - Wikidata - wikimedia - TriplyDB

High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

http://www.wikidata.org/entity/Q35823810

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Multiple Functions of the Eya Phosphotyrosine Phosphatase Oxytetracycline induces DNA damage and epigenetic changes: a possible risk for human and animal health?HMGA2 promotes vasculogenic mimicry and tumor aggressiveness by upregulating Twist1 in gastric carcinoma The tyrosine kinase inhibitor imatinib prevents lung injury and death after intravenous LPS in mice.DNA damage precedes apoptosis during the regression of the interdigital tissue in vertebrate embryos.MicroRNA-3196 is inhibited by H2AX phosphorylation and attenuates lung cancer cell apoptosis by downregulating PUMA.HMGA2 plays an important role in Cr (VI)-induced autophagy.Transcriptional activation of FN1 and IL11 by HMGA2 promotes the malignant behavior of colorectal cancer.DNA damage and gene transcription: accident or necessity?Integrative proteomic analysis reveals reprograming tumor necrosis factor signaling in epithelial mesenchymal transition.DNA damage and repair regulate neuronal gene expression.Misato underlies visceral myopathy in Drosophila.Binding of high mobility group A proteins to the mammalian genome occurs as a function of AT-content.Reconstructing differentiation networks and their regulation from time series single cell expression data.Genetics of the human Y chromosome and its association with male infertility.Oleanolic acid rejuvenates testicular function through attenuating germ cell DNA damage and apoptosis via deactivation of NF-κB, p53 and p38 signalling pathways.EGFR tyrosine kinase inhibitor HS-10182 increases radiation sensitivity in non-small cell lung cancers with EGFR T790M mutation.Let-7c inhibits migration and epithelial-mesenchymal transition in head and neck squamous cell carcinoma by targeting IGF1R and HMGA2.

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High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

http://www.wikidata.org/entity/Q35823810

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

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High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

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High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

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High mobility group protein-mediated transcription requires DNA damage marker γ-H2AX.

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Aditi Mehta

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Claudia Cosentino

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Diya Hasan

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Gianni Carraro

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Julio Cordero

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Marcus Krüger

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Nihan Ozturk

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Raul Mostoslavsky

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Saverio Bellusci

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Model-based analysis of ChIP-Seq (MACS)

HMGA2 exhibits dRP/AP site cleavage activity and protects cancer cells from DNA-damage-induced cytotoxicity during chemotherapy

H3.3/H2A.Z double variant-containing nucleosomes mark 'nucleosome-free regions' of active promoters and other regulatory regions

ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks

Suppression of nonhomologous end joining repair by overexpression of HMGA2

Linking genome and proteome by mass spectrometry: large-scale identification of yeast proteins from two dimensional gels

Gene Expression Omnibus: NCBI gene expression and hybridization array data repository

Fast gapped-read alignment with Bowtie 2

FACT and the proteasome promote promoter chromatin disassembly and transcriptional initiation.

Spt16-Pob3 and the HMG protein Nhp6 combine to form the nucleosome-binding factor SPN.

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837-850

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scholarly article

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10.1038/CR.2015.67

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7

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25

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Guillermo Barreto

Carlos Sebastian

Indrabahadur Singh

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2015-06-05T00:00:00Z

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277778822

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1048808983

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