The E1 protein of human papillomavirus type 16 is dispensable for maintenance replication of the viral genome.
about
Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6The Role of the DNA Damage Response throughout the Papillomavirus Life CyclePapillomaviruses use recombination-dependent replication to vegetatively amplify their genomes in differentiated cellsRecent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Activation of NF-κB by human papillomavirus 16 E1 limits E1-dependent viral replication through degradation of E1.The Cell Cycle Timing of Human Papillomavirus DNA Replication.HPV16 and 18 genome amplification show different E4-dependence, with 16E4 enhancing E1 nuclear accumulation and replicative efficiency via its cell cycle arrest and kinase activation functions.The biology of papillomavirus latency.Highly Efficient CRISPR/Cas9-Mediated Cloning and Functional Characterization of Gastric Cancer-Derived Epstein-Barr Virus Strains.HPV-Based Screening, Triage, Treatment, and Followup Strategies in the Management of Cervical Intraepithelial Neoplasia.Current understanding of the role of the Brd4 protein in the papillomavirus lifecycle.Human papillomavirus molecular biology and disease association.The E1 proteins.Immunological characterization of a modified vaccinia virus Ankara vector expressing the human papillomavirus 16 E1 proteinInitial amplification of the HPV18 genome proceeds via two distinct replication mechanisms.Modulation of the DNA damage response during the life cycle of human papillomaviruses.Host cell restriction factors that limit transcription and replication of human papillomavirus.Human papillomavirus episome stability is reduced by aphidicolin and controlled by DNA damage response pathways.HPV16 E7 Genetic Conservation Is Critical to Carcinogenesis.Carcinogenic human papillomavirus infection.Human papilloma virus: An etiological and prognostic factor for oral cancer?A new cell culture model to genetically dissect the complete human papillomavirus life cycle.Plasmid Partitioning by Human Tumor Viruses.Keratinocyte differentiation induces APOBEC3A, 3B, and mitochondrial DNA hypermutation.
P2860
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P2860
The E1 protein of human papillomavirus type 16 is dispensable for maintenance replication of the viral genome.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
The E1 protein of human papill ...... plication of the viral genome.
@ast
The E1 protein of human papill ...... plication of the viral genome.
@en
type
label
The E1 protein of human papill ...... plication of the viral genome.
@ast
The E1 protein of human papill ...... plication of the viral genome.
@en
prefLabel
The E1 protein of human papill ...... plication of the viral genome.
@ast
The E1 protein of human papill ...... plication of the viral genome.
@en
P2093
P2860
P356
P1433
P1476
The E1 protein of human papill ...... plication of the viral genome.
@en
P2093
Kenji Yamato
Mako Narisawa-Saito
Masatoshi Fujita
Nagayasu Egawa
Shin-Ichi Ohno
Takashi Yugawa
Tohru Kiyono
Tomomi Nakahara
Yukikazu Natori
P2860
P304
P356
10.1128/JVI.06450-11
P407
P577
2012-01-11T00:00:00Z