Hepatitis C virus infection of human T lymphocytes is mediated by CD5. - Wikidata - wikimedia - TriplyDB

Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

http://www.wikidata.org/entity/Q35826843

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Hepatitis C virus molecular evolution: transmission, disease progression and antiviral therapy Direct effects of hepatitis C virus on the lymphoid cells All-in-one processing of heterogeneous human cell grafts for gene and cell therapy.Occult infection with hepatitis C virus: friend or foe?Differential expression of candidate virus receptors in human T lymphocytes prone or resistant to infection with patient-derived hepatitis C virus.Persistence of hepatitis C virus during and after otherwise clinically successful treatment of chronic hepatitis C with standard pegylated interferon α-2b and ribavirin therapy Patient-derived hepatitis C virus inhibits CD4⁺ but not CD8⁺ T lymphocyte proliferation in primary T cells.Conserved Motifs within Hepatitis C Virus Envelope (E2) RNA and Protein Independently Inhibit T Cell Activation.Persistence of Hepatitis C Virus Traces after Spontaneous Resolution of Hepatitis C Specialization of Hepatitis C Virus Envelope Glycoproteins for B Lymphocytes in Chronically Infected Patients.Seronegative hepatitis C virus infection in a child infected via mother-to-child transmission Hepatitis C virus has a genetically determined lymphotropism through co-receptor B7.2.Hepatitis C virus upregulates B-cell receptor signaling: a novel mechanism for HCV-associated B-cell lymphoproliferative disorders.Seronegative hepatitis C virus infection.Natural killer cells in hepatitis C virus infection.Exploiting scavenger receptors in cancer immunotherapy: Lessons from CD5 and SR-B1.Seronegative and occult hepatitis C virus infections in patients with hematological disorders.Hepatitis C virus (HCV) interaction with astrocytes: nonproductive infection and induction of IL-18.Hepatitis C virus load and expression of a unique subset of cellular genes in circulating lymphoid cells differentiate non-responders from responders to pegylated interferon alpha-ribavirin treatment.Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4+ and CD8+ T Lymphocytes In Vitro.Clinicopathologic characteristics and outcomes of transformed diffuse large B-cell lymphoma in hepatitis C virus-infected patients.

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P2860

Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

http://www.wikidata.org/entity/Q35826843

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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Journal of Virology

P1476

Hepatitis C virus infection of human T lymphocytes is mediated by CD5.

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P2093

Annie Y Chen

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Mohammed A Sarhan

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Tomasz I Michalak

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Tram N Q Pham

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P2860

Initiation of hepatitis C virus infection is dependent on cholesterol and cooperativity between CD81 and scavenger receptor B type I

Genetic diversity and tissue compartmentalization of the hepatitis C virus genome in blood mononuclear cells, liver, and serum from chronic hepatitis C patients.

VX-950, a Novel Hepatitis C Virus (HCV) NS3-4A Protease Inhibitor, Exhibits Potent Antiviral Activities in HCV Replicon Cells

Advantages of a single-cycle production assay to study cell culture-adaptive mutations of hepatitis C virus

Tight Junction Proteins Claudin-1 and Occludin Control Hepatitis C Virus Entry and Are Downregulated during Infection To Prevent Superinfection

The Tight Junction-Associated Protein Occludin Is Required for a Postbinding Step in Hepatitis C Virus Entry and Infection

Human occludin is a hepatitis C virus entry factor required for infection of mouse cells

Production of infectious hepatitis C virus in tissue culture from a cloned viral genome

Claudin-1 is a hepatitis C virus co-receptor required for a late step in entry

Cell entry of hepatitis C virus requires a set of co-receptors that include the CD81 tetraspanin and the SR-B1 scavenger receptor

P304

3723-3735

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scholarly article

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10.1128/JVI.06956-11

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7

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86

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2012-01-25T00:00:00Z

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22278227

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3302518

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