Activation of HIF-1α and LL-37 by commensal bacteria inhibits Candida albicans colonization. - Wikidata - wikimedia - TriplyDB

Activation of HIF-1α and LL-37 by commensal bacteria inhibits Candida albicans colonization.

Activation of HIF-1α and LL-37 by commensal bacteria inhibits Candida albicans colonization.

http://www.wikidata.org/entity/Q35832535

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How Chemotherapy Increases the Risk of Systemic Candidiasis in Cancer Patients: Current Paradigm and Future Directions Candida albicans Inhibits Pseudomonas aeruginosa Virulence through Suppression of Pyochelin and Pyoverdine Biosynthesis Experimental Models of Vaginal Candidiasis and Their Relevance to Human Candidiasis Roles of the intestinal microbiota in pathogen protection.Immunity against fungi.Antifungal Tc17 cells are durable and stable, persisting as long-lasting vaccine memory without plasticity towards IFNγ cells Role of the intestinal mucosa in acute gastrointestinal GVHD.The Fungal Mycobiome and Its Interaction with Gut Bacteria in the Host.Antibiotic-Induced Changes in the Intestinal Microbiota and Disease.Entinostat up-regulates the CAMP gene encoding LL-37 via activation of STAT3 and HIF-1α transcription factors.β-glucan Exposure on the Fungal Cell Wall Tightly Correlates with Competitive Fitness of Candida Species in the Mouse Gastrointestinal Tract.Adaptation of Candida albicans to commensalism in the gut.Cyclic mechanical stretch down-regulates cathelicidin antimicrobial peptide expression and activates a pro-inflammatory response in human bronchial epithelial cells.Interaction of Candida Species with the Skin.Fungal dysbiosis: immunity and interactions at mucosal barriers.Regulation of inflammation by microbiota interactions with the host.Severe Gut Microbiota Dysbiosis Is Associated With Poor Growth in Patients With Short Bowel Syndrome.Potential for Monitoring Gut Microbiota for Diagnosing Infections and Graft-versus-Host Disease in Cancer and Stem Cell Transplant Patients.An Ocular Commensal Protects against Corneal Infection by Driving an Interleukin-17 Response from Mucosal γδ T Cells.The microbiome in hematopoietic stem cell transplant recipients and cancer patients: Opportunities for clinical advances that reduce infection.Identifying host immune effectors critical for protection against Candida albicans infections.Metagenomic Shotgun Sequencing and Unbiased Metabolomic Profiling Identify Specific Human Gut Microbiota and Metabolites Associated with Immune Checkpoint Therapy Efficacy in Melanoma Patients Immunological Consequences of Intestinal Fungal Dysbiosis.The complexities of bacterial-fungal interactions in the mammalian gastrointestinal tract.The Transcriptional Response of Candida albicans to Weak Organic Acids, Carbon Source, and MIG1 Inactivation Unveils a Role for HGT16 in Mediating the Fungistatic Effect of Acetic Acid.The Human Gut Microbial Metabolome Modulates Fungal Growth via the TOR Signaling Pathway.Commensal Fungi Recapitulate the Protective Benefits of Intestinal Bacteria.The yeast form of the fungus Candida albicans promotes persistence in the gut of gnotobiotic mice.HIF-1alpha and infectious diseases: a new frontier for the development of new therapies.Modulating Host Signaling Pathways to Promote Resistance to Infection by Candida albicans.Host-microbe interactions: commensal fungi in the gut.Innate Immunity to Mucosal Candida Infections.Understanding the role of host immune responses in invasive candidiasis.Probiotics and antimicrobial protein and peptide levels in preterm infants.Lactobacillus paracasei CNCM I-3689 reduces vancomycin-resistant Enterococcus persistence and promotes Bacteroidetes resilience in the gut following antibiotic challenge.Barrett's esophagus is associated with a distinct oral microbiome.The Microbiome and Hematopoietic Cell Transplantation: Past, Present, and Future.Commensal Fungi in Health and Disease.Investigating Colonization of the Healthy Adult Gastrointestinal Tract by Fungi.Tipping the Balance: Adaptation in Polymicrobial Environments

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Activation of HIF-1α and LL-37 by commensal bacteria inhibits Candida albicans colonization.

http://www.wikidata.org/entity/Q35832535

description

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

@ast

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

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type

label

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

@ast

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

@en

prefLabel

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

@ast

Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

@en

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Nature Medicine

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Activation of HIF-1α and LL-37 ...... Candida albicans colonization.

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Di Fan

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Jiwoong Kim

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Laura A Coughlin

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Lora V Hooper

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Megan M Neubauer

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Min Soo Kim

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Tiffany R Simms-Waldrip

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Xiaowei Zhan

http://www.w3.org/2001/XMLSchema#string

Yang Xie

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P2860

Angiogenins: a new class of microbicidal proteins involved in innate immunity

Obesity alters gut microbial ecology

QIIME allows analysis of high-throughput community sequencing data

Symbiotic bacteria direct expression of an intestinal bactericidal lectin

The relative antibacterial activity of four penicillins

Gut flora in health and disease

Microbial-gut interactions in health and disease. Antibiotic-associated diarrhoea

Induction of colonic regulatory T cells by indigenous Clostridium species

An immunomodulatory molecule of symbiotic bacteria directs maturation of the host immune system

The microbial metabolites, short-chain fatty acids, regulate colonic Treg cell homeostasis

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4496259

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