Mechanisms of CHD5 Inactivation in neuroblastomas. - Wikidata - wikimedia - TriplyDB

Mechanisms of CHD5 Inactivation in neuroblastomas.

Mechanisms of CHD5 Inactivation in neuroblastomas.

http://www.wikidata.org/entity/Q35836491

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Chromodomain-helicase-DNA binding protein 5, 7 and pronecrotic mixed lineage kinase domain-like protein serve as potential prognostic biomarkers in patients with resected pancreatic adenocarcinomas Role of CHD5 in human cancers: 10 years later Chd5 requires PHD-mediated histone 3 binding for tumor suppression Research progress of neuroblastoma related gene variations.Differential expression and sex chromosome association of CHD3/4 and CHD5 during spermatogenesis Architects of the genome: CHD dysfunction in cancer, developmental disorders and neurological syndromes Therapeutic targets for neuroblastomas.The tumour suppressor CHD5 forms a NuRD-type chromatin remodelling complex.Retinoic acid-induced CHD5 upregulation and neuronal differentiation of neuroblastoma.Epigenetic dysregulation in laryngeal squamous cell carcinoma.The chromatin remodeler chd5 is necessary for proper head development during embryogenesis of Danio rerio.Chromodomain helicase DNA binding protein 5 plays a tumor suppressor role in human breast cancer Role of microRNAs in epigenetic silencing of the CHD5 tumor suppressor gene in neuroblastomas.Exome and deep sequencing of clinically aggressive neuroblastoma reveal somatic mutations that affect key pathways involved in cancer progression Decreased expression of the CHD5 gene and its clinicopathological significance in breast cancer: Correlation with aberrant DNA methylation.Silencing of CHD5 gene by promoter methylation in leukemia.Apoptotic cell death in neuroblastoma.The Chromodomain Helicase DNA-Binding Chromatin Remodelers: Family Traits that Protect from and Promote Cancer.Children's Oncology Group's 2013 blueprint for research: neuroblastoma.The role of genetic and epigenetic alterations in neuroblastoma disease pathogenesis.The roles and regulation of Polycomb complexes in neural development.Mutational dynamics between primary and relapse neuroblastomas.The tumor suppressor Chd5 is induced during neuronal differentiation in the developing mouse brain.p53, SKP2, and DKK3 as MYCN Target Genes and Their Potential Therapeutic Significance.Low CHD5 expression activates the DNA damage response and predicts poor outcome in patients undergoing adjuvant therapy for resected pancreatic cancer.Decreased expression of chromodomain helicase DNA-binding protein 5 is an unfavorable prognostic marker in patients with primary gallbladder carcinoma.CHD5 a tumour suppressor is epigenetically silenced in hepatocellular carcinoma.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

http://www.wikidata.org/entity/Q35836491

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Mechanisms of CHD5 Inactivation in neuroblastomas.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Clinical Cancer Research

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Mechanisms of CHD5 Inactivation in neuroblastomas.

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Garrett M Brodeur

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Hiroshi Koyama

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Jennifer E Light

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Mayumi Higashi

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Patrick W McGrady

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Tiangang Zhuang

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Venkatadri Kolla

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Wendy B London

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Characterization of the CHD family of proteins

A region of consistent deletion in neuroblastoma maps within human chromosome 1p36.2-36.3

Terminology and morphologic criteria of neuroblastic tumors: recommendations by the International Neuroblastoma Pathology Committee

Amplification of N-myc in untreated human neuroblastomas correlates with advanced disease stage

CHD5 is a tumor suppressor at human 1p36

Induction of apoptosis in fibroblasts by c-myc protein

Global MYCN transcription factor binding analysis in neuroblastoma reveals association with distinct E-box motifs and regions of DNA hypermethylation.

Inferring a tumor progression model for neuroblastoma from genomic data.

Genome-wide analysis of neuroblastomas using high-density single nucleotide polymorphism arrays.

Distinct high-profile methylated genes in colorectal cancer

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