Cxcr3 and its ligand CXCL10 are expressed by inflammatory cells infiltrating lung allografts and mediate chemotaxis of T cells at sites of rejection.
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CXCR3/CXCL10 interactions in the development of hypersensitivity pneumonitisCXCR3/CXCL10 expression in the synovium of children with juvenile idiopathic arthritis.Effector mechanisms of rejectionPulmonary vaccination as a novel treatment for lung fibrosisSynthetic double-stranded RNAs are adjuvants for the induction of T helper 1 and humoral immune responses to human papillomavirus in rhesus macaquesMicrofluidic mazes to characterize T-cell exploration patterns following activation in vitro.Pin1 modulates the type 1 immune responseCXCR3 ligands contribute to Th1-induced inflammation but not to homing of Th1 cells into the lung.CXCL9 and CXCL10 accelerate acute transplant rejection mediated by alloreactive memory T cells in a mouse retransplantation model.CXCL10 and IL-6: Markers of two different forms of intra-amniotic inflammation in preterm labor.Human cytomegalovirus IE1 protein elicits a type II interferon-like host cell response that depends on activated STAT1 but not interferon-γCXC chemokine ligand (CXCL) 9 and CXCL10 are antagonistic costimulation molecules during the priming of alloreactive T cell effectorsMaternal floor infarction/massive perivillous fibrin deposition: a manifestation of maternal antifetal rejection?A selective and potent CXCR3 antagonist SCH 546738 attenuates the development of autoimmune diseases and delays graft rejection.Interferon-inducible protein 10, but not monokine induced by gamma interferon, promotes protective type 1 immunity in murine Klebsiella pneumoniae pneumonia.Intranasal administration of dsRNA analog poly(I:C) induces interferon-α receptor-dependent accumulation of antigen experienced T cells in the airways.CXCR3 in T cell function.Innate immune activation potentiates alloimmune lung disease independent of chemokine (C-X-C motif) receptor 3.Endothelial injury: cause and effect of alloimmune inflammation.The immunosuppressant Protosappanin A diminished recipient T cell migration into allograft via inhibition of IP-10 in rat heart transplant.Chemokine receptor-dependent alloresponses.Chemokines and their receptors in human renal allotransplantation.CXCR3 chemokine receptor immunoreactivity in primary cutaneous malignant melanoma: correlation with clinicopathological prognostic factorsLong-term exposure of chemokine CXCL10 causes bronchiolitis-like inflammation.Cumulative exposure to gamma interferon-dependent chemokines CXCL9 and CXCL10 correlates with worse outcome after lung transplantEffect of different interferonα2 preparations on IP10 and ET-1 release from human lung cells.Recipient iNOS but not eNOS deficiency reduces luminal narrowing in tracheal allograftsBLT1-mediated T cell trafficking is critical for rejection and obliterative bronchiolitis after lung transplantation.CD4 T cell depletion exacerbates acute Mycobacterium tuberculosis while reactivation of latent infection is dependent on severity of tissue depletion in cynomolgus macaques.Transforming growth factor-beta 1 delivery from microporous scaffolds decreases inflammation post-implant and enhances function of transplanted isletsBronchoalveolar lavage as a tool to predict, diagnose and understand bronchiolitis obliterans syndrome.Chronic inflammation of the placenta: definition, classification, pathogenesis, and clinical significance.CXCL10/IP-10: a missing link between inflammation and anti-angiogenesis in preeclampsia?Therapeutic efficacy of CXCR3 blockade in an experimental model of severe sepsis.Chemokines and transplant vasculopathy.CXCR3 deficiency increases susceptibility to genital herpes simplex virus type 2 infection: Uncoupling of CD8+ T-cell effector function but not migration.Inhibiting CXCR3-dependent CD8+ T cell trafficking enhances tolerance induction in a mouse model of lung rejectionCXCR3 ligands are associated with the continuum of diffuse alveolar damage to chronic lung allograft dysfunction.Elevated CXCL10 (IP-10) in bronchoalveolar lavage fluid is associated with acute cellular rejection after human lung transplantation.Characterization of the fetal blood transcriptome and proteome in maternal anti-fetal rejection: evidence of a distinct and novel type of human fetal systemic inflammatory response.
P2860
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P2860
Cxcr3 and its ligand CXCL10 are expressed by inflammatory cells infiltrating lung allografts and mediate chemotaxis of T cells at sites of rejection.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@ast
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@en
type
label
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@ast
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@en
prefLabel
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@ast
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@en
P2093
P2860
P1476
Cxcr3 and its ligand CXCL10 ar ...... T cells at sites of rejection.
@en
P2093
C Agostini
F Calabrese
G Semenzato
P2860
P304
P356
10.1016/S0002-9440(10)64126-0
P407
P577
2001-05-01T00:00:00Z