Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice - Wikidata - wikimedia - TriplyDB

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

http://www.wikidata.org/entity/Q35843423

about

New Molecular Insights of Insulin in Diabetic Cardiomyopathy Inhibition of MicroRNA 195 Prevents Apoptosis and Multiple-Organ Injury in Mouse Models of Sepsis.Epigenetic regulation in heart failure.BDNF-mediates Down-regulation of MicroRNA-195 Inhibits Ischemic Cardiac Apoptosis in Rats.MicroRNA-195 regulates proliferation, migration, angiogenesis and autophagy of endothelial progenitor cells by targeting GABARAPL1 Disruption of calpain reduces lipotoxicity-induced cardiac injury by preventing endoplasmic reticulum stress.Role of microRNAs in the pathogenesis of diabetic cardiomyopathy.Diabetic Cardiomyopathy: An Immunometabolic Perspective.Epigenetics and Cardiovascular Disease in Diabetes.α-Linolenic acid and exercise training independently, and additively, decrease blood pressure and prevent diastolic dysfunction in obese Zucker rats.Effects of miRNAs on myocardial apoptosis by modulating mitochondria related proteins.Macro- and microvascular endothelial dysfunction in diabetes.Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy.Pathophysiological Fundamentals of Diabetic Cardiomyopathy.Role of microRNA in diabetic cardiomyopathy: From mechanism to intervention.Concise Review: Challenges in Regenerating the Diabetic Heart: A Comprehensive Review.Vesicle miR-195 derived from Endothelial Cells Inhibits Expression of Serotonin Transporter in Vessel Smooth Muscle Cells.MicroRNA as a Therapeutic Target in Cardiac Remodeling.Roles and Mechanisms of Herbal Medicine for Diabetic Cardiomyopathy: Current Status and Perspective.Pathological Effects of Exosomes in Mediating Diabetic Cardiomyopathy.Inhibition of miR-186-5p contributes to high glucose-induced injury in AC16 cardiomyocytes.Molecular mechanism of diabetic cardiomyopathy and modulation of microRNA function by synthetic oligonucleotides.miR-30c and miR-181a synergistically modulate p53-p21 pathway in diabetes induced cardiac hypertrophy.Deciphering Non-coding RNAs in Cardiovascular Health and Disease.HIF-1, Metabolism, and Diabetes in the Embryonic and Adult Heart Targeting miRNA for Therapy of Juvenile and Adult Diabetic Cardiomyopathy

P2860

Q26752678-A010B68A-6FB0-46F8-BDEF-61FF5F6F6EB5 Q36816577-CEFF619F-B508-4BB0-895C-24A367C5F052 Q37114038-DC3D38A0-183D-4925-A267-309BABE1D97C Q37148557-9CF54728-98CE-4EE6-9C16-4A7D3BD7D33E Q37340099-E453C5E6-4665-417F-B0CC-2F483196D745 Q37344309-BC2F678A-6AEC-4B5E-A8D0-936704BEDCAA Q37700697-74B196F6-AF79-4D22-B93F-679630EC0B7C Q37741735-603ACB35-9B3B-4482-A9A9-F4C24963095C Q38605096-1DB2CA0B-2A9B-4916-AF54-A53E4F8C8CD5 Q38874431-C53F847C-09B8-4DA5-B3AB-1DFBA7082C3D Q39051228-970CFFE6-E8F6-430E-B292-272EE5B35D9F Q39065434-2F25B278-F8C7-4F00-A306-7B343E758B2A Q39190427-765A2D18-59DD-4B02-99CC-8FE6802B286E Q39195672-8F1805B9-0FB4-4EC7-A67F-DC964B415104 Q39201286-20CD64F1-9451-496A-BDD6-BEB603013451 Q39390326-89DD0018-B71A-4EA5-8713-3BF51A74728F Q42317706-5F4E76F5-94CC-4573-86CB-2276B334697F Q44732086-E567BAAF-EBB8-48FF-A17E-54799E6BBD9F Q46946033-CBB18419-87CB-4BA3-A61A-D395FE3DB629 Q47763435-C88B56EA-D98D-455E-A289-931962190950 Q49335803-3EEEEAAD-EB9B-4DF3-B28F-89C7C21BAFCA Q52642353-F7207F3E-66C4-4299-9D94-91DD0029B771 Q53095027-3F01FC5E-5B3E-4464-A51E-937A3FE21609 Q55668859-D7057A03-55F3-4B98-8BF5-5D57BBFC1005 Q58777512-0DC52F84-178C-454E-9506-69DEB90F0DD2 Q58998162-17C8A6B6-6EBA-4DB0-88A5-6149E1FE3D30

P2860

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

http://www.wikidata.org/entity/Q35843423

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@ast

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@en

type

label

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@ast

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@en

prefLabel

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@ast

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@en

P1433

Q35843423-3B4112E2-AEAB-4B73-835B-CDB469EC9DDB

P1476

Q35843423-44C82499-6C94-4F8E-8D31-3A558353687A

P2093

Q35843423-0936F84D-28C0-4842-A73F-D013D3CFEB4C

Q35843423-3AAF2E28-4069-4EA7-A45D-D3EE0D80A1F4

Q35843423-94917E10-685A-461D-BED2-35AE200B585C

Q35843423-A89888E0-CAB6-4674-84E8-FBB38FA9DB7B

Q35843423-B1B609AE-74E6-45D5-8966-7D31FB1376F1

Q35843423-B4C7972F-D451-45E3-8181-DCD441148A92

Q35843423-C2D1A5F9-19DA-4016-B6E7-BEB00AA69308

Q35843423-DE738869-97F4-4819-B82A-48E304A35314

Q35843423-F3FF54D1-0EFE-47C1-B35A-3C4695730A95

Q35843423-F7350E13-D5AD-45AB-96E4-2BB9A738FF09

P2860

Q35843423-019E4070-E8E8-4E1B-874F-E162D8D9E57D

Q35843423-05D3041B-DE94-4DBF-A3C8-A79AA42CFEE2

Q35843423-170B5B0B-6CDE-405C-B126-003C5EBC1E10

Q35843423-24ABCBC5-A10F-4BB0-96F9-EFAE90EAE513

Q35843423-2677F3C2-9F6F-4087-8503-B75B11CC00F3

Q35843423-28DB3660-2D88-4974-A452-A67EA42E69B0

Q35843423-2BE3DE0D-648A-4E5D-ACDB-826442139847

Q35843423-2E6B8510-BE4B-41A9-8B31-82CEB2A053D9

Q35843423-3EE15CAC-F0A0-4B66-A339-CC297DAE0B72

Q35843423-42AF9738-EB2A-449F-A426-59A9211B0933

P2888

Q35843423-86ED6CC9-65FA-4016-9975-565A9534F94F

P304

Q35843423-970AC6BD-0121-412A-B00C-F81E8FAD6657

P31

Q35843423-915E2CC9-BF8A-460D-BDC4-D5ECEB0EBCDC

P356

Q35843423-1B2D7613-F1C1-4B44-83D7-BE5C7CFAAC92

P433

Q35843423-F8EB2596-B3D3-4BAB-88CD-8260F27262CA

P478

Q35843423-D0AAF426-F1B8-48B9-976B-B74A5FB1DE0B

P577

Q35843423-AAEB199E-7E8F-4579-8133-E4EFEED89957

P5875

Q35843423-20091746-01A6-4AA7-AD07-2944B4A414CC

P698

Q35843423-28A83860-0096-4BC0-A087-E750565FB12D

P932

Q35843423-B96DA3EA-7FEF-486A-8FFA-C0EEC81DE848

P356

S00125-015-3622-8

P1433

P1476

Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice

@en

P2093

Dong Zheng

http://www.w3.org/2001/XMLSchema#string

Grace Wang

http://www.w3.org/2001/XMLSchema#string

Guo-Chang Fan

http://www.w3.org/2001/XMLSchema#string

James C Lacefield

http://www.w3.org/2001/XMLSchema#string

Jian Ma

http://www.w3.org/2001/XMLSchema#string

Jianmin Li

http://www.w3.org/2001/XMLSchema#string

Minghui Li

http://www.w3.org/2001/XMLSchema#string

Rui Ni

http://www.w3.org/2001/XMLSchema#string

Ruizhen Chen

http://www.w3.org/2001/XMLSchema#string

Tianqing Peng

http://www.w3.org/2001/XMLSchema#string

P2860

Micro-RNA-195 and -451 regulate the LKB1/AMPK signaling axis by targeting MO25

miR-15 and miR-16 induce apoptosis by targeting BCL2

MiR-15 family regulates postnatal mitotic arrest of cardiomyocytes

A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure

The functions of animal microRNAs

bantam encodes a developmentally regulated microRNA that controls cell proliferation and regulates the proapoptotic gene hid in Drosophila

Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript

Mechanisms of gene silencing by double-stranded RNA

Intravenous gene therapy with PIM-1 via a cardiotropic viral vector halts the progression of diabetic cardiomyopathy through promotion of prosurvival signaling

mir-17-92 cluster is required for and sufficient to induce cardiomyocyte proliferation in postnatal and adult hearts

P2888

s00125-015-3622-8

P304

1949-1958

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S00125-015-3622-8

http://www.w3.org/2001/XMLSchema#string

P433

8

http://www.w3.org/2001/XMLSchema#string

P478

58

http://www.w3.org/2001/XMLSchema#string

P577

2015-05-21T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

277080968

http://www.w3.org/2001/XMLSchema#string

P698

25994075

http://www.w3.org/2001/XMLSchema#string

P932

4499474

http://www.w3.org/2001/XMLSchema#string