Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest. - Wikidata - wikimedia - TriplyDB

Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

http://www.wikidata.org/entity/Q35846720

about

Drug therapy in cardiac arrest: a review of the literature Glucocorticoids as an emerging pharmacologic agent for cardiopulmonary resuscitation.Dynamin-related protein 1 as a therapeutic target in cardiac arrest Estrogen fails to facilitate resuscitation from ventricular fibrillation in male rats.Resveratrol Improves Survival and Prolongs Life Following Hemorrhagic Shock Persistently Altered Brain Mitochondrial Bioenergetics After Apparently Successful Resuscitation From Cardiac Arrest.Impaired cerebral mitochondrial oxidative phosphorylation function in a rat model of ventricular fibrillation and cardiopulmonary resuscitation.Adverse postresuscitation myocardial effects elicited by buffer-induced alkalemia ameliorated by NHE-1 inhibition in a rat model of ventricular fibrillation.Early Effects of Prolonged Cardiac Arrest and Ischemic Postconditioning during Cardiopulmonary Resuscitation on Cardiac and Brain Mitochondrial Function in Pigs.Cerebral mitochondrial dysfunction associated with deep hypothermic circulatory arrest in neonatal swine.Neuroprotective effect of Shenfu Injection () following cardiac arrest in pig correlates with improved mitochondrial function and cerebral glucose uptake.

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

http://www.wikidata.org/entity/Q35846720

description

2012 nî lūn-bûn

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

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J.CCC.2012.02.001

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Critical Care Clinics

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Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

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Jeejabai Radhakrishnan

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Raúl J Gazmuri

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P2860

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

Erythropoietin prevents motor neuron apoptosis and neurologic disability in experimental spinal cord ischemic injury

Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria

Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress

Activation of aldehyde dehydrogenase-2 reduces ischemic damage to the heart

Mammalian caspases: structure, activation, substrates, and functions during apoptosis

OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion

Erythropoietin is a paracrine mediator of ischemic tolerance in the brain: evidence from an in vitro model

Mitochondria and apoptosis

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