The extracellular adherence protein (Eap) of Staphylococcus aureus inhibits wound healing by interfering with host defense and repair mechanisms. - Wikidata - wikimedia - TriplyDB

The extracellular adherence protein (Eap) of Staphylococcus aureus inhibits wound healing by interfering with host defense and repair mechanisms.

The extracellular adherence protein (Eap) of Staphylococcus aureus inhibits wound healing by interfering with host defense and repair mechanisms.

http://www.wikidata.org/entity/Q35848578

about

Vascular endothelial growth factor (VEGF)-induced up-regulation of CCN1 in osteoblasts mediates proangiogenic activities in endothelial cells and promotes fracture healing Host-pathogen interactions between the skin and Staphylococcus aureus.Surfactant protein A (SP-A)-mediated clearance of Staphylococcus aureus involves binding of SP-A to the staphylococcal adhesin eap and the macrophage receptors SP-A receptor 210 and scavenger receptor class A Staphylococcus aureus extracellular adherence protein triggers TNFα release, promoting attachment to endothelial cells via protein A.Inhibition of breast cancer cell adhesion and bone metastasis by the extracellular adherence protein of Staphylococcus aureus An Update on Clinical Burden, Diagnostic Tools, and Therapeutic Options of Staphylococcus aureus Collagen and hyaluronan at wound sites influence early polymicrobial biofilm adhesive events.Staphylococcus aureus: an introduction Regulation of tissue infiltration by neutrophils: role of integrin α3β1 and other factors.Leptospiral outer membrane protein microarray, a novel approach to identification of host ligand-binding proteins.The Staphylococcus aureus extracellular adherence protein (Eap) adopts an elongated but structured conformation in solution.Interaction of the microbiome with the innate immune response in chronic wounds.Gene therapy and wound healing Molecular mechanisms of Staphylococcus aureus iron acquisition.Microbiology of the skin and the role of biofilms in infection.Innate and adaptive immune responses against Staphylococcus aureus skin infections.Leucocyte recruitment in inflammation and novel endogenous negative regulators thereof.Mapping the transcription start points of the Staphylococcus aureus eap, emp, and vwb promoters reveals a conserved octanucleotide sequence that is essential for expression of these genes.Shaping of cutaneous function by encounters with commensals.Fibrinogen Is at the Interface of Host Defense and Pathogen Virulence in Staphylococcus aureus Infection.Novel Evasion Mechanisms of the Classical Complement Pathway.Smart Dressings Based on Nanostructured Fibers Containing Natural Origin Antimicrobial, Anti-Inflammatory, and Regenerative Compounds.Staphylococcus aureus Infection in Humanized Mice: A New Model to Study Pathogenicity Associated With Human Immune Response.Staphylococcus epidermidis Esp degrades specific proteins associated with Staphylococcus aureus biofilm formation and host-pathogen interaction A clinical evaluation of the efficacy and safety of singlet oxygen in cleansing and disinfecting stagnating wounds.Efficacy of Trans-cinnamaldehyde and Eugenol in Reducing Acinetobacter baumannii Adhesion to and Invasion of Human Keratinocytes and Controlling Wound Infection In Vitro.Effect of negative pressure on growth, secretion and biofilm formation of Staphylococcus aureus.Role of SCCmec type in resistance to the synergistic activity of oxacillin and cefoxitin in MRSA.Fur is required for the activation of virulence gene expression through the induction of the sae regulatory system in Staphylococcus aureus.Iron-regulated biofilm formation in Staphylococcus aureus Newman requires ica and the secreted protein Emp.eap Gene as novel target for specific identification of Staphylococcus aureus.Endogenous developmental endothelial locus-1 limits ischaemia-related angiogenesis by blocking inflammation.Protective immunization against Staphylococcus aureus infection in a novel experimental wound model in mice.Novel aspects in the regulation of the leukocyte adhesion cascade.Staphylococcus aureus colonization and non-influenza respiratory viruses: Interactions and synergism mechanisms

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P2860

The extracellular adherence protein (Eap) of Staphylococcus aureus inhibits wound healing by interfering with host defense and repair mechanisms.

http://www.wikidata.org/entity/Q35848578

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The extracellular adherence pr ...... defense and repair mechanisms.

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Astrid Sobke

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Athanasios N Athanasopoulos

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Darius Schneider

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Hans-Peter Hammes

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Holger Weber

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Mathias Herrmann

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Matina Economopoulou

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Muzaffar Hussain

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Peter P Nawroth

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Sabine A Eming

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P2860

Staphylococcus aureus infections

Role of tissue factor in hemostasis, thrombosis, and vascular development

The junctional adhesion molecule-C promotes neutrophil transendothelial migration in vitro and in vivo

Impairment of skin wound healing in beta-1,4-galactosyltransferase-deficient mice with reduced leukocyte recruitment

Traffic signals for lymphocyte recirculation and leukocyte emigration: the multistep paradigm

Angiogenesis in health and disease

Wound healing--aiming for perfect skin regeneration

Cutaneous wound healing

Leukocyte trans-endothelial migration: JAMs add new pieces to the puzzle.

Identification of a Staphylococcus aureus extracellular matrix-binding protein with broad specificity.

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2720-2727

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scholarly article

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10.1182/BLOOD-2005-08-3140

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7

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Hellmut G Augustin

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2005-11-29T00:00:00Z

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1895382

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