Pathology of Berkeley sickle cell mice: similarities and differences with human sickle cell disease. - Wikidata - wikimedia - TriplyDB

Pathology of Berkeley sickle cell mice: similarities and differences with human sickle cell disease.

Pathology of Berkeley sickle cell mice: similarities and differences with human sickle cell disease.

http://www.wikidata.org/entity/Q35848672

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Mechanism of testosterone deficiency in the transgenic sickle cell mouse.P-selectin-mediated platelet-neutrophil aggregate formation activates neutrophils in mouse and human sickle cell disease.Cognitive and behavior deficits in sickle cell mice are associated with profound neuropathologic changes in hippocampus and cerebellum.Lung vaso-occlusion in sickle cell disease mediated by arteriolar neutrophil-platelet microemboli.Morphological and functional platelet abnormalities in Berkeley sickle cell mice.Establishment of a transgenic sickle-cell mouse model to study the pathophysiology of priapism.Global gene expression profiling of endothelium exposed to heme reveals an organ-specific induction of cytoprotective enzymes in sickle cell disease.Pain-related behaviors and neurochemical alterations in mice expressing sickle hemoglobin: modulation by cannabinoids.Hydroxyurea therapy requires HbF induction for clinical benefit in a sickle cell mouse model.Drug-loaded sickle cells programmed ex vivo for delayed hemolysis target hypoxic tumor microvessels and augment tumor drug delivery.TNF-α, IFN-γ, IL-10, and IL-4 levels were elevated in a murine model of human sickle cell anemia maintained on a high protein/calorie diet.Heme oxygenase-1 is a modulator of inflammation and vaso-occlusion in transgenic sickle mice.High protein diet attenuates histopathologic organ damage and vascular leakage in transgenic murine model of sickle cell anemia Sickle cell disease increases high mobility group box 1: a novel mechanism of inflammation.Vasculopathy and pulmonary hypertension in sickle cell disease.Sensitization of nociceptive spinal neurons contributes to pain in a transgenic model of sickle cell disease.Sickle cell disease: old discoveries, new concepts, and future promise.Hemolysis in sickle cell mice causes pulmonary hypertension due to global impairment in nitric oxide bioavailability Updated Mechanisms of Sickle Cell Disease-Associated Chronic pain Comparative Analysis of Pain Behaviours in Humanized Mouse Models of Sickle Cell Anemia.Red blood cell alloimmunization in sickle cell disease: pathophysiology, risk factors, and transfusion management.Subacute Hemolysis in Sickle Cell Mice Causes Priapism Secondary to NO Imbalance and PDE5 Dysregulation Morphine promotes renal pathology in sickle mice.Placenta growth factor augments airway hyperresponsiveness via leukotrienes and IL-13.A novel hemoglobin-binding peptide reduces cell-free hemoglobin in murine hemolytic anemia.Use of mouse models to study the mechanisms and consequences of RBC clearance.No evidence for cell activation or brain vaso-occlusion with plerixafor mobilization in sickle cell mice Neurobiological mechanisms of pain in sickle cell disease.Histopathology of experimentally induced asthma in a murine model of sickle cell disease.Hypoxia-mediated impaired erythrocyte Lands' Cycle is pathogenic for sickle cell disease.C-reactive protein and interleukin-6 are decreased in transgenic sickle cell mice fed a high protein diet.Attenuated RhoA/Rho-kinase signaling in penis of transgenic sickle cell mice Correction of murine sickle cell disease using gamma-globin lentiviral vectors to mediate high-level expression of fetal hemoglobin.A novel human gamma-globin gene vector for genetic correction of sickle cell anemia in a humanized sickle mouse model: critical determinants for successful correction Electroacupuncture in conscious free-moving mice reduces pain by ameliorating peripheral and central nociceptive mechanisms.Regulation of iron absorption in hemoglobinopathies.Malnutrition in Sickle Cell Anemia: Implications for Infection, Growth, and Maturation.Inhaled carbon monoxide reduces leukocytosis in a murine model of sickle cell disease Targeting Mac-1-mediated leukocyte-RBC interactions uncouples the benefits for acute vaso-occlusion and chronic organ damage.Alloimmunization to transfused HOD red blood cells is not increased in mice with sickle cell disease.

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P2860

Pathology of Berkeley sickle cell mice: similarities and differences with human sickle cell disease.

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2005 nî lūn-bûn

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Pathology of Berkeley sickle c ...... ith human sickle cell disease.

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Barry S Coller

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Carol A Bodian

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Cheryl A Hillery

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Elizabeth A Manci

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Gerard A Lutty

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P2860

Retinal degeneration mutants in the mouse

Quantitation of microvascular plasma perfusion and neuronal microtubule-associated protein in ischemic mouse brain by laser-scanning confocal microscopy.

Identification of a nonsense mutation in the rod photoreceptor cGMP phosphodiesterase beta-subunit gene of the rd mouse.

Early renal changes in sickle cell anemia.

Early splenomegaly in homozygous sickle-cell disease: An indicator of susceptibility to infection.

Clinicopathologic analysis of cardiac dysfunction in 52 patients with sickle cell anemia.

Acute chest syndrome in sickle cell disease: clinical presentation and course. Cooperative Study of Sickle Cell Disease.

Acute infarction of long bones in children with sickle cell anemia.

Transgenic and gene knock-out mouse models of sickle cell anemia and the thalassemias.

Evolution of the retinal black sunburst in sickling haemoglobinopathies

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