Altered granulopoietic profile and exaggerated acute neutrophilic inflammation in mice with targeted deficiency in the sialyltransferase ST6Gal I
about
Pharmacogenomics of adverse drug reactionsA novel role for CD55 in granulocyte homeostasis and anti-bacterial host defenseST6Gal-I regulates macrophage apoptosis via α2-6 sialylation of the TNFR1 death receptorFluorinated per-acetylated GalNAc metabolically alters glycan structures on leukocyte PSGL-1 and reduces cell binding to selectins.Altered eosinophil profile in mice with ST6Gal-1 deficiency: an additional role for ST6Gal-1 generated by the P1 promoter in regulating allergic inflammation.Role for hepatic and circulatory ST6Gal-1 sialyltransferase in regulating myelopoiesis.Anti-inflammatory IgG production requires functional P1 promoter in β-galactoside α2,6-sialyltransferase 1 (ST6Gal-1) geneEmerging role of alpha2,6-sialic acid as a negative regulator of galectin binding and function.Treatment response in Kawasaki disease is associated with sialylation levels of endogenous but not therapeutic intravenous immunoglobulin GNMR detection and characterization of sialylated glycoproteins and cell surface polysaccharidesNMR characterization of immunoglobulin G Fc glycan motion on enzymatic sialylation.Identification of novel plasma glycosylation-associated markers of aging.Systems-level studies of glycosyltransferase gene expression and enzyme activity that are associated with the selectin binding function of human leukocytes.p40phox expression regulates neutrophil recruitment and function during the resolution phase of intestinal inflammation.Disruption of thymopoiesis in ST6Gal I-deficient miceDendritic cells: a spot on sialic Acid.Remodeling of marrow hematopoietic stem and progenitor cells by non-self ST6Gal-1 sialyltransferase.Platelets support extracellular sialylation by supplying the sugar donor substrate.Pharmacogenetics and human genetic polymorphisms.The blood-borne sialyltransferase ST6Gal-1 is a negative systemic regulator of granulopoiesis.Immunological aspects of congenital disorders of glycosylation (CDG): a review.Circulating blood and platelets supply glycosyltransferases that enable extrinsic extracellular glycosylation.Chorionic gonadotropin alleviates thioglycollate-induced peritonitis by affecting macrophage function.Identification of PTPN22, ST6GAL1 and JAZF1 as psoriasis risk genes demonstrates shared pathogenesis between psoriasis and diabetes.Extrinsic sialylation is dynamically regulated by systemic triggers in vivo.The phagocytic capacity and immunological potency of human dendritic cells is improved by α2,6-sialic acid deficiency.Systemic ST6Gal-1 Is a Pro-survival Factor for Murine Transitional B Cells
P2860
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P2860
Altered granulopoietic profile and exaggerated acute neutrophilic inflammation in mice with targeted deficiency in the sialyltransferase ST6Gal I
description
2006 nî lūn-bûn
@nan
2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
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2006年论文
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2006年论文
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name
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@ast
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@en
type
label
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@ast
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@en
prefLabel
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@ast
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@en
P2093
P2860
P1433
P1476
Altered granulopoietic profile ...... the sialyltransferase ST6Gal I
@en
P2093
Ashlee Urbasic
Brahm H Segal
Joseph T Lau
Julie R Ostberg
Mehrab Nasirikenari
P2860
P304
P356
10.1182/BLOOD-2006-04-014779
P407
P577
2006-07-18T00:00:00Z