c-Rel plays a key role in deficient activation of B cells from a non-X-linked hyper-IgM patient.
about
c-Rel deficiency increases caspase-4 expression and leads to ER stress and necrosis in EBV-transformed cellsCCAAT/enhancer-binding protein β and NF-κB mediate high level expression of chemokine genes CCL3 and CCL4 by human chondrocytes in response to IL-1β.REL, encoding a member of the NF-kappaB family of transcription factors, is a newly defined risk locus for rheumatoid arthritisPTIP chromatin regulator controls development and activation of B cell subsets to license humoral immunity in mice.
P2860
c-Rel plays a key role in deficient activation of B cells from a non-X-linked hyper-IgM patient.
description
2006 nî lūn-bûn
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name
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@ast
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@en
type
label
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@ast
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@en
prefLabel
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@ast
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@en
P2093
P2860
P1433
P1476
c-Rel plays a key role in defi ...... on-X-linked hyper-IgM patient.
@en
P2093
Charles Song
Frank L Sinquett
Kristina T Lu
Lori R Covey
Rebecca L Dryer
P2860
P304
P356
10.1182/BLOOD-2006-03-008839
P407
P577
2006-08-08T00:00:00Z