Diabetes promotes an inflammatory macrophage phenotype and atherosclerosis through acyl-CoA synthetase 1. - Wikidata - wikimedia - TriplyDB

Diabetes promotes an inflammatory macrophage phenotype and atherosclerosis through acyl-CoA synthetase 1.

Diabetes promotes an inflammatory macrophage phenotype and atherosclerosis through acyl-CoA synthetase 1.

http://www.wikidata.org/entity/Q35849515

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Adhesive capsulitis of the shoulder and diabetes: a meta-analysis of prevalence The Anti-Inflammatory and Antibacterial Action of Nanocrystalline Silver and Manuka Honey on the Molecular Alternation of Diabetic Foot Ulcer: A Comprehensive Literature Review Lipids and the endothelium: bidirectional interactions Dietary polyunsaturated fatty acids and inflammation: the role of phospholipid biosynthesis Modulating the Gut Microbiota Improves Glucose Tolerance, Lipoprotein Profile and Atherosclerotic Plaque Development in ApoE-Deficient Mice Glucose induces sensitivity to oxygen deprivation and modulates insulin/IGF-1 signaling and lipid biosynthesis in Caenorhabditis elegans.Metabolomics and diabetes: analytical and computational approaches Genetic association of long-chain acyl-CoA synthetase 1 variants with fasting glucose, diabetes, and subclinical atherosclerosis Macrophages in homeostatic immune function.Testing the role of myeloid cell glucose flux in inflammation and atherosclerosis miR33 inhibition overcomes deleterious effects of diabetes mellitus on atherosclerosis plaque regression in mice.Impact of a synthetic cannabinoid (CP-47,497-C8) on protein expression in human cells: evidence for induction of inflammation and DNA damage.Regulation of inflammatory phenotype in macrophages by a diabetes-induced long noncoding RNA.Distinct role of FoxO1 in M-CSF- and GM-CSF-differentiated macrophages contributes LPS-mediated IL-10: implication in hyperglycemia.Effects of sulfasalazine and tofacitinib on the protein profile of articular chondrocytes.Signalling mechanisms regulating phenotypic changes in breast cancer cells.Long-chain acyl-CoA synthetase in fatty acid metabolism involved in liver and other diseases: an update Effects of High Fat Feeding and Diabetes on Regression of Atherosclerosis Induced by Low-Density Lipoprotein Receptor Gene Therapy in LDL Receptor-Deficient Mice Uncomplicating the Macrovascular Complications of Diabetes: The 2014 Edwin Bierman Award Lecture Physiological Consequences of Compartmentalized Acyl-CoA Metabolism.Myeloid Cell Prostaglandin E2 Receptor EP4 Modulates Cytokine Production but Not Atherogenesis in a Mouse Model of Type 1 Diabetes.Meal frequency patterns and glycemic properties of maternal diet in relation to preterm delivery: Results from a large prospective cohort study Characterization of the promoter region of the bovine long-chain acyl-CoA synthetase 1 gene: Roles of E2F1, Sp1, KLF15, and E2F4.Endothelial acyl-CoA synthetase 1 is not required for inflammatory and apoptotic effects of a saturated fatty acid-rich environment.Macrophage Phenotype and Function in Different Stages of Atherosclerosis.Acyl-CoA synthetase 1 is induced by Gram-negative bacteria and lipopolysaccharide and is required for phospholipid turnover in stimulated macrophages.Neurogenic factor-induced Langerhans cell activation in diabetic mice with mechanical allodynia Metabolic reprogramming through fatty acid transport protein 1 (FATP1) regulates macrophage inflammatory potential and adipose inflammation.Cyclooxygenase inhibition improves endothelial vasomotor dysfunction of visceral adipose arterioles in human obesity.Bone marrow-derived and peritoneal macrophages have different inflammatory response to oxLDL and M1/M2 marker expression - implications for atherosclerosis research.Toll-like receptor agonists promote prolonged triglyceride storage in macrophages.BACH2: a marker of DNA damage and ageing.Resolvins, specialized proresolving lipid mediators, and their potential roles in metabolic diseases 2013 Russell Ross memorial lecture in vascular biology: cellular and molecular mechanisms of diabetes mellitus-accelerated atherosclerosis.Lysosomes integrate metabolic-inflammatory cross-talk in primary macrophage inflammasome activation Inflammation and diabetes-accelerated atherosclerosis: myeloid cell mediators.Fatty acids and cardiac disease: fuel carrying a message.Acyl-CoA metabolism and partitioning.Extended Multiplexing of Tandem Mass Tags (TMT) Labeling Reveals Age and High Fat Diet Specific Proteome Changes in Mouse Epididymal Adipose Tissue.Macrophage metabolism in atherosclerosis.

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P2860

Diabetes promotes an inflammatory macrophage phenotype and atherosclerosis through acyl-CoA synthetase 1.

http://www.wikidata.org/entity/Q35849515

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Proceedings of the National Academy of Sciences of the United States of America

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Diabetes promotes an inflammat ...... through acyl-CoA synthetase 1.

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Alan Chait

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Anuradha Vivekanandan-Giri

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Charles N Serhan

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Farah Kramer

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Jay W Heinecke

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Jenny E Kanter

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Karin E Bornfeldt

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Kathleen R Braun

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Lei O Li

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Lev Becker

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P2860

Intensive Diabetes Treatment and Cardiovascular Disease in Patients with Type 1 Diabetes

A novel prostaglandin E receptor 4-associated protein participates in antiinflammatory signaling

Long-chain acyl-CoA synthetase 4 modulates prostaglandin E₂ release from human arterial smooth muscle cells

Human aldose reductase expression accelerates diabetic atherosclerosis in transgenic mice

Insulin resistance, hyperglycemia, and atherosclerosis

Increased monocytic activity and biomarkers of inflammation in patients with type 1 diabetes

Overexpression of rat long chain acyl-coa synthetase 1 alters fatty acid metabolism in rat primary hepatocytes

Transcriptional profiling of the human monocyte-to-macrophage differentiation and polarization: new molecules and patterns of gene expression

Elevated glucose and diabetes promote interleukin-12 cytokine gene expression in mouse macrophages.

Elevated cyclic AMP and PDE4 inhibition induce chemokine expression in human monocyte-derived macrophages

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109

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2012-01-17T00:00:00Z

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221808843

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22308341

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atherosclerosis

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3311324

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