Cytomegalovirus infection induces high levels of cyclins, phosphorylated Rb, and p53, leading to cell cycle arrest.
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Repression of HMGA2 gene expression by human cytomegalovirus involves the IE2 86-kilodalton protein and is necessary for efficient viral replication and inhibition of cyclin A transcriptionUse of differential display analysis to assess the effect of human cytomegalovirus infection on the accumulation of cellular RNAs: induction of interferon-responsive RNAsHuman cytomegalovirus infection leads to accumulation of geminin and inhibition of the licensing of cellular DNA replication.Neuropathogenesis of congenital cytomegalovirus infection: disease mechanisms and prospects for interventionThe ORF3 protein of porcine circovirus type 2 interacts with porcine ubiquitin E3 ligase Pirh2 and facilitates p53 expression in viral infectionPhosphorylation of Golgi Peripheral Membrane Protein Grasp65 Is an Integral Step in the Formation of the Human Cytomegalovirus Cytoplasmic Assembly CompartmentMurine cytomegalovirus stimulates cellular thymidylate synthase gene expression in quiescent cells and requires the enzyme for replicationCyclin-dependent kinase-like function is shared by the beta- and gamma- subset of the conserved herpesvirus protein kinasesCyclin-dependent kinase activity controls the onset of the HCMV lytic cycleCyclin A degradation by primate cytomegalovirus protein pUL21a counters its innate restriction of virus replication.Inhibition of human cytomegalovirus immediate-early gene expression by cyclin A2-dependent kinase activityChronic viral infection and primary central nervous system malignancy.Infection of cells with human cytomegalovirus during S phase results in a blockade to immediate-early gene expression that can be overcome by inhibition of the proteasome.p53 and RPA are sequestered in viral replication centers in the nuclei of cells infected with human cytomegalovirusCytomegalovirus-induced embryopathology: mouse submandibular salivary gland epithelial-mesenchymal ontogeny as a modelViral and cell cycle-regulated kinases in cytomegalovirus-induced pseudomitosis and replication.Human herpesvirus 6A (HHV-6A) and HHV-6B alter E2F1/Rb pathways and E2F1 localization and cause cell cycle arrest in infected T cellsViral mimicry of Cdc2/cyclin-dependent kinase 1 mediates disruption of nuclear lamina during human cytomegalovirus nuclear egress.Human cytomegalovirus protein pUL117 targets the mini-chromosome maintenance complex and suppresses cellular DNA synthesis.Human cytomegalovirus and human herpesvirus 6 genes that transform and transactivateA viral microRNA down-regulates multiple cell cycle genes through mRNA 5'UTRsPolo-like kinase 1 as a target for human cytomegalovirus pp65 lower matrix protein.Effects of human cytomegalovirus major immediate-early proteins in controlling the cell cycle and inhibiting apoptosis: studies with ts13 cellsHuman papillomavirus type 16 E1 E4-induced G2 arrest is associated with cytoplasmic retention of active Cdk1/cyclin B1 complexes.Erratum: Differential expressed genes in ECV304 Endothelial-like Cells infected with Human Cytomegalovirus.Noise cancellation: viral fine tuning of the cellular environment for its own genome replication.Inhibition of cell division by the human cytomegalovirus IE86 protein: role of the p53 pathway or cyclin-dependent kinase 1/cyclin B1.Identification of positive and negative regulatory regions involved in regulating expression of the human cytomegalovirus UL94 late promoter: role of IE2-86 and cellular p53 in mediating negative regulatory function.Phosphorylation of the human cytomegalovirus 86-kilodalton immediate-early protein IE2.Human cytomegalovirus persistently infects aortic endothelial cells.Recombinant, replication-defective adenovirus gene transfer vectors induce cell cycle dysregulation and inappropriate expression of cyclin proteins.Dysregulation of cyclin E gene expression in human cytomegalovirus-infected cells requires viral early gene expression and is associated with changes in the Rb-related protein p130.The human cytomegalovirus IE86 protein can block cell cycle progression after inducing transition into the S phase of permissive cells.Role of human cytomegalovirus immediate-early proteins in cell growth control.The murine gammaherpesvirus 68 v-cyclin gene is an oncogene that promotes cell cycle progression in primary lymphocytes.The Epstein-Barr virus protein BRLF1 activates S phase entry through E2F1 induction.Reactivation of the human cytomegalovirus major immediate-early regulatory region and viral replication in embryonal NTera2 cells: role of trichostatin A, retinoic acid, and deletion of the 21-base-pair repeats and modulator.Human cytomegalovirus cell-to-cell spread in the absence of an essential assembly protein.Herpesvirus lytic replication and the cell cycle: arresting new developmentsExon 3 of the human cytomegalovirus major immediate-early region is required for efficient viral gene expression and for cellular cyclin modulation.
P2860
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P2860
Cytomegalovirus infection induces high levels of cyclins, phosphorylated Rb, and p53, leading to cell cycle arrest.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@ast
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@en
type
label
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@ast
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@en
prefLabel
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@ast
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@en
P2093
P2860
P1433
P1476
Cytomegalovirus infection indu ...... leading to cell cycle arrest.
@en
P2093
D D Richman
D H Spector
E A Fortunato
P2860
P304
P407
P577
1995-11-01T00:00:00Z